DOI . ORG {}

Analyzed Page
Matching Content Categories
CMS
Monthly Traffic Estimate
How Does Doi.org Make Money
Keywords
Topics
Schema
External Links
Analytics And Tracking
Libraries
Hosting Providers
CDN Services

We began analyzing https://link.springer.com/article/10.1007/s002230001148, but it redirected us to https://link.springer.com/article/10.1007/s002230001148. The analysis below is for the second page.

Title[redir]:
Birth Weight, Birth Length, and Bone Density in Prepubertal Children: Evidence for an Association That May Be Mediated by Genetic Factors | Calcified Tissue International
Description:
There is an incomplete understanding of the contribution of early growth to bone accrual in childhood. The aims of this longitudinal study were to examine the association between growth variables at birth, 1 month, and 8 years and bone density in prepubertal children. Weight and length at both birth and 1 month of age were measured in 1988 as part of a prospective study for sudden infant death syndrome. A total of 330 children (47%) and 278 of their mothers were then contacted in 1996 for measurement of anthropometrics and bone density. Birth weight, birth length, and length gain (but not weight gain) in the first month all made significant contributions to areal bone density (BMD, g/cm2) at all sites at age 8 even after taking into account subsequent weight and height gain (model R2 14–39% depending on variable and site). Adjustment for potential environmental confounders did not alter these findings, however, adjustment for maternal BMD markedly reduced the early life associations (particularly for birth weight). Though early life factors were weakly associated with bone mineral apparent density (BMAD, g/cm3) in correlation analysis, subsequent height and weight gain were the only significant independent contributors to BMAD. In conclusion, early life anthropometrics make little contribution to BMAD (other than through their correlation with later growth) but make significant independent contributions to BMD suggesting that the growth trajectory of bone is determined very early in life. In addition, the contribution of body size at birth to bone growth in early life appears to be mediated by genetic factors although it is possible that it may be mediated by poorly measured or as yet unidentified determinants of body size at birth.

Matching Content Categories {📚}

Family & Parenting
Science
Education

Content Management System {📝}

What CMS is doi.org built with?

Custom-built

No common CMS systems were detected on Doi.org, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of doi.org audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month

Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

check SE Ranking
check Ahrefs
check Similarweb
check Ubersuggest
check Semrush

How Does Doi.org Make Money? {💸}

We can't see how the site brings in money.

Not all websites are made for profit; some exist to inform or educate users. Or any other reason why people make websites. And this might be the case. Doi.org might be earning cash quietly, but we haven't detected the monetization method.

Keywords {🔍}

bone, birth, article, weight, density, length, growth, content, early, life, privacy, cookies, month, access, information, publish, research, search, children, mediated, factors, gain, make, data, log, journal, tissue, prepubertal, association, genetic, jones, dwyer, contribution, study, age, significant, bmd, mineral, bmad, body, size, discover, development, springer, site, optional, analysis, personal, parties, policy,

Topics {✒️}

month download article/chapter population health research privacy choices/manage cookies full article pdf bone mineral density areal bone density made significant contributions significant independent contributors european economic area scope submit manuscript potential environmental confounders bone accrual conditions privacy policy account subsequent weight early life associations early life appears accepting optional cookies genetic factors published biomechanics development early life factors bone growth main content log journal finder publish body size bone density early growth article log check access related subjects instant access growth variables growth trajectory article cite privacy policy personal data article jones books a optional cookies birth weight manage preferences maternal diabetes infant adiposity genetic factors weight gain data protection essential cookies cookies skip subsequent height institution subscribe journal publish

