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DOI . ORG {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Keywords
  7. Topics
  8. Schema
  9. External Links
  10. Analytics And Tracking
  11. Libraries
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We began analyzing https://link.springer.com/article/10.1007/s11894-005-0062-5, but it redirected us to https://link.springer.com/article/10.1007/s11894-005-0062-5. The analysis below is for the second page.

Title[redir]:
Wilson disease: New insights into pathogenesis, diagnosis, and future therapy | Current Gastroenterology Reports
Description:
Wilson disease is caused by disease-specific mutations of the copper transporting ATPase, ATP7B. The diagnosis is established by clinical and biochemical means, though advances in molecular diagnostics will someday permit de novo diagnosis. The patient may present with hepatic, neurologic, or psychiatric symptoms, or a combination of these. Both environmental and extragenic effects contribute to the varied phenotypic presentations of this disease. Patients can be treated effectively with chelating agents or zinc salts, or with liver transplantation. Liver cell transplant and gene therapy offer potential cures for this disorder, but at present only data from preclinical studies on animal models are available. Future advances in immunotolerization and gene therapy will likely enable human trials for treatment of this disorder and other genetic disorders of hepatic metabolism.

Matching Content Categories {πŸ“š}

  • Science
  • Health & Fitness
  • Education

Content Management System {πŸ“}

What CMS is doi.org built with?

Custom-built

No common CMS systems were detected on Doi.org, and no known web development framework was identified.

Traffic Estimate {πŸ“ˆ}

What is the average monthly size of doi.org audience?

πŸ™οΈ Massive Traffic: 50M - 100M visitors per month


Based on our best estimate, this website will receive around 98,426,998 visitors per month in the current month.

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How Does Doi.org Make Money? {πŸ’Έ}

We're unsure how the site profits.

While profit motivates many websites, others exist to inspire, entertain, or provide valuable resources. Websites have a variety of goals. And this might be one of them. Doi.org could have a money-making trick up its sleeve, but it's undetectable for now.

Keywords {πŸ”}

disease, article, google, scholar, pubmed, wilson, cas, copper, gene, liver, wilsons, atpb, lec, rats, hepatic, transplantation, genet, gastroenterology, phenotypic, hum, rat, expression, privacy, cookies, content, data, diagnosis, schilsky, cell, studies, menkes, toxicosis, normal, function, analysis, publish, search, therapy, mutations, patients, human, access, biochem, model, demonstrated, correction, information, log, journal, research,

Topics {βœ’οΈ}

n-terminal metal-binding sites crigler-najjar syndrome type month download article/chapter long-evans cinnamon rats vivo site-directed mutagenesis chimeric rna/dna oligonucleotides udp-glucuronosyltransferase gene defect hepatocellular copper metabolism copper transporting atpase full article pdf human liver cdna privacy choices/manage cookies hepatic metabolism prevent copper toxicosis article schilsky normal liver cells hepatic gene correction liver cell lines wilson disease gene copper metabolic pathway van de sluis factor ix gene liver cell transplant wilson disease locus biliary copper excretion kren bt hepatic copper disease-specific mutants disease protein plays copper transport defect menkes patient fibroblasts rats successfully transplanted normal rat hepatocytes european economic area related subjects gitlin jd defective cellular localization proper cellular localization genotype-phenotype correlation alter gene expression human wilson protein hepatoma cell lines enable human trials conditions privacy policy endosomal/lysosomal vesicles iron overload disorders achieve selective repopulation wilson disease patients future therapy published varied phenotypic presentations

Schema {πŸ—ΊοΈ}

WebPage:
      mainEntity:
         headline:Wilson disease: New insights into pathogenesis, diagnosis, and future therapy
         description:Wilson disease is caused by disease-specific mutations of the copper transporting ATPase, ATP7B. The diagnosis is established by clinical and biochemical means, though advances in molecular diagnostics will someday permit de novo diagnosis. The patient may present with hepatic, neurologic, or psychiatric symptoms, or a combination of these. Both environmental and extragenic effects contribute to the varied phenotypic presentations of this disease. Patients can be treated effectively with chelating agents or zinc salts, or with liver transplantation. Liver cell transplant and gene therapy offer potential cures for this disorder, but at present only data from preclinical studies on animal models are available. Future advances in immunotolerization and gene therapy will likely enable human trials for treatment of this disorder and other genetic disorders of hepatic metabolism.
         datePublished:
         dateModified:
         pageStart:26
         pageEnd:31
         sameAs:https://doi.org/10.1007/s11894-005-0062-5
         keywords:
            Wilson Disease
            Copper Metabolism
            Menkes Disease
            ATP7B Gene
            Hepatic Copper
            Gastroenterology
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            issn:
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               1522-8037
            volumeNumber:7
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               type:ImageObject
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         author:
               name:Michael L. Schilsky
               affiliation:
                     name:The New York Weill Cornell Medical Center
                     address:
                        name:Center for Liver Disease and Transplantation, The New York Weill Cornell Medical Center, New York, USA
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ScholarlyArticle:
      headline:Wilson disease: New insights into pathogenesis, diagnosis, and future therapy
      description:Wilson disease is caused by disease-specific mutations of the copper transporting ATPase, ATP7B. The diagnosis is established by clinical and biochemical means, though advances in molecular diagnostics will someday permit de novo diagnosis. The patient may present with hepatic, neurologic, or psychiatric symptoms, or a combination of these. Both environmental and extragenic effects contribute to the varied phenotypic presentations of this disease. Patients can be treated effectively with chelating agents or zinc salts, or with liver transplantation. Liver cell transplant and gene therapy offer potential cures for this disorder, but at present only data from preclinical studies on animal models are available. Future advances in immunotolerization and gene therapy will likely enable human trials for treatment of this disorder and other genetic disorders of hepatic metabolism.
      datePublished:
      dateModified:
      pageStart:26
      pageEnd:31
      sameAs:https://doi.org/10.1007/s11894-005-0062-5
      keywords:
         Wilson Disease
         Copper Metabolism
         Menkes Disease
         ATP7B Gene
         Hepatic Copper
         Gastroenterology
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      isPartOf:
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      author:
            name:Michael L. Schilsky
            affiliation:
                  name:The New York Weill Cornell Medical Center
                  address:
                     name:Center for Liver Disease and Transplantation, The New York Weill Cornell Medical Center, New York, USA
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      name:Current Gastroenterology Reports
      issn:
         1534-312X
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      volumeNumber:7
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      name:Current Medicine Group
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         url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
         type:ImageObject
      name:The New York Weill Cornell Medical Center
      address:
         name:Center for Liver Disease and Transplantation, The New York Weill Cornell Medical Center, New York, USA
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      url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
Person:
      name:Michael L. Schilsky
      affiliation:
            name:The New York Weill Cornell Medical Center
            address:
               name:Center for Liver Disease and Transplantation, The New York Weill Cornell Medical Center, New York, USA
               type:PostalAddress
            type:Organization
PostalAddress:
      name:Center for Liver Disease and Transplantation, The New York Weill Cornell Medical Center, New York, USA
WebPageElement:
      isAccessibleForFree:
      cssSelector:.main-content

External Links {πŸ”—}(144)

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