NCBI . NLM . NIH . GOV {
}
We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC5107943/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC5107943/. The analysis below is for the second page.
Title[redir]:
Didox and resveratrol sensitize colorectal cancer cells to doxorubicin via activating apoptosis and ameliorating P-glycoprotein activity - PMC
Description:
Doxorubicin (DOX) has limited efficacy in colorectal cancer due to multi-drug resistance. Resveratrol (RES) and didox (DID) are polyhydroxyphenols with potential chemosensitizing effects. Herein, we assessed the chemomodulatory effects of RES and ...
Title[redir]:
Didox and resveratrol sensitize colorectal cancer cells to doxorubicin via activating apoptosis and ameliorating P-glycoprotein activity - PMC
Description:
Doxorubicin (DOX) has limited efficacy in colorectal cancer due to multi-drug resistance. Resveratrol (RES) and didox (DID) are polyhydroxyphenols with potential chemosensitizing effects. Herein, we assessed the chemomodulatory effects of RES and ...
Matching Content Categories {📚}
- Science
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Content Management System {📝}
What CMS is ncbi.nlm.nih.gov built with?
Custom-built
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Traffic Estimate {📈}
What is the average monthly size of ncbi.nlm.nih.gov audience?
🌠 Phenomenal Traffic: 5M - 10M visitors per month
Based on our best estimate, this website will receive around 6,398,976 visitors per month in the current month.
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How Does Ncbi.nlm.nih.gov Make Money? {💸}
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Keywords {🔍}
dox, res, cells, doi, cell, pubmed, google, scholar, cancer, hct, combination, significantly, fig, gene, expression, pgp, effect, apoptosis, activity, treatment, compared, resveratrol, concentration, cytotoxicity, control, free, pmc, caspase, lines, significant, sphase, population, cycle, colorectal, article, induced, didox, doxorubicin, arrest, showed, cytotoxic, effects, decreased, mdr, bax, increased, intracellular, increase, bclxl, reductase,
Topics {✒️}
reverse primer tgg-gtg-tcc-caa-agt-agg-ag reverse primer ctg-aag-agc-tcc-tcc-acc-ac reverse primer ggt-acc-tgc-aaa-ctc-tga-gca forward primer tgc-acc-acc-aac-tgc-ttag reverse primer ctg-agt-caggcc-ctt-ctg-tc reverse primer gat-gca-ggg-atg-atg-ttc67 mole/μg p-gp molecules g0/g1-phase cell population oxygen-derived free radicals p-glycoprotein-mediated multidrug resistance g2/m-phase cell population p-gp inhibitory activity p-gp efflux pump ameliorating p-glycoprotein activity age-related bone loss g2/m-phase population compared p-gp pumping activity pmc beta search energy-dependent transporter pump short-term hematopoietic toxicity human breast carcinoma positive p-gp inhibitor standard p-gp blocker apolipoprotein e-deficient mice potential tumor chemo-sensitizers p-gp-inhibition induced covalent p-gp inhibitor strong s-phase arrest suppresses pro-inflammatory profiles s-phase arrest induced direct pro-apoptotic function potential anti-hiv-1 activity cytotoxic s-phase delay pgp-glo™ assay buffer trail-receptor-mediated apoptosis g2/m-phase population s-phase specific agents g2/m-phase arrest62 increased s-phase population biotin-zvkd-fmk inhibitor atm/p53-dependent pathway cell cycle-dependent cytotoxicity pgp-glo™ assay systems g0/g1-phase compared p-glycoprotein pump gbc-sd cell line doxorubicin-induced growth inhibition showed anti-tumor effects potentiating dox-induced cytotoxicity28 antagonize dox-induced cytotoxicity
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