DOI . ORG {
}
We began analyzing https://link.springer.com/article/10.1007/s00109-006-0133-6, but it redirected us to https://link.springer.com/article/10.1007/s00109-006-0133-6. The analysis below is for the second page.
Title[redir]:
Hypoxia-induced genetic instability—a calculated mechanism underlying tumor progression | Journal of Molecular Medicine
Description:
The cause of human cancers is imputed to the genetic alterations at nucleotide and chromosomal levels of ill-fated cells. It has long been recognized that genetic instability—the hallmark of human cancers—is responsible for the cellular changes that confer progressive transformation on cancerous cells. How cancer cells acquire genetic instability, however, is unclear. We propose that tumor development is a result of expansion and progression—two complementary aspects that collaborate with the tumor microenvironment—hypoxia in particular, on genetic alterations through the induction of genetic instability. In this article, we review the recent literature regarding how hypoxia functionally impairs various DNA repair pathways resulting in genetic instability and discuss the biomedical implications in cancer biology and treatment.
Title[redir]:
Hypoxia-induced genetic instability—a calculated mechanism underlying tumor progression | Journal of Molecular Medicine
Description:
The cause of human cancers is imputed to the genetic alterations at nucleotide and chromosomal levels of ill-fated cells. It has long been recognized that genetic instability—the hallmark of human cancers—is responsible for the cellular changes that confer progressive transformation on cancerous cells. How cancer cells acquire genetic instability, however, is unclear. We propose that tumor development is a result of expansion and progression—two complementary aspects that collaborate with the tumor microenvironment—hypoxia in particular, on genetic alterations through the induction of genetic instability. In this article, we review the recent literature regarding how hypoxia functionally impairs various DNA repair pathways resulting in genetic instability and discuss the biomedical implications in cancer biology and treatment.
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Keywords {🔍}
pubmed, google, scholar, cas, article, cancer, cell, hypoxia, tumor, instability, genetic, huang, cells, dna, repair, factor, hif, hypoxiainducible, biol, glazer, res, mol, human, hifalpha, gene, bindra, usa, kim, nature, natl, acad, sci, response, expression, hypoxiainduced, stress, harris, cellular, hypoxic, mutagenesis, nat, med, proc, ratcliffe, embo, role, privacy, cookies, content, journal,
Topics {✒️}
basic-helix-loop-helix-pas heterodimer regulated ischemia-induced k-ras mutations month download article/chapter double-strand break repair von hippel-lindau protein dna-damaging stress exposures dna-dependent protein kinase hmre11/hrad50 protein complex hypoxia-induced genetic instability hypoxia-induced genetic instability hypoxia-inducible factor 1alpha hypoxia-inducible factor-1alpha o2-dependent degradation domain hypoxia-inducible factor-1 alpha hypoxia-induced replication arrest stimulus-sensitive proline hydroxylation driving genomic instability diminished dna repair genetic instability induced hypoxia-inducible factors retinoblastoma tumor suppressor full article pdf o2-regulated prolyl hydroxylation persistent genetic instability oxygen-dependent proteolysis key regulatory factor hypoxic tumor cells hif-1alpha gene microenvironment-induced mutagenesis tumor microenvironment—hypoxia privacy choices/manage cookies hypoxia-inducible factor 1 hypoxia-inducible factor-1 hypoxia-inducible factor hypoxia inducible factor-1 human rad21 gene aphidicolin-induced arrest hif-1alpha binding hypoxic cancer cells genomic instability common fragile sites murine tumor cells semenza gl dna repair human tumor cells related subjects cancer cells induced cancer biology cellular stress response atr/atm targets
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description:The cause of human cancers is imputed to the genetic alterations at nucleotide and chromosomal levels of ill-fated cells. It has long been recognized that genetic instability—the hallmark of human cancers—is responsible for the cellular changes that confer progressive transformation on cancerous cells. How cancer cells acquire genetic instability, however, is unclear. We propose that tumor development is a result of expansion and progression—two complementary aspects that collaborate with the tumor microenvironment—hypoxia in particular, on genetic alterations through the induction of genetic instability. In this article, we review the recent literature regarding how hypoxia functionally impairs various DNA repair pathways resulting in genetic instability and discuss the biomedical implications in cancer biology and treatment.
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HIF
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Tumor microenvironment
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Molecular Medicine
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description:The cause of human cancers is imputed to the genetic alterations at nucleotide and chromosomal levels of ill-fated cells. It has long been recognized that genetic instability—the hallmark of human cancers—is responsible for the cellular changes that confer progressive transformation on cancerous cells. How cancer cells acquire genetic instability, however, is unclear. We propose that tumor development is a result of expansion and progression—two complementary aspects that collaborate with the tumor microenvironment—hypoxia in particular, on genetic alterations through the induction of genetic instability. In this article, we review the recent literature regarding how hypoxia functionally impairs various DNA repair pathways resulting in genetic instability and discuss the biomedical implications in cancer biology and treatment.
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