NCBI . NLM . NIH . GOV {
}
We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC6235890/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC6235890/. The analysis below is for the second page.
Title[redir]:
Mettl3-mediated m6A RNA methylation regulates the fate of bone marrow mesenchymal stem cells and osteoporosis - PMC
Description:
N6-methyladenosine (m6A) is the most abundant epigenetic modification in eukaryotic mRNAs and is essential for multiple RNA processing events during mammalian development and disease control. Here we show that conditional knockout of the m6A ...
Title[redir]:
Mettl3-mediated m6A RNA methylation regulates the fate of bone marrow mesenchymal stem cells and osteoporosis - PMC
Description:
N6-methyladenosine (m6A) is the most abundant epigenetic modification in eukaryotic mRNAs and is essential for multiple RNA processing events during mammalian development and disease control. Here we show that conditional knockout of the m6A ...
Matching Content Categories {π}
- Education
- Science
- Health & Fitness
Content Management System {π}
What CMS is ncbi.nlm.nih.gov built with?
Custom-built
No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.
Traffic Estimate {π}
What is the average monthly size of ncbi.nlm.nih.gov audience?
π Phenomenal Traffic: 5M - 10M visitors per month
Based on our best estimate, this website will receive around 6,398,900 visitors per month in the current month.
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How Does Ncbi.nlm.nih.gov Make Money? {πΈ}
We find it hard to spot revenue streams.
Some websites aren't about earning revenue; they're built to connect communities or raise awareness. There are numerous motivations behind creating websites. This might be one of them. Ncbi.nlm.nih.gov could be getting rich in stealth mode, or the way it's monetizing isn't detectable.
Keywords {π}
doi, bone, mettl, mice, pubmed, google, scholar, mscs, pmc, fig, article, free, pthr, marrow, cell, cells, pth, differentiation, mrna, data, staining, rna, supplementary, analysis, osteogenic, translation, knockout, prxcremettlflfl, adipogenic, nature, parathyroid, osteoporosis, expression, performed, signaling, stem, control, distal, conditional, hormone, femurs, scale, bar, analyses, mettlflfl, receptor, controls, treatment, gene, nmethyladenosine,
Topics {βοΈ}
rabbit anti-mettl3/mt-a70 author contributions specific-pathogen-free conditions osterix-expressing mesenchymal progenitors rna n6-methyladenosine methylation n6-methyladenosine binding protein mettl3-mediated m6a methylation estrogen deficiency-induced osteoporosis rna n6-methyladenosine methyltransferase rabbit polyclonal anti-fabp4 parathyroid hormone-related peptide free n6-methyladenosine parathyroid hormone-related protein n6-methyladenosine-modified rna oligo-attached magnetic beads wnt/Ξ²-catenin signalling extracellular signal-regulated kinases mis-regulated m6a modification n6-methyladenosine-dependent regulation cfx96 real-time system pth-mediated bone remodeling pmc beta search wild-type band reverse lc-ms/ms data lc-ms/ms analysis mettl3-mettl14 complex footnotes publisher' activating bone-lining cells pth-regulated gene list data availability statements 50βΞΌg/ml ascorbic acid pth1rfl/fl mice reported pth-induced camp accumulation wild-type band forward mettl3-deficient mscs underwent il-7/stat5/socs pathways bone micro-architecture lead methods generation ad-pth1r successfully restored license information pmcid fda-approved anabolic drugs a-dependent translation control msc m6a merip-seq post hoc test phospho-creb rabbit mab mettl3ki/ki female mice regulates mammalian spermatogenesis pth-regulated gene expression mettl3fl/fl mice whilst quantitative rt-pcr showed
Questions {β}
- Mechanisms of disease: is osteoporosis the obesity of bone?
- Whatβs the matter with MAT?
Social Networks {π}(6)
External Links {π}(150)
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- https://doi.org/10.1016/j.molcel.2012.10.015's revenue stream
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- https://scholar.google.com/scholar_lookup?journal=Bone&title=Molecular%20and%20cellular%20mechanisms%20of%20the%20anabolic%20effect%20of%20intermittent%20PTH&author=RL%20Jilka&volume=40&publication_year=2007&pages=1434-1446&pmid=17517365&doi=10.1016/j.bone.2007.03.017&'s financial summary
- Explore the financials of https://doi.org/10.1074/jbc.M513380200
- How much profit does https://scholar.google.com/scholar_lookup?journal=J.%20Biol.%20Chem.&title=Distinct%20beta-arrestin-%20and%20G%20protein-dependent%20pathways%20for%20parathyroid%20hormone%20receptor-stimulated%20ERK1/2%20activation&author=D%20Gesty-Palmer&volume=281&publication_year=2006&pages=10856-10864&pmid=16492667&doi=10.1074/jbc.M513380200& make?
- Income figures for https://doi.org/10.1016/j.bone.2006.06.010
- Discover the revenue of https://scholar.google.com/scholar_lookup?journal=Bone&title=Intermittent%20treatment%20with%20parathyroid%20hormone%20(PTH)%20as%20well%20as%20a%20non-peptide%20small%20molecule%20agonist%20of%20the%20PTH1%20receptor%20inhibits%20adipocyte%20differentiation%20in%20human%20bone%20marrow%20stromal%20cells&author=DJ%20Rickard&volume=39&publication_year=2006&pages=1361-1372&pmid=16904389&doi=10.1016/j.bone.2006.06.010&
- What's the financial gain of https://doi.org/10.1038/s41593-017-0057-1?
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