DOI . ORG {
}
We began analyzing https://link.springer.com/chapter/10.1007/978-3-030-32656-2_9, but it redirected us to https://link.springer.com/chapter/10.1007/978-3-030-32656-2_9. The analysis below is for the second page.
Title[redir]:
The Bone Microenvironment in Prostate Cancer Metastasis | SpringerLink
Description:
The propensity of prostate cancer cells to seed the skeleton and then progress into clinically relevant metastatic tumors is widely recognized and a major cause of morbidity and mortality for patients. The natural history of prostate adenocarcinoma most frequently...
Title[redir]:
The Bone Microenvironment in Prostate Cancer Metastasis | SpringerLink
Description:
The propensity of prostate cancer cells to seed the skeleton and then progress into clinically relevant metastatic tumors is widely recognized and a major cause of morbidity and mortality for patients. The natural history of prostate adenocarcinoma most frequently...
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Keywords {🔍}
pubmed, google, scholar, cancer, article, cas, prostate, bone, central, cell, metastasis, cells, metastatic, res, metastases, receptor, chapter, rev, nat, stem, tumor, patients, nature, niche, clin, carcinoma, med, growth, fatatis, androgen, hematopoietic, urol, dormancy, mol, mechanisms, molecular, differentiation, oncol, neuroendocrine, medicine, progression, breast, biol, skeletal, treatment, privacy, cookies, content, information, publish,
Topics {✒️}
pubmed google scholar month download article/chapter targeting tumor–stromal interactions extracellular signal-regulated kinase platelet-derived growth factor multi-functional complex organ parathyroid hormone-related protein castration-resistant prostate cancer tartrate-resistant acid phosphatase small-cell carcinoma features ct-guided bone biopsies hormone-sensitive prostate cancer human hematopoietic stem pain-related neurochemical reorganization stem cell quiescence bone-metastatic prostate cancer pre-clinical mouse models gas6 receptor status androgen receptor signaling chemokine receptor cx3cr1 privacy choices/manage cookies human prostate cancer small cell carcinoma bone-metastasis-free survival osteoblastic cells regulate stem cell niche device instant download prostate cancer stem bone cell interactions cancer stem cells osteoblastic metastatic lesions metastatic prostate cancer metastatic prostate cancer editor information editors cell-autonomous features placebo-controlled trial subclonal metastatic expansion prostate cancer cells treat metastatic cancer prostate-specific antigen bone metastatic niche masonic cancer center advanced prostate cancer stage simultaneously presents neuroendocrine prostate cancer cell-autonomous driving cxcr4 antagonist circulating tumour cells cancer cells cooperate chapter dinatale
Questions {❓}
- Farach-Carson, Prostate cancer and neuroendocrine differentiation: more neuronal, less endocrine?
- Poupon, A ‘class action’ against the microenvironment: do cancer cells cooperate in metastasis?
Schema {🗺️}
ScholarlyArticle:
headline:The Bone Microenvironment in Prostate Cancer Metastasis
pageEnd:184
pageStart:171
image:https://media.springernature.com/w153/springer-static/cover/book/978-3-030-32656-2.jpg
genre:
Biomedical and Life Sciences
Biomedical and Life Sciences (R0)
isPartOf:
name:Prostate Cancer
isbn:
978-3-030-32656-2
978-3-030-32655-5
type:Book
publisher:
name:Springer International Publishing
logo:
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type:ImageObject
type:Organization
author:
name:Anthony DiNatale
affiliation:
name:Drexel University College of Medicine
address:
name:Department of Pharmacology and Physiology, Drexel University College of Medicine, Philadelphia, USA
type:PostalAddress
type:Organization
name:Thomas Jefferson University
address:
name:Program in Prostate Cancer, Sidney Kimmel Cancer Center, Thomas Jefferson University, Philadelphia, USA
type:PostalAddress
type:Organization
type:Person
name:Alessandro Fatatis
affiliation:
name:Drexel University College of Medicine
address:
name:Department of Pharmacology and Physiology, Drexel University College of Medicine, Philadelphia, USA
type:PostalAddress
type:Organization
name:Thomas Jefferson University
address:
name:Program in Prostate Cancer, Sidney Kimmel Cancer Center, Thomas Jefferson University, Philadelphia, USA
type:PostalAddress
type:Organization
email:[email protected]
type:Person
keywords:
description:The propensity of prostate cancer cells to seed the skeleton and then progress into clinically relevant metastatic tumors is widely recognized and a major cause of morbidity and mortality for patients. The natural history of prostate adenocarcinoma most frequently begins with a tumor diagnosed at a localized stage, which is successfully treated by surgical and/or radiation therapy modalities. A relevant percentage of patients are clinically cured but approximately 20–30% will develop biochemical signs of recurrence, which respond to the inhibition of androgen receptor (AR) signaling by hormone-deprivation and receptor antagonists, before the inevitable transition into castration-resistant prostate cancer (CRPC). This stage simultaneously presents with or is rapidly followed by secondary tumors, which involve the skeleton in more than 90% of cases (mCRPC). While generalization in clinical practice is always unwise, it is indisputable that bone-metastatic prostate cancer is virtually incurable. Decades of research have revealed that the tissue microenvironment provided by the bone marrow is as important as the cell-autonomous features of tumor cells in fostering the right conditions that lead to establishment and progression of metastatic tumors in the skeleton.
datePublished:2019
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