NCBI . NLM . NIH . GOV {
}
We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC9530026/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC9530026/. The analysis below is for the second page.
Title[redir]:
Erythrocyte mitogen-activated protein kinases mediate hemolytic events under osmotic and oxidative stress and in hemolytic diseases - PMC
Description:
p38 MAPKs are key regulators of cellular adaptation to various stress stimuli, however, their role in mediating erythrocyte cell death and hemolysis is largely unknown. We hypothesized that activation of erythrocyte p38 MAPK is a common event in the ...
Title[redir]:
Erythrocyte mitogen-activated protein kinases mediate hemolytic events under osmotic and oxidative stress and in hemolytic diseases - PMC
Description:
p38 MAPKs are key regulators of cellular adaptation to various stress stimuli, however, their role in mediating erythrocyte cell death and hemolysis is largely unknown. We hypothesized that activation of erythrocyte p38 MAPK is a common event in the ...
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Keywords {🔍}
mapk, erythrocyte, google, scholar, hemolysis, pubmed, cell, doi, erythrocytes, fig, stress, inhibitor, sickle, total, osmotic, mapkapk, pmc, activation, protein, scio, disease, phosphorylation, test, article, human, μmoll, inhibition, inhibitors, expression, signaling, free, rsk, mkk, blood, oxidative, diamide, data, pathways, shock, kinase, study, band, cells, hypoosmotic, phosphorylated, kinases, presence, compared, membrane, role,
Topics {✒️}
p38-regulated/activated protein kinase reds-iii rbc-omics study 129p2-hbb-b1tm1unc hbb-b2tm1unc/ tnf-related apoptosis-induced ligand mitogen-activated protein kinases mapk-activated protein kinases g-protein-coupled receptors rab5-mediated neurodegenerative disease gonadotropin-releasing hormone receptor genome-wide association studies demonstrated p38-mapk-mediated eryptosis mixed-effects anova analysis erythrocyte-programmed cell death ras/mapk signaling cascade mapk-mediated signaling pathways supplementary materials included epinephrine promoted adhesion promotes membrane injury hypoxia-induced erythrocyte dehydration pmc beta search stem cell factor mapk-based therapeutic approaches inhibits tumor growth recent blood donation/transfusion erythropoietin-induced erythroid differentiation mediated γ globin membrane-bound proteins p38 map kinase differentiation stage-specific activation diamide-induced oxidative stress cell-free hemoglobin p38α-mapkapk2 signaling axis knockout-transgenic mouse model testosterone-dependent sex differences sickle β-globin hemoglobin-free ghosts suicidal erythrocyte death washed erythrocyte concentrates osmotic stress-treated cells increased membrane association erk map kinase hypoxia-induced dehydration p38alpha mapk signaling ribosomal s6 kinase osmotic shock-treated erythrocytes [google scholar] [13] vitalant research institute sickle cell disease including kruskal-wallis red blood cells
Questions {❓}
- Son Y, Cheong YK, Kim NH, Chung HT, Kang DG, Pae HO, Mitogen-activated protein kinases and reactive oxygen species: how can ROS activate MAPK pathways?
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External Links {🔗}(124)
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