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Title[redir]:
Targeted disruption of the mouse transforming growth factor-β1 gene results in multifocal inflammatory disease | Nature
Description:
Transforming growth factor-β1 (TGF-β1) is a multifunctional growth factor that has profound regulatory effects on many developmental and physiological processes. Disruption of the TGF-β1 gene by homologous recombination in murine embryonic stem cells enables mice to be generated that carry the disrupted allele. Animals homozygous for the mutated TGF-β1 allele show no gross developmental abnormalities, but about 20 days after birth they succumb to a wasting syndrome accompanied by a multifocal, mixed inflammatory cell response and tissue necrosis, leading to organ failure and death. TGF-β1-deficient mice may be valuable models for human immune and inflammatory disorders, including autoimmune diseases, transplant rejection and graft versus host reactions.
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google, scholar, cas, article, nature, pubmed, immun, access, ads, cell, content, usa, cookies, tgfβ, biol, development, privacy, growth, sporn, exp, proc, natn, acad, sci, data, inflammatory, disease, open, med, cincinnati, advertising, information, subscribe, october, disruption, transforming, factorβ, gene, multifocal, shull, ormsby, kier, allen, thomas, doetschman, immune, diseases, institution, buy, cancer,
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nature portfolio permissions reprints privacy policy autocrine tgf-β-positive feedback advertising tgf-β1-deficient mice social media subscribe nature tgf-β signaling nature 301 nature 334 nature 312 nature 359 nature tgf-β1 gene multifunctional growth factor personal data springerlink instant access data protection permissions peptide growth factors multifocal inflammatory disease profound regulatory effects tgf-β1 privacy explore content subscription content european economic area wasting syndrome accompanied institutional subscriptions read phase 1a study healthy volunteers lyrialle accepting optional cookies including autoimmune diseases journals search log issue learn bone marrow epcs anti-tgfβ3 antibody manage preferences gross developmental abnormalities article purchase access content article cite gabriele proetzel dawn calvin human immune immune balance article shull https
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