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NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Social Networks
  9. External Links
  10. Analytics And Tracking
  11. Libraries
  12. Hosting Providers
  13. CDN Services

We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC7393508/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC7393508/. The analysis below is for the second page.

Title[redir]:
Auranofin mitigates systemic iron overload and induces ferroptosis via distinct mechanisms - PMC
Description:
Iron homeostasis is essential for health; moreover, hepcidin-deficiency results in iron overload in both hereditary hemochromatosis and iron-loading anemia. Here, we identified iron modulators by functionally screening hepcidin agonists using a ...

Matching Content Categories {📚}

  • Education
  • Science
  • Health & Fitness

Content Management System {📝}

What CMS is ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 6,398,976 visitors per month in the current month.

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How Does Ncbi.nlm.nih.gov Make Money? {💸}

We don’t know how the website earns money.

Not every website is profit-driven; some are created to spread information or serve as an online presence. Websites can be made for many reasons. This could be one of them. Ncbi.nlm.nih.gov could be secretly minting cash, but we can't detect the process.

Keywords {🔍}

aur, doi, hepcidin, iron, mice, expression, fig, cells, pubmed, google, scholar, mrna, ferroptosis, hepatic, hfe, pmc, article, huh, levels, cell, free, male, found, treatment, effect, measured, treated, female, hamp, mgkg, significantly, supplementary, data, highdose, txnrd, metabolism, serum, activity, inhibitor, pathway, liver, drug, thioredoxin, increased, body, blood, auranofin, drugs, signaling, estrogen,

Topics {✒️}

nf-κb/il-6/stat3 signaling pathway 5 mg/kg body weight/day apoptosis inhibitor z-vad-fmk specific pathogen-free facility high-dose aur‒induced decrease fda-approved anti-rheumatoid arthritis nf-κb/il-6/stat3 axis hepcidin-deficiency related disorders mapk/erk signaling pathway 5 mg/kg body weight 25 mg/kg body weight 1 mg/kg body weight 10 mg/kg body weight kaplan–meier survival curves author contributions pmc beta search gold-induced aplastic anemia high-dose aur showed treating iron-related diseases nf-κb inhibitor bay11–7082 high-dose aur increased induce il-6/hepcidin signaling gpr30-bmp6-dependent signaling tumor necrosis factor-alpha bmp6/bmp receptor complex tfr2/hfe complex triggers prevent hydroperoxide-induced ferroptosis recently identified iron-dependent long-term aur treatment license information pmcid myd88-dependent il-6 production transferrin-induced hepcidin expression nf-κb/il-6 pathway downstream pro-inflammatory cytokines perls’ prussian blue hepatic tnf-α expression high doses—inducing ferroptosis bmp/smad/hepcidin axis increased phospho-stat3 levels splenic tnf-α expression male c57bl/6j mice hepcidin-deficient mouse model female wild-type c57bl/6 master iron-regulating hormone lipid peroxidation-dependent form aur-regulated iron parameters iron-overload murine models il-1β mrna levels anti-ra drug auranofin canonical hepcidin-regulating pathways

External Links {🔗}(112)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • jQuery
  • jQuery module (jquery-3.6.0)
  • Zoom.js

Emails and Hosting {✉️}

Mail Servers:

  • nihcesxway.hub.nih.gov
  • nihcesxway2.hub.nih.gov
  • nihcesxway3.hub.nih.gov
  • nihcesxway4.hub.nih.gov
  • nihcesxway5.hub.nih.gov

Name Servers:

  • dns1-ncbi.ncbi.nlm.nih.gov
  • dns2-ncbi.ncbi.nlm.nih.gov
  • lhcns1.nlm.nih.gov
  • lhcns2.nlm.nih.gov

CDN Services {📦}

  • Ncbi

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