Here's how NCBI.NLM.NIH.GOV makes money* and how much!

*Please read our disclaimer before using our estimates.
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NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Social Networks
  9. External Links
  10. Analytics And Tracking
  11. Libraries
  12. Hosting Providers
  13. CDN Services

We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC6223858/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC6223858/. The analysis below is for the second page.

Title[redir]:
Efferocytosis in the tumor microenvironment - PMC
Description:
Within the course of a single minute, millions of cells in the human body will undergo programmed cell death in response to physiological or pathological cues. The diminished energetic capacity of an apoptotic cell renders the cell incapable of ...

Matching Content Categories {📚}

  • Science
  • Education
  • Health & Fitness

Content Management System {📝}

What CMS is ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Ncbi.nlm.nih.gov Make Money? {💸}

We don't see any clear sign of profit-making.

Many websites are intended to earn money, but some serve to share ideas or build connections. Websites exist for all kinds of purposes. This might be one of them. Ncbi.nlm.nih.gov could be secretly minting cash, but we can't detect the process.

Keywords {🔍}

doi, cell, cells, pubmed, google, scholar, efferocytosis, apoptotic, tumor, pmc, article, free, cancer, mertk, phagocytes, death, dying, tissue, mammary, microenvironment, ptdser, clearance, inflammation, macrophages, apoptosis, gland, progression, receptor, nature, phagocytosis, process, necrosis, gas, protein, biol, engulfment, tumors, signaling, phagocyte, tyrosine, secondary, macrophage, breast, involution, expression, immunity, antiinflammatory, tim, res, membrane,

Topics {✒️}

c1q–calreticulin–phosphatidylserine interactions yield antigen-dependent anti-tumor immunity anti-inflammatory cytokine-encoding genes efferocytosis-induced anti-inflammatory cytokines wild-type bone marrow post-lactational involution removes peroxisome proliferator-activated receptor post-partum mammary gland p63/p73 target gene monoclonal anti-phosphatidylserine antibody anti-inflammatory phenotype generated apoptotic human t-cells triple-negative breast cancers specific anti-migratory effects pmc beta search apoptotic jurkat cells—existence highly pro-inflammatory event phosphatidylserine-dependent homeostatic response wound-healing signaling events inflammation-resolving cytokines produced ligand-activated lxrα/pparγ activate small-cell lung cancer elmo/dock180/rac module membrane-encapsulated dying cell efferocytosis-induced tumor progression resultant anti-inflammatory response epithelial cell-directed efferocytosis integrin-activating protein expressed fully immune-competent mice produce anti-inflammatory cytokines unconventional rac-gef activity alphavbeta5 integrin-mediated phagocytosis autophagy-related proteins ulk1 pro-tumorogenic effect primarily postpartum mammary gland mertk-gas6 bridging combinations mertk small-molecule inhibitor mitochondrial fission influences mertk-mediated efferocytosis promotes mitochondrial fission promotes inflammation-induced tissue damage receptor tyrosine kinase pro-inflammatory damps released complete apoptotic program mertk-related rtks axl pro-tolerogenic tumor microenvironment human primary fibroblasts ptdser-binding molecules expressed lymphoblastic leukemia/lymphoma post-lactational involution [11

Social Networks {👍}(6)

External Links {🔗}(208)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • jQuery
  • jQuery module (jquery-3.6.0)
  • Zoom.js

Emails and Hosting {✉️}

Mail Servers:

  • nihcesxway.hub.nih.gov
  • nihcesxway2.hub.nih.gov
  • nihcesxway3.hub.nih.gov
  • nihcesxway4.hub.nih.gov
  • nihcesxway5.hub.nih.gov

Name Servers:

  • dns1-ncbi.ncbi.nlm.nih.gov
  • dns2-ncbi.ncbi.nlm.nih.gov
  • lhcns1.nlm.nih.gov
  • lhcns2.nlm.nih.gov

CDN Services {📦}

  • Ncbi

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