Schema {🗺️}

WebPage:
      mainEntity:
         headline:Birth Weight, Birth Length, and Bone Density in Prepubertal Children: Evidence for an Association That May Be Mediated by Genetic Factors
         description: There is an incomplete understanding of the contribution of early growth to bone accrual in childhood. The aims of this longitudinal study were to examine the association between growth variables at birth, 1 month, and 8 years and bone density in prepubertal children. Weight and length at both birth and 1 month of age were measured in 1988 as part of a prospective study for sudden infant death syndrome. A total of 330 children (47%) and 278 of their mothers were then contacted in 1996 for measurement of anthropometrics and bone density. Birth weight, birth length, and length gain (but not weight gain) in the first month all made significant contributions to areal bone density (BMD, g/cm2) at all sites at age 8 even after taking into account subsequent weight and height gain (model R2 14–39% depending on variable and site). Adjustment for potential environmental confounders did not alter these findings, however, adjustment for maternal BMD markedly reduced the early life associations (particularly for birth weight). Though early life factors were weakly associated with bone mineral apparent density (BMAD, g/cm3) in correlation analysis, subsequent height and weight gain were the only significant independent contributors to BMAD. In conclusion, early life anthropometrics make little contribution to BMAD (other than through their correlation with later growth) but make significant independent contributions to BMD suggesting that the growth trajectory of bone is determined very early in life. In addition, the contribution of body size at birth to bone growth in early life appears to be mediated by genetic factors although it is possible that it may be mediated by poorly measured or as yet unidentified determinants of body size at birth.
         datePublished:2014-02-11T00:00:00Z
         dateModified:2014-02-11T00:00:00Z
         pageStart:304
         pageEnd:308
         sameAs:https://doi.org/10.1007/s002230001148
         keywords:
            Key words: Bone density — Childhood — Growth — Genetics — Body size.
            Biochemistry
            general
            Endocrinology
            Orthopedics
            Cell Biology
         image:
         isPartOf:
            name:Calcified Tissue International
            issn:
               1432-0827
               0171-967X
            volumeNumber:67
            type:
               Periodical
               PublicationVolume
         publisher:
            name:Springer-Verlag
            logo:
               url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
               type:ImageObject
            type:Organization
         author:
               name:G. Jones
               affiliation:
                     name:Menzies Centre for Population Health Research, GPO Box 252-23, Hobart, Tasmania, Australia, 7001
                     address:
                        name:Menzies Centre for Population Health Research, GPO Box 252-23, Hobart, Tasmania, Australia, 7001, Australia
                        type:PostalAddress
                     type:Organization
               type:Person
               name:T. Dwyer
               affiliation:
                     name:Menzies Centre for Population Health Research, GPO Box 252-23, Hobart, Tasmania, Australia, 7001
                     address:
                        name:Menzies Centre for Population Health Research, GPO Box 252-23, Hobart, Tasmania, Australia, 7001, Australia
                        type:PostalAddress
                     type:Organization
               type:Person
         isAccessibleForFree:
         hasPart:
            isAccessibleForFree:
            cssSelector:.main-content
            type:WebPageElement
         type:ScholarlyArticle
      context:https://schema.org
ScholarlyArticle:
      headline:Birth Weight, Birth Length, and Bone Density in Prepubertal Children: Evidence for an Association That May Be Mediated by Genetic Factors
      description: There is an incomplete understanding of the contribution of early growth to bone accrual in childhood. The aims of this longitudinal study were to examine the association between growth variables at birth, 1 month, and 8 years and bone density in prepubertal children. Weight and length at both birth and 1 month of age were measured in 1988 as part of a prospective study for sudden infant death syndrome. A total of 330 children (47%) and 278 of their mothers were then contacted in 1996 for measurement of anthropometrics and bone density. Birth weight, birth length, and length gain (but not weight gain) in the first month all made significant contributions to areal bone density (BMD, g/cm2) at all sites at age 8 even after taking into account subsequent weight and height gain (model R2 14–39% depending on variable