DOI . ORG {
}
We began analyzing https://link.springer.com/chapter/10.1007/978-1-4614-0106-3_12, but it redirected us to https://link.springer.com/chapter/10.1007/978-1-4614-0106-3_12. The analysis below is for the second page.
Title[redir]:
Nuclear Factor-κB in Immunity and Inflammation: The Treg and Th17 Connection | SpringerLink
Description:
Although nuclear factor-κB (NF-κB) is generally considered to be a pro-inflammatory transcription factor, recent studies indicate that it also plays a critical role in the development of an anti-inflammatory T cell subset called regulatory T (Treg...
Title[redir]:
Nuclear Factor-κB in Immunity and Inflammation: The Treg and Th17 Connection | SpringerLink
Description:
Although nuclear factor-κB (NF-κB) is generally considered to be a pro-inflammatory transcription factor, recent studies indicate that it also plays a critical role in the development of an anti-inflammatory T cell subset called regulatory T (Treg...
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Keywords {🔍}
google, scholar, cas, article, pubmed, cells, regulatory, journal, cell, crel, immunology, foxp, transcription, factor, immunity, chen, medicine, development, nuclear, experimental, nfkappa, expression, nature, liou, chapter, treg, function, role, mice, differentiation, gerondakis, interleukin, grumont, essential, research, inflammation, nfkappab, autoimmune, ruan, access, molecular, lymphocyte, kaltschmidt, immunological, privacy, cookies, content, information, publish, biology,
Topics {✒️}
phorbol ester 12-o-tetradecanoylphorbol-13-acetate retinoid-related orphan receptor-γ granulocyte-macrophage colony-stimulating factor transforming growth factor-beta natural cd4+cd25+foxp3+ regulatory rel/nf-kappab transcription factors month download article/chapter antigen receptor-mediated stimulation n-terminal regulatory domain transcription factor nf-kappab nf-kappab signal pathway nf-kappab signalling pathway nf-kappab-bound substrate peripheral cd4+cd25− naive c-rel-related transcripts il-23-mediated il-17 production transcription factor c-rel pro-inflammatory transcription factor chicken c-rel protooncogene understand rel/nf-kappab murine c-rel transcription rat pancreatic beta-cells dose-dependent cross-talk pro-inflammatory il-17-producing nuclear factor-kappab c-rel -deficient mice transcription factor nf-kappa regulatory t-cell fate mouse c-rel protein cell clones induced cell-mediated immune responses nuclear factor-κb tgf-beta signaling innate immunity ii privacy choices/manage cookies nf-κb target genes amyloid beta peptides pivotal transcription factor transcription factor foxp3 foxp3 transcription factor cell apoptosis nuclear factor kappa nuclear factor-kappa device instant download pro-inflammatory th17 cells chapter current topics cd4+cd25+ regulatory cd4+ cd25+ regulatory tgf-beta induction editor information editors
Schema {🗺️}
ScholarlyArticle:
headline:Nuclear Factor-κB in Immunity and Inflammation: The Treg and Th17 Connection
pageEnd:221
pageStart:207
image:https://media.springernature.com/w153/springer-static/cover/book/978-1-4614-0106-3.jpg
genre:
Biomedical and Life Sciences
Biomedical and Life Sciences (R0)
isPartOf:
name:Current Topics in Innate Immunity II
isbn:
978-1-4614-0106-3
978-1-4614-0105-6
type:Book
publisher:
name:Springer New York
logo:
url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
type:ImageObject
type:Organization
author:
name:Qingguo Ruan
affiliation:
name:University of Pennsylvania School of Medicine
address:
name:Department of Pathology and Laboratory Medicine, 712 Stellar-Chance Laboratories, University of Pennsylvania School of Medicine, Philadelphia, USA
type:PostalAddress
type:Organization
type:Person
name:Youhai H. Chen
affiliation:
name:University of Pennsylvania School of Medicine
address:
name:Department of Pathology and Laboratory Medicine, 712 Stellar-Chance Laboratories, University of Pennsylvania School of Medicine, Philadelphia, USA
type:PostalAddress
type:Organization
email:[email protected]
type:Person
keywords:NF-κB, c-Rel, Inflammation, Th17, Treg
description:Although nuclear factor-κB (NF-κB) is generally considered to be a pro-inflammatory transcription factor, recent studies indicate that it also plays a critical role in the development of an anti-inflammatory T cell subset called regulatory T (Treg
Treg
) cells. Two NF-κB proteins, c-Rel
c-Rel
and p65, drive the development of Treg
Treg
cells by promoting the formation of a Foxp3-specific enhanceosome. Consequently, c-Rel
c-Rel
-deficient mice have marked reductions in Treg
Treg
cells, and c-Rel
c-Rel
-deficient T cells are compromised in Treg
Treg
cell differentiation. However, with the exception of Foxp3, most NF-κB target genes in immune cells are pro-inflammatory. These include several Th17
Th17
-related cytokine genes and the retinoid-related orphan receptor-γ (Rorg or Rorc) that specifies Th17
Th17
differentiation and lineage-specific function. T cells deficient in c-Rel
c-Rel
or p65 are significantly compromised in Th17
Th17
differentiation, and c-Rel
c-Rel
-deficient mice are defective in Th17
Th17
responses. Thus, NF-κB is required for the development of both anti-inflammatory Treg
Treg
and pro-inflammatory Th17
Th17
cells.
datePublished:2012
isAccessibleForFree:
hasPart:
isAccessibleForFree:
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type:WebPageElement
context:https://schema.org
Book:
name:Current Topics in Innate Immunity II
isbn:
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name:Springer New York
logo:
url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
type:ImageObject
name:University of Pennsylvania School of Medicine
address:
name:Department of Pathology and Laboratory Medicine, 712 Stellar-Chance Laboratories, University of Pennsylvania School of Medicine, Philadelphia, USA
type:PostalAddress
name:University of Pennsylvania School of Medicine
address:
name:Department of Pathology and Laboratory Medicine, 712 Stellar-Chance Laboratories, University of Pennsylvania School of Medicine, Philadelphia, USA
type:PostalAddress
ImageObject:
url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
Person:
name:Qingguo Ruan
affiliation:
name:University of Pennsylvania School of Medicine
address:
name:Department of Pathology and Laboratory Medicine, 712 Stellar-Chance Laboratories, University of Pennsylvania School of Medicine, Philadelphia, USA
type:PostalAddress
type:Organization
name:Youhai H. Chen
affiliation:
name:University of Pennsylvania School of Medicine
address:
name:Department of Pathology and Laboratory Medicine, 712 Stellar-Chance Laboratories, University of Pennsylvania School of Medicine, Philadelphia, USA
type:PostalAddress
type:Organization
email:[email protected]
PostalAddress:
name:Department of Pathology and Laboratory Medicine, 712 Stellar-Chance Laboratories, University of Pennsylvania School of Medicine, Philadelphia, USA
name:Department of Pathology and Laboratory Medicine, 712 Stellar-Chance Laboratories, University of Pennsylvania School of Medicine, Philadelphia, USA
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