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We began analyzing https://link.springer.com/article/10.1007/BF03029778, but it redirected us to https://link.springer.com/article/10.1007/BF03029778. The analysis below is for the second page.

Title[redir]:
Torsades de pointes: Prevention and therapy | Cardiovascular Drugs and Therapy
Description:
Torsades de pointes (TdP) is a life-threatening ventricular tachycardia that occurs in the setting of a prolonged QT interval and is most frequently related to administration of antiarrhythmic drugs. Patients with organic heart disease, with low serum electrolyte levels, with a previous episode of TdP and with bradycardia or baseline QT prolongation may be at increased risk of developing TdP. After initiation of a QT prolonging therapy, the dosage should be modified if the QT interval reaches 560โ€“600 ms. Cessation of medication and immediate hospitalization are indicated in the presence of lightheadedness, syncope, or increased frequency and complexity of ventricular premature beats. The conventional therapy of TdP with isoproterenol or cardiac pacing, although usually effective, has certain disadvantages. Isoproterenol is contraindicated in patients with hypertension or ischemic heart disease, whereas institution of cardiac pacing requires skilled personnel and fluoroscopy. Recently, infusion of magnesium sulfate has been shown to abolish TdP both in the clinical and experimental setting. Compared with conventional therapy, magnesium sulfate has the advantage of safety and simplicity of its administration. In doubtful cases, if does not aggravate a ventricular tachycardia that is not TdP, as may occur with isoproterenol. This advantage and the prompt effectiveness of the drug in four clinical series, including 31 patients, support the use of magnesium sulfate as the first line of therapy for TdP.

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  • Health & Fitness
  • Education
  • Science

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๐ŸŒ  Phenomenal Traffic: 5M - 10M visitors per month


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Keywords {๐Ÿ”}

google, scholar, pubmed, cas, article, ventricular, pointes, cardiol, magnesium, torsade, torsades, therapy, tzivoni, heart, keren, patients, tachycardia, clinical, tdp, sulfate, cardiac, privacy, cookies, content, drugs, access, fibrillation, quinidine, med, data, publish, search, prolongation, syncope, pacing, treatment, atypical, features, coll, long, polymorphous, induced, effect, information, log, journal, research, andre, dan, interval,

Topics {โœ’๏ธ}

open-access database amiodarone-induced ventricular fibrillation month download article/chapter life-threatening ventricular tachycardia disopyramide-induced ventricular fibrillation torsades de pointes long-short initiating sequence torsade de pointes ๏ฟฝtorsade de pointesโ€ polymorphous ventricular tachycardia article cardiovascular drugs atypical ventricular tachycardia full article pdf ventricular tachyarrhythmias induced polymorphopus ventricular tachycardia paroxysmal ventricular fibrillation privacy choices/manage cookies related subjects la tachycardie ventriculaire arrhythmia previously induced article keren baseline qt prolongation ventricular premature beats chronic atrial arrhythmias intractable ventricular tachyarrhythmias unusual ventricular arrhythmia ventricular tachycardia check access instant access prolonged qt interval long qt syndrome long qt syndromes intravenous magnesium sulphate ventricular arrhythmias european economic area frequently related vander ark cr delayed repolarization syndromes torsioni di punta organophosphorus insecticide poisining canine purkinje fibers stimulation rate bikur cholim hospital conditions privacy policy ventricular fibrillation qt prolonging therapy accepting optional cookies article log qt prolongation main content log

Questions {โ“}

  • Drug-Induced QT Prolongation and Torsades de Pointes: An All-Exclusive Relationship or Time for an Amicable Separation?
  • Good, bad or indifferent?

Schema {๐Ÿ—บ๏ธ}

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      headline:Torsades de pointes: Prevention and therapy
      description:Torsades de pointes (TdP) is a life-threatening ventricular tachycardia that occurs in the setting of a prolonged QT interval and is most frequently related to administration of antiarrhythmic drugs. Patients with organic heart disease, with low serum electrolyte levels, with a previous episode of TdP and with bradycardia or baseline QT prolongation may be at increased risk of developing TdP. After initiation of a QT prolonging therapy, the dosage should be modified if the QT interval reaches 560โ€“600 ms. Cessation of medication and immediate hospitalization are indicated in the presence of lightheadedness, syncope, or increased frequency and complexity of ventricular premature beats. The conventional therapy of TdP with isoproterenol or cardiac pacing, although usually effective, has certain disadvantages. Isoproterenol is contraindicated in patients with hypertension or ischemic heart disease, whereas institution of cardiac pacing requires skilled personnel and fluoroscopy. Recently, infusion of magnesium sulfate has been shown to abolish TdP both in the clinical and experimental setting. Compared with conventional therapy, magnesium sulfate has the advantage of safety and simplicity of its administration. In doubtful cases, if does not aggravate a ventricular tachycardia that is not TdP, as may occur with isoproterenol. This advantage and the prompt effectiveness of the drug in four clinical series, including 31 patients, support the use of magnesium sulfate as the first line of therapy for TdP.
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