Here's how NCBI.NLM.NIH.GOV makes money* and how much!

*Please read our disclaimer before using our estimates.
Loading...

NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers
  14. CDN Services

We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC4542009/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC4542009/. The analysis below is for the second page.

Title[redir]:
Tumor Angiogenesis - PMC
Description:
No description found...

Matching Content Categories {📚}

  • Science
  • Education
  • Health & Fitness

Content Management System {📝}

What CMS is ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

check SE Ranking
check Ahrefs
check Similarweb
check Ubersuggest
check Semrush

How Does Ncbi.nlm.nih.gov Make Money? {💸}

We don't see any clear sign of profit-making.

Websites don't always need to be profitable; some serve as platforms for education or personal expression. Websites can serve multiple purposes. And this might be one of them. Ncbi.nlm.nih.gov could be getting rich in stealth mode, or the way it's monetizing isn't detectable.

Keywords {🔍}

doi, cells, google, scholar, pubmed, vegf, cancer, tumor, angiogenesis, endothelial, growth, antiangiogenic, vascular, cell, pmc, circulating, factor, article, blood, drug, receptor, drugs, vegfr, therapy, chemotherapy, receptors, tumors, free, signaling, treatment, dll, resistance, antibodies, vessels, science, bone, pathway, nat, clinical, levels, mice, stem, rev, progenitors, clin, med, survival, target, effects, expressed,

Topics {✒️}

cd11b+gr1+ myeloid-suppressor cells platelet-derived growth factor bone marrow–derived cells stromal-cell–derived factor 1 cd11b+gr1+ myeloid cells vhl von hippel–lindau called kinase-insert domain pmc beta search anti-plgf inhibits growth internally expressed vegfr1/flt1 vegf-induced adult neovascularization small-cell lung cancer proangiogenic tie2-expressing monocytes dll4–notch signaling interferes dll–notch receptor signaling continuous low-dose therapy activated endothelial cells metastatic renal-cell cancer notch–dll4 signaling pathway autocrine growth factor pmc copyright notice increased endothelial-cell survival small-molecule antiangiogenic rtkis drug-free periods7 low-dose metronomic chemotherapy cancer stem-cell hypothesis supplementary materials included primary breast cancer notch–dll4 signaling system sdf-1-cxcr4 signaling pathway promotes tumor growth placental growth factor long-term antiangiogenic therapy anti–vegfr-2 antibody therapy g-csf stimulates angiogenesis endothelial progenitor cells receptor tyrosine kinases notch–deltalike ligand full-length receptor tumor-inhibiting antiangiogenic drugs experimental drug-resistant cancer renal-cell carcinoma circulating endothelial cells multiple deltalike ligands cell-surface vegf receptors therapy-induced acute recruitment gr1+cd11b+ cells circulating endothelial progenitors block tumour growth signaling vascular morphogenesis

Questions {❓}

  • 11,12 These concerns raise the following questions with respect to improving antiangiogenic therapy: How do such drugs work, and how does bevacizumab increase the efficacy of chemotherapy?
  • Angiogenesis: an organizing principle for drug discovery?
  • Anti-Dll4 therapy: can we block tumour growth by increasing angiogenesis?
  • Antiangiogenic therapy: a universal chemosensitization strategy for cancer?
  • How do tumors become resistant to antiangiogenic drugs?
  • Where is VEGF in the body?

Social Networks {👍}(6)

External Links {🔗}(209)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • jQuery
  • jQuery module (jquery-3.6.0)

Emails and Hosting {✉️}

Mail Servers:

  • nihcesxway.hub.nih.gov
  • nihcesxway2.hub.nih.gov
  • nihcesxway3.hub.nih.gov
  • nihcesxway4.hub.nih.gov
  • nihcesxway5.hub.nih.gov

Name Servers:

  • dns1-ncbi.ncbi.nlm.nih.gov
  • dns2-ncbi.ncbi.nlm.nih.gov
  • lhcns1.nlm.nih.gov
  • lhcns2.nlm.nih.gov

CDN Services {📦}

  • Ncbi

4.67s.