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cells, qsox, nrf, hcc, doi, egfr, sorafenib, fig, levels, cell, tumor, expression, patients, pubmed, google, scholar, treatment, ferroptosis, signaling, tissues, antioxidant, usa, pmc, article, protein, compared, ros, activation, data, mhcchqsox, free, found, cancer, analysis, capacity, carcinoma, results, overexpression, egf, hepatocellular, death, rab, open, tab, control, knockdown, hepbshqsox, high, redox, nuclear,

Topics {✒️}

antioxidant n-acetyl-l-cysteine small-molecule multi-kinase inhibitors pleno-dce-puro vector cox-mantel log-rank test pleno-dce-puro-vehicle n-acetyl-l-cysteine p62-keap1-nrf2 pathway protects s-adenosyl homocysteine hydrolase ligand-induced endosomal transfer ligand-induced lysosomal degradation high-rank gene sets oxidative stress-induced damage p62/sqstm1 promotes malignancy formula length × width2 × π/6 pmc beta search goat anti-rabbit igg post-transcriptional control mechanism nrf2-dependent metabolic reprogramming small-cell lung cancers line molecular-target drug log2-transformed fold change nrf2-mediated cell proliferation promote sorafenib-induced ferroptosis promoting ubiquitination-mediated degradation egf-induced egfr signaling promoting sorafenib-induced ferroptosis sorafenib-induced lipid peroxidation ros-modulated coordinated regulation sorafenib-induced cell death canonical keap1-mediated regulation iron-dependent cell death epidermal growth factor drug-resistant mechanisms underlying hep3b/shqsox1 exhibited egfr-dependent tumor types publisher site pdf cancer-related processes hep3b/shqsox1 cell lines cancer-acquired drug resistance egfr/nrf2 signaling pathway hcv-positive hepatocellular carcinoma stimulate mhcc97h/qsox1 cells pro-oncogenic gene reactive oxygen species nadp+/nadph assay kit serum-free medium c-kit signaling resulting master transcriptional factor enhance sorafenib-induced ferroptosis hep3b/shqsox1 cells showed

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• Targeting cancer cells by ROS-mediated mechanisms: a radical therapeutic approach?

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