and site). Adjustment for potential environmental confounders did not alter these findings, however, adjustment for maternal BMD markedly reduced the early life associations (particularly for birth weight). Though early life factors were weakly associated with bone mineral apparent density (BMAD, g/cm3) in correlation analysis, subsequent height and weight gain were the only significant independent contributors to BMAD. In conclusion, early life anthropometrics make little contribution to BMAD (other than through their correlation with later growth) but make significant independent contributions to BMD suggesting that the growth trajectory of bone is determined very early in life. In addition, the contribution of body size at birth to bone growth in early life appears to be mediated by genetic factors although it is possible that it may be mediated by poorly measured or as yet unidentified determinants of body size at birth.
      datePublished:2014-02-11T00:00:00Z
      dateModified:2014-02-11T00:00:00Z
      pageStart:304
      pageEnd:308
      sameAs:https://doi.org/10.1007/s002230001148
      keywords:
         Key words: Bone density — Childhood — Growth — Genetics — Body size.
         Biochemistry
         general
         Endocrinology
         Orthopedics
         Cell Biology
      image:
      isPartOf:
         name:Calcified Tissue International
         issn:
            1432-0827
            0171-967X
         volumeNumber:67
         type:
            Periodical
            PublicationVolume
      publisher:
         name:Springer-Verlag
         logo:
            url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
            type:ImageObject
         type:Organization
      author:
            name:G. Jones
            affiliation:
                  name:Menzies Centre for Population Health Research, GPO Box 252-23, Hobart, Tasmania, Australia, 7001
                  address:
                     name:Menzies Centre for Population Health Research, GPO Box 252-23, Hobart, Tasmania, Australia, 7001, Australia
                     type:PostalAddress
                  type:Organization
            type:Person
            name:T. Dwyer
            affiliation:
                  name:Menzies Centre for Population Health Research, GPO Box 252-23, Hobart, Tasmania, Australia, 7001
                  address:
                     name:Menzies Centre for Population Health Research, GPO Box 252-23, Hobart, Tasmania, Australia, 7001, Australia
                     type:PostalAddress
                  type:Organization
            type:Person
      isAccessibleForFree:
      hasPart:
         isAccessibleForFree:
         cssSelector:.main-content
         type:WebPageElement
["Periodical","PublicationVolume"]:
      name:Calcified Tissue International
      issn:
         1432-0827
         0171-967X
      volumeNumber:67
Organization:
      name:Springer-Verlag
      logo:
         url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
         type:ImageObject
      name:Menzies Centre for Population Health Research, GPO Box 252-23, Hobart, Tasmania, Australia, 7001
      address:
         name:Menzies Centre for Population Health Research, GPO Box 252-23, Hobart, Tasmania, Australia, 7001, Australia
         type:PostalAddress
      name:Menzies Centre for Population Health Research, GPO Box 252-23, Hobart, Tasmania, Australia, 7001
      address:
         name:Menzies Centre for Population Health Research, GPO Box 252-23, Hobart, Tasmania, Australia, 7001, Australia
         type:PostalAddress
ImageObject:
      url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
Person:
      name:G. Jones
      affiliation:
            name:Menzies Centre for Population Health Research, GPO Box 252-23, Hobart, Tasmania, Australia, 7001
            address:
               name:Menzies Centre for Population Health Research, GPO Box 252-23, Hobart, Tasmania, Australia, 7001, Australia
               type:PostalAddress
            type:Organization
      name:T. Dwyer
      affiliation:
            name:Menzies Centre for Population Health Research, GPO Box 252-23, Hobart, Tasmania, Australia, 7001
            address:
               name:Menzies Centre for Population Health Research, GPO Box 252-23, Hobart, Tasmania, Australia, 7001, Australia
               type:PostalAddress
            type:Organization
PostalAddress:
      name:Menzies Centre for Population Health Research, GPO Box 252-23, Hobart, Tasmania, Australia, 7001, Australia
      name:Menzies Centre for Population Health Research, GPO Box 252-23, Hobart, Tasmania, Australia, 7001, Australia
WebPageElement:
      isAccessibleForFree:
      cssSelector:.main-content

External Links {🔗}(62)

Analytics and Tracking {📊}

Google Tag Manager

Libraries {📚}

Clipboard.js
Prism.js

Emails and Hosting {✉️}

Mail Servers:

mx.zoho.eu
mx2.zoho.eu
mx3.zoho.eu

Name Servers:

josh.ns.cloudflare.com
zita.ns.cloudflare.com

CDN Services {📦}

Crossref

4.38s.