NCBI . NLM . NIH . GOV {
}
We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC4671820/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC4671820/. The analysis below is for the second page.
Title[redir]:
CD5L/AIM regulates lipid biosythesis and restrains Th17 cell pathogenicity - PMC
Description:
Th17 cells play a critical role in host defense against extracellular pathogens and tissue homeostasis, but can induce autoimmunity. The mechanisms implicated in balancing ‘pathogenic’ and ‘non-pathogenic’ Th17 cell states remain largely unknown. We ...
Title[redir]:
CD5L/AIM regulates lipid biosythesis and restrains Th17 cell pathogenicity - PMC
Description:
Th17 cells play a critical role in host defense against extracellular pathogens and tissue homeostasis, but can induce autoimmunity. The mechanisms implicated in balancing ‘pathogenic’ and ‘non-pathogenic’ Th17 cell states remain largely unknown. We ...
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Keywords {🔍}
cells, cdl, doi, figure, cell, rorγt, expression, pubmed, google, scholar, nonpathogenic, pathogenic, pmc, article, free, pufa, pathogenicity, mice, function, differentiation, differentiated, nature, acid, tgfβil, vivo, eae, signature, naïve, proinflammatory, genes, lipid, expressed, fatty, regulate, sfa, ligand, binding, ilr, module, singlecell, regulates, regulator, cholesterol, analyzed, analysis, data, functional, effector, vitro, cns,
Topics {✒️}
single-cell rna-seq profiles scavenger receptor cysteine-rich single-cell rna-seq analysis single-cell rna-seq data double-blind placebo-controlled trial single-cell rna-seq anti-interleukin-17-receptor antibody full-length mrna-seq pmc beta search t-cell receptor specific acetyl-coa carboxylase influences processed rna-seq samples unsorted single-cell analysis free fatty acids luxol fast blue-hematoxylin cd4+cd62l+cd44−cd25− cd4+cd62l−cd44+rorγtgfp+ myelin oligodendrocyte glycoprotein-specific regulate th17-cell function single-cell based proinflammatory antagonize il-23-driven pathogenicity il-17a precedes development il-23rgfp reporter mice de novo synthesis cd3+cd4+il-17a il-1β+il-6+il-23 multiple sclerosis society pro-inflammatory cytokine il-23 previously defined pro-inflammatory detect free fa macrophage-derived aim collagen-induced arthritis model disease-inducing th17 cells aureus-specific th17 cells pro-inflammatory signature genes peripheral lymphoid organs human multiple sclerosis tgf-β3+il-6+il-23 specific commensal bacteria supplementary materials included tgf-β1+il-6+il-23 population-level genomic profiles albicans-specific th17 cells rorγt-ligand dependent mechanism experimental autoimmune encephalomyelitis central nervous system cd5l-rv 2d2 recipients rescue cd5l-induced suppression hypoxia-inducible factor 1 suppress rorγt-dependent transcription
Questions {❓}
- How does CD5L regulate Th17 cell pathogenicity?
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