NCBI . NLM . NIH . GOV {
}
We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC7260024/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC7260024/. The analysis below is for the second page.
Title[redir]:
B cellāintrinsic TLR9 expression is protective in murine lupus - PMC
Description:
Toll-like receptor 9 (TLR9) is a regulator of disease pathogenesis in systemic lupus erythematosus (SLE). Why TLR9 represses disease while TLR7 and MyD88 have the opposite effect remains undefined. To begin to address this question, we created 2 ...
Title[redir]:
B cellāintrinsic TLR9 expression is protective in murine lupus - PMC
Description:
Toll-like receptor 9 (TLR9) is a regulator of disease pathogenesis in systemic lupus erythematosus (SLE). Why TLR9 represses disease while TLR7 and MyD88 have the opposite effect remains undefined. To begin to address this question, we created 2 ...
Matching Content Categories {š}
- Science
- Education
- Health & Fitness
Content Management System {š}
What CMS is ncbi.nlm.nih.gov built with?
Custom-built
No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.
Traffic Estimate {š}
What is the average monthly size of ncbi.nlm.nih.gov audience?
šš Tremendous Traffic: 10M - 20M visitors per month
Based on our best estimate, this website will receive around 11,857,728 visitors per month in the current month.
check SE Ranking
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How Does Ncbi.nlm.nih.gov Make Money? {šø}
We see no obvious way the site makes money.
While profit motivates many websites, others exist to inspire, entertain, or provide valuable resources. Websites have a variety of goals. And this might be one of them. Ncbi.nlm.nih.gov could be secretly minting cash, but we can't detect the process.
Keywords {š}
tlr, doi, mice, cells, disease, pubmed, google, scholar, cell, lupus, pmc, figure, article, expression, cdcre, free, tlrflfl, sle, deletion, mrllpr, data, overexpression, immunol, antibodies, pathogenesis, rosatlr, tolllike, cellspecific, role, neutrophils, allele, systemic, nephritis, lineages, model, renal, receptor, effects, deficiency, loss, supplemental, protective, murine, antibody, lines, cre, control, antinucleosome, autoantibody, models,
Topics {āļø}
cd19-creānegative littermates served cd19-cre tlr9fl/fl mice cd11c-cre tlr9fl/fl mice cd19-cre myd88fl/fl mice mrp8-cre tlr9fl/fl mice lysm-cre tlr9fl/fl mice tlr9fl/fl mrl-mpj-faslpr/ cd19-cre mrl/lpr mice cd19-cre tlr9fl/fl myd88-deficient lupus-prone mice cd19-cre tlr9-depleted mice mrl/lpr lupus-prone background cellāspecific tlr9-overexpressing mice anti-double-stranded dna antibodies endocytosis-free dna sensing transgenic tlr9-gfp mice ammonium-chloride-potassium buffer antiānucleosome/antiārna antibodies mrl/lpr wt mice wt mrl/lpr mice supplementary materials included fine-speckled staining indicative diseased mrl/lpr mice cd11c-cre depleted greater cd19-cre rosatlr9 mice pmc beta search lysm-cre tlr9flfl mice cell typeāspecific roles cre-negative rosatlr9 mice tlr9-dependent checkpoint governs rosa-eyfp reporter mice autoimmune-prone genetic backgrounds antiādouble stranded dna cellāspecific tlr9 deficiency igg anti-dsdna antibodies extinguishing antiānucleosome antibodies myeloid-cre deleting strains fas-intact mrl model mrl/lpr mouse model cre-negative rosatlr9 fcgr2bā/ chromosomeālinked autoimmune accelerator cell typeāspecific effects cell-specific tlr9 trafficking antiānucleosome antibody titers fas-sufficient mrl mice pmc copyright notice antiāchromatin antibody response myeloid-specific tlr9 deficiency cellāspecific tlr9 deletion tlr9fl/fl genotype
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