NCBI . NLM . NIH . GOV {
}
We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC4531541/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC4531541/. The analysis below is for the second page.
Title[redir]:
CoMEt: a statistical approach to identify combinations of mutually exclusive alterations in cancer - PMC
Description:
Cancer is a heterogeneous disease with different combinations of genetic alterations driving its development in different individuals. We introduce CoMEt, an algorithm to identify combinations of alterations that exhibit a pattern of mutual ...
Title[redir]:
CoMEt: a statistical approach to identify combinations of mutually exclusive alterations in cancer - PMC
Description:
Cancer is a heterogeneous disease with different combinations of genetic alterations driving its development in different individuals. We introduce CoMEt, an algorithm to identify combinations of alterations that exhibit a pattern of mutual ...
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- Science
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What CMS is ncbi.nlm.nih.gov built with?
Custom-built
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Traffic Estimate {π}
What is the average monthly size of ncbi.nlm.nih.gov audience?
π Phenomenal Traffic: 5M - 10M visitors per month
Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
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How Does Ncbi.nlm.nih.gov Make Money? {πΈ}
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Earning money isn't the goal of every website; some are designed to offer support