NCBI . NLM . NIH . GOV {
}
We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC5983341/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC5983341/. The analysis below is for the second page.
Title[redir]:
Blocking IFNAR1 inhibits multiple myeloma–driven Treg expansion and immunosuppression - PMC
Description:
Despite significant advances in the treatment of multiple myeloma (MM), most patients succumb to disease progression. One of the major immunosuppressive mechanisms that is believed to play a role in myeloma progression is the expansion of regulatory ...
Title[redir]:
Blocking IFNAR1 inhibits multiple myeloma–driven Treg expansion and immunosuppression - PMC
Description:
Despite significant advances in the treatment of multiple myeloma (MM), most patients succumb to disease progression. One of the major immunosuppressive mechanisms that is believed to play a role in myeloma progression is the expansion of regulatory ...
Matching Content Categories {📚}
- Science
- Education
- Health & Fitness
Content Management System {📝}
What CMS is ncbi.nlm.nih.gov built with?
Custom-built
No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.
Traffic Estimate {📈}
What is the average monthly size of ncbi.nlm.nih.gov audience?
🌠 Phenomenal Traffic: 5M - 10M visitors per month
Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
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How Does Ncbi.nlm.nih.gov Make Money? {💸}
We can't figure out the monetization strategy.
Many websites are intended to earn money, but some serve to share ideas or build connections. Websites exist for all kinds of purposes. This might be one of them. Ncbi.nlm.nih.gov could be getting rich in stealth mode, or the way it's monetizing isn't detectable.
Keywords {🔍}
tregs, cells, doi, mice, treg, compared, figure, expression, pubmed, google, scholar, control, myeloma, cell, immune, type, patients, progression, vkmyc, pmc, significant, data, regulatory, vkmycinjected, article, survival, tumor, ifn, free, group, analysis, expansion, microenvironment, injected, email, checkpoint, nontregs, significantly, shown, multiple, human, supplemental, increase, observed, cdcd, gene, cancer, function, signaling, performed,
Topics {✒️}
cd4+cd25+foxp3+cd127–/lo tregs vkmyc-injected mice demonstrates dana-farber cancer institute flow-sorting cd4+cd25+ cells ifn-α/β signaling pathway rag2-ko immune-deficient mice vkmyc-injected dereg mice pmc beta search vkmyc-injected mice compared vkmyc-injected bm cd4+ cd4+cd25+foxp3+ treg compartment ifn–induced biologic responses regulating treg-mediated immunosuppression favourable treg/th17 balance adaptor triggering rig-i transplantable vkmyc cells injected sorted wild-type supplementary materials included immune checkpoint receptor–expressing inf-α homologous genes 5tgm1-injected c57bl/kalwrij mice ifn-stimulated 15-kda protein ip-10/cxcl10 attracts regulatory anti–murine cd25 mab vkmyc cell injection vkmyc-injected mice vkmyc injected mice coculturing vkmyc cells antigen-presenting cell-independent t-regulatory cell functions ifnar1-blocking antibody treatment pmc copyright notice inducible regulatory t-cells single-cell mass cytometry cd138-expressing plasma cells vkmyc-injected bm rna-seq transcript abundances prevents/delays tumor progression anti-cd25 antibody treatment anti-ifnar1 antibody led prevent cell-cell interactions ifn–dependent immune responses received wild-type tregs cd138-enriched plasma cells post-germinal center malignancies cell-cell contact-dependent control versus vkmyc spanning-tree progression analysis ccr7-chemokine receptor expression myeloma-promoting treg deregulation
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