Here's how NCBI.NLM.NIH.GOV makes money* and how much!

*Please read our disclaimer before using our estimates.
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NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers
  14. CDN Services

We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC4661850/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC4661850/. The analysis below is for the second page.

Title[redir]:
Therapeutic Implications for Overcoming Radiation Resistance in Cancer Therapy - PMC
Description:
Ionizing radiation (IR), such as X-rays and gamma (γ)-rays, mediates various forms of cancer cell death such as apoptosis, necrosis, autophagy, mitotic catastrophe, and senescence. Among them, apoptosis and mitotic catastrophe are the main ...

Matching Content Categories {📚}

  • Science
  • Health & Fitness
  • Education

Content Management System {📝}

What CMS is ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 6,399,052 visitors per month in the current month.

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How Does Ncbi.nlm.nih.gov Make Money? {💸}

We see no obvious way the site makes money.

Earning money isn't the goal of every website; some are designed to offer support or promote social causes. People have different reasons for creating websites. This might be one such reason. Ncbi.nlm.nih.gov might be cashing in, but we can't detect the method they're using.

Keywords {🔍}

doi, cancer, pubmed, google, scholar, cells, cell, radiation, dna, tumor, free, pmc, article, apoptosis, pathway, resistance, radiotherapy, response, repair, irinduced, death, damage, signaling, radioresistance, res, human, treatment, radiationinduced, role, protein, activation, kinase, expression, effects, pathways, activity, autophagy, cellular, inhibition, therapy, ionizing, therapeutic, target, mol, biol, senescence, tumors, akt, kim, anticancer,

Topics {✒️}

/c-jun n-terminal kinase dna–protein cross-links induced nf-kappab-mediated her2 overexpression traf-mediated nf-κb activation wnt/β-catenin pathway mediates targeting phosphatidylinositide3-kinase/akt pathway pi3k/akt-nf-kappab pathway pi3k-akt-mtor signaling pathway nf-κb-induced prosurvival genes egfr/pi3k/akt signaling pathway tnf-related apoptosis-inducing ligand carboxyl-terminal transactivation domain gamma-radiation-induced cell death low-dose ir-induced accumulation survivin-mediated radio-adaptive response rtk-pi3k-akt pathway cyclin d1/14-3-3ζ complex dna–pk catalytic subunit hypoxia-inducible factor-1 alpha multi-domain adaptor protein pmc beta search dna double-strand breaks dna–protein cross-links high-dose ir-induced apoptosis double-membrane vesicle called phosphatidylinositol 3-kinase/protein kinase single-strand break repair dna-dependent protein kinase hrad50-hmre11-p95 complex ceramide-activated protein phosphatase constitutively activated nf-κb blocking radiation-induced stabilization ceramide-activated protein kinase fih-dependent p300 dissociation post-doctoral research program stress-activated protein kinase wnt/beta-catenin signaling cell cycle-dependent manner wnt/β-catenin signaling nvp-bez-235 enhances radiosensitization wnt/β-catenin pathway ir-induced il-1β expression pi3k/akt/mtor pathway [39] pi3k/akt/mtor pathway nf-κb expression induced pi3k/akt signaling pathway activate cyclin-dependent kinases antiangiogenic/vascular targeting agents radiation-induced cell signaling radiation-induced cell death

Questions {❓}

  • Although there is evidence that necroptosis is involved in IR-induced cancer cell death, the role of this type of death is a very inchoate area of research (novel mechanism of IR-induced cell death?
  • Cancer caused by X-rays—A random event?
  • Enhancing radiotherapy with cyclooxygenase-2 enzyme inhibitors: A rational advance?
  • How Can We Overcome Tumor Hypoxia in Radiation Therapy?
  • Notch signaling in pancreatic cancer: Oncogene or tumor suppressor?
  • Radiation-induced bystander effects: What are they, and how relevant are they to human radiation exposures?
  • The role of Notch in tumorigenesis: Oncogene or tumour suppressor?

Social Networks {👍}(6)

External Links {🔗}(498)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • jQuery
  • jQuery module (jquery-3.6.0)
  • Zoom.js

Emails and Hosting {✉️}

Mail Servers:

  • nihcesxway.hub.nih.gov
  • nihcesxway2.hub.nih.gov
  • nihcesxway3.hub.nih.gov
  • nihcesxway4.hub.nih.gov
  • nihcesxway5.hub.nih.gov

Name Servers:

  • dns1-ncbi.ncbi.nlm.nih.gov
  • dns2-ncbi.ncbi.nlm.nih.gov
  • lhcns1.nlm.nih.gov
  • lhcns2.nlm.nih.gov

CDN Services {📦}

  • Ncbi

6.12s.