NCBI . NLM . NIH . GOV {
}
We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC5420102/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC5420102/. The analysis below is for the second page.
Title[redir]:
Dipeptidyl peptidase-4 (DPP-4) inhibition with linagliptin reduces western diet-induced myocardial TRAF3IP2 expression, inflammation and fibrosis in female mice - PMC
Description:
Diastolic dysfunction (DD), a hallmark of obesity and primary defect in heart failure with preserved ejection fraction, is a predictor of future cardiovascular events. We previously reported that linagliptin, a dipeptidyl peptidase-4 inhibitor, ...
Title[redir]:
Dipeptidyl peptidase-4 (DPP-4) inhibition with linagliptin reduces western diet-induced myocardial TRAF3IP2 expression, inflammation and fibrosis in female mice - PMC
Description:
Diastolic dysfunction (DD), a hallmark of obesity and primary defect in heart failure with preserved ejection fraction, is a predictor of future cardiovascular events. We previously reported that linagliptin, a dipeptidyl peptidase-4 inhibitor, ...
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- Health & Fitness
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- Fitness & Wellness
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Custom-built
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What is the average monthly size of ncbi.nlm.nih.gov audience?
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Keywords {🔍}
doi, linagliptin, expression, pubmed, google, scholar, cardiac, trafip, fig, mice, heart, pmc, fibrosis, article, myocardial, free, dpp, activation, oxidative, wdinduced, dysfunction, stress, diastolic, increased, group, inhibited, western, inflammation, obesity, diabetes, protein, fed, assay, inhibition, levels, effects, results, collagen, insulin, inflammatory, aroor, diet, wdfed, migration, bar, data, email, female, cardiovascular, type,
Topics {✒️}
isovolumic contraction time isovolumic relaxation time alpha-smooth muscle actin stress-activated protein kinase mda/4-hne levels lysyl oxidase myocardial performance index interleukin-10 released macrophage chemoattractant dipeptidylpeptidase inhibition col iα col iiiα1 intermyofibrillar mitochondria gapdh 4-hydroxynonenal cmc growth factor expression aldosterone-induced cardiomyocyte growth imf mt nuclear factor-kappab inhibitor control diet inappropriate renin-angiotensin-aldosterone system nf-kappa b-activating protein p-38 mapk activation renin-angiotensin-aldosterone system total p-65 inflammatory response p-38 mapk cardiovascular disease nadph oxidase activity ivrt activated p-65 response control intermyofibrillar imf increased mda/4hne levels impaired diastolic relaxation pmc beta search primary defect nox2-dependent cardiomyocyte hypertrophy aldosterone-induced nf-κb activation il-10 protein expression cardiomyocyte cross-sectional area elevated mpi threonine/serine kinase mpi disease collagen cross-linking
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