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We began analyzing https://link.springer.com/article/10.1007/BF00692148, but it redirected us to https://link.springer.com/article/10.1007/BF00692148. The analysis below is for the second page.

Title[redir]:
Status spongiosus of nervous tissue | Acta Neuropathologica
Description:
The diverse nature of status spongiosus of nervous tissue is revealed by electron microscopy in a variety of experimental models. Intracellular, extracellular and intramyelinic accumulation of fluid as well as artefactitious dissolution of myelin may account for spongiform changes. In cerebral edema following trauma the perivascular and perineuronal vacuoles in the cortex correspond to swollen astrocytic processes whereas in the white matter the sponginess mainly reflects enlarged extracellular spaces. Vacuolation of swollen dendrites, termnal axons and perikaryal cytoplasm of nerve cells characterizes the transmissible spongiform encephalopathies. Axonal swelling produced by anoxia accounts for the spongy changes seen in the optic nerve in acute glaucoma. Selective accumulation of fluid within myelin sheaths occurs in isonicotinic acid hydrazide poisoning. Aggregates of irregularly curved, lamellar structures, derived from the artifactitious disintegration of myelin, form grape-like deposits or Buscaino bodies which, when dissolved, appear as vacuoles that may be confused with other spongiform states.

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  • Education
  • Science
  • Social Networks

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πŸ™οΈ Massive Traffic: 50M - 100M visitors per month


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Keywords {πŸ”}

google, scholar, neurol, neuropath, brain, experimental, edema, exp, nervous, cerebral, article, arch, spongy, degeneration, central, system, nature, path, chic, content, acta, status, spongiform, york, springer, tissue, lampert, myelin, access, structure, amer, eds, seitelberger, klatzo, privacy, cookies, research, spongiosus, electron, studies, white, matter, nerve, hydrocephalus, fine, ultrastructural, induced, disease, zimmerman, gajdusek,

Topics {βœ’οΈ}

month download article/chapter familial spongy degeneration experimental spongiform encephalopathy experimental spongy degeneration cerebral spongy degeneration van bogaert-bertrand disease buscaino bodies related subjects electron microscopic studies isonicotinic acid hydrazide transmissible spongiform encephalopathies full article pdf privacy choices/manage cookies experimental spongiform encephalopathies experimental cyanide encephalopathy spongy degeneration article adornato experimental kuru encephalopathy axonal swelling produced high sodium content central nervous system check access instant access nervous tissue maintained spongiform states creutzfeldt-jakob disease experimental obstructive hydrocephalus potassium cyanide solution potassium-rich media status spongiosus cold induced edema european economic area adaptive autophagy reprogramming membrane atpase inhibitor pentose phosphate pathway metachromatic material found cuprous complexes formed excerpta medica found carbon monoxide gas experimental mouse gliomas experimental leukoencephalopathies due conditions privacy policy oxidative enzyme activity cerebral edema induced swollen astrocytic processes experimental globoid cells brain disorders induced experimental cerebral edema accepting optional cookies brain research

Questions {❓}

  • Mucocytes of the brainβ€”Are they artifacts?

Schema {πŸ—ΊοΈ}

WebPage:
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         description:The diverse nature of status spongiosus of nervous tissue is revealed by electron microscopy in a variety of experimental models. Intracellular, extracellular and intramyelinic accumulation of fluid as well as artefactitious dissolution of myelin may account for spongiform changes. In cerebral edema following trauma the perivascular and perineuronal vacuoles in the cortex correspond to swollen astrocytic processes whereas in the white matter the sponginess mainly reflects enlarged extracellular spaces. Vacuolation of swollen dendrites, termnal axons and perikaryal cytoplasm of nerve cells characterizes the transmissible spongiform encephalopathies. Axonal swelling produced by anoxia accounts for the spongy changes seen in the optic nerve in acute glaucoma. Selective accumulation of fluid within myelin sheaths occurs in isonicotinic acid hydrazide poisoning. Aggregates of irregularly curved, lamellar structures, derived from the artifactitious disintegration of myelin, form grape-like deposits or Buscaino bodies which, when dissolved, appear as vacuoles that may be confused with other spongiform states.
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      headline:Status spongiosus of nervous tissue
      description:The diverse nature of status spongiosus of nervous tissue is revealed by electron microscopy in a variety of experimental models. Intracellular, extracellular and intramyelinic accumulation of fluid as well as artefactitious dissolution of myelin may account for spongiform changes. In cerebral edema following trauma the perivascular and perineuronal vacuoles in the cortex correspond to swollen astrocytic processes whereas in the white matter the sponginess mainly reflects enlarged extracellular spaces. Vacuolation of swollen dendrites, termnal axons and perikaryal cytoplasm of nerve cells characterizes the transmissible spongiform encephalopathies. Axonal swelling produced by anoxia accounts for the spongy changes seen in the optic nerve in acute glaucoma. Selective accumulation of fluid within myelin sheaths occurs in isonicotinic acid hydrazide poisoning. Aggregates of irregularly curved, lamellar structures, derived from the artifactitious disintegration of myelin, form grape-like deposits or Buscaino bodies which, when dissolved, appear as vacuoles that may be confused with other spongiform states.
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