
NCBI . NLM . NIH . GOV {
}
Title[redir]:
Lycopene Inhibits Epithelial–Mesenchymal Transition and Promotes Apoptosis in Oral Cancer via PI3K/AKT/m-TOR Signal Pathway - PMC
Description:
Oral cancer (OC) is one of the most common cancers around the world. Despite the progress in treatment, the prognosis of OC remains poor, especially for patients with advanced diseases. It urges the development of novel therapeutic options against ...
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Keywords {🔍}
doi, lyc, cancer, cells, google, scholar, cell, pubmed, lycopene, apoptosis, assay, emt, pathway, pmc, oral, pikaktmtor, treatment, tumor, signaling, figure, group, article, mice, free, proliferation, anticancer, manner, human, control, xenograft, dosedependent, cal, results, inhibited, growth, progression, migration, ecadherin, study, compared, concentrations, treated, nlm, inhibits, therapeutic, process, effects, scc, colony, formation,
Topics {✒️}
pi3k/akt/m-tor signaling pathway pi3k/akt/mtor-mediated pathways pi3k/akt/mtor signaling pathway pi3k-akt-mtor signaling pathway pi3k/akt/m-tor signaling pi3k/akt/mtor pathway alterations p-m-tor/m-tor wnt/β‐catenin signaling pathway pi3k/akt/mtor pathway anti-p-m-tor enhanced gap-junctional communication enhances gap-junction communication pmc beta search akt signaling pathway serum-free rpmi-1640 medium inhibit epithelial–mesenchymal transition investigating naturally-occurring substances effective anti-carcinogenic agent anti-m-tor n-cadherin promotes motility ice-cold ripa buffer pan-cancer proteogenomic atlas exert anti-cancer effects p-m-tor serum-free media lyc dose-dependently suppressed p-pi3k/pi3k low e-cadherin expression p-akt/akt free radic res high n-cadherin expression wnt/β-catenin publisher site pdf e-cadherin/n-cadherin multiple signaling pathways chemo-luminescent signal anti-n-cadherin antibodies epithelial–mesenchymal transition e-cadherin promotes metastasis anti-p-pi3k anti-p-akt low-dose lyc group /akt/mammalian target concentration-dependent manner compared epithelial-mesenchymal transitions plumbagin induces g2/ m-tor reactive oxygen species dose-dependently decreased license information pmcid
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