Here's how NCBI.NLM.NIH.GOV makes money* and how much!

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NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers
  14. CDN Services

We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC9871625/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC9871625/. The analysis below is for the second page.

Title[redir]:
Role of inflammation, immunity, and oxidative stress in hypertension: New insights and potential therapeutic targets - PMC
Description:
Hypertension is regarded as the most prominent risk factor for cardiovascular diseases, which have become a primary cause of death, and recent research has demonstrated that chronic inflammation is involved in the pathogenesis of hypertension. Both ...

Matching Content Categories {📚}

  • Health & Fitness
  • Science
  • Fitness & Wellness

Content Management System {📝}

What CMS is ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 6,398,900 visitors per month in the current month.

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How Does Ncbi.nlm.nih.gov Make Money? {💸}

We see no obvious way the site makes money.

Earning money isn't the goal of every website; some are designed to offer support or promote social causes. People have different reasons for creating websites. This might be one such reason. Ncbi.nlm.nih.gov might have a hidden revenue stream, but it's not something we can detect.

Keywords {🔍}

doi, hypertension, pubmed, google, scholar, cells, blood, pressure, free, pmc, article, vascular, endothelial, inflammation, inflammatory, stress, oxidative, role, immune, patients, ros, activation, system, cytokines, renal, mice, production, hypertensive, injury, activity, activated, effect, cell, angiotensin, macrophages, dysfunction, function, diseases, damage, expression, remodeling, mechanisms, response, nlrp, receptor, evidence, nadph, enos, therapeutic, disease,

Topics {✒️}

2017 acc/aha/aapa/abc/acpm/ags/apha/ash/aspc/nma/pcna guideline pi3k/akt-mapk pathway related m-csf-deficient mice activated renin-angiotensin-aldosterone system pi3k/akt-mapk pathway prevents lysophosphatidylcholine-induced injury brain-gut-bone marrow axis broad-spectrum tetracycline antibiotic increased wall/lumen ratio endothelium-dependent vasoconstrictor factor jak2/stat3-dependent manner b-lymphocyte-deficient mice angiotensin-induced vascular injury central renin-angiotensin system na-k-2cl-cotransporter gp130/jak/stat pathway rho-kinase dependent fashion hypothalamus-pituitary-adrenal axis angiotensin ii-induced hypertension nlr-family pyrin domain pmc beta search ang ii-induced elevation anti-il17ra monoclonal antibody nitro-oxidative stress induced tab funding statement hyperglycemia-induced endothelial dysfunction beta-adrenergic receptor blocker ras-mediated signal pathways monocyte-platelet aggregate formation telmisartan upregulates gtp-cyclohydrolase nadph oxidase-dependent manner oxygen free radicals t-cell-mediated actions il-1r1 receptor inhibitor low-grade inflammatory condition free contraction induced increased firmicutes/bacteroides ratios impaired gut-brain axis blood-brain barrier permeability salt-sensitive dahl rats dahl salt-sensitive rats deoxycorticosterone acetate-salt hypertension mrc-ila heart study impaired blood-brain barrier hypertensive end-organ damage renal ischemia-reperfusion injury prohibited atp-induced hypertension large-scale clinical trials altering t-lymphocyte infiltration lymphocyte-mediated immune responses

Questions {❓}

  • Does immunosuppressant medication lower blood pressure and arterial stiffness in patients with chronic kidney disease?
  • IL-1β and IL-18: Inflammatory markers or mediators of hypertension?
  • Increased serum levels of interleukin-1beta in the systemic circulation of patients with essential hypertension: Additional risk factor for atherogenesis in hypertensive patients?
  • Inflammation, immunity, and oxidative stress in hypertension-partners in crime?
  • Microglia, autonomic nervous system, immunity and hypertension: Is there a link?
  • Multifarious molecular signaling cascades of cardiac hypertrophy: Can the muddy waters be cleared?
  • Nitric oxide synthase inhibition and oxidative stress in cardiovascular diseases: possible therapeutic targets?
  • Preactivated monocytes from hypertensive patients as a factor for atherosclerosis?
  • Reactive oxygen species, vascular oxidative stress, and redox signaling in hypertension: What is the clinical significance?
  • T Lymphocytes: A role in hypertension?

External Links {🔗}(338)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • jQuery
  • jQuery module (jquery-3.6.0)
  • Zoom.js

Emails and Hosting {✉️}

Mail Servers:

  • nihcesxway.hub.nih.gov
  • nihcesxway2.hub.nih.gov
  • nihcesxway3.hub.nih.gov
  • nihcesxway4.hub.nih.gov
  • nihcesxway5.hub.nih.gov

Name Servers:

  • dns1-ncbi.ncbi.nlm.nih.gov
  • dns2-ncbi.ncbi.nlm.nih.gov
  • lhcns1.nlm.nih.gov
  • lhcns2.nlm.nih.gov

CDN Services {📦}

  • Ncbi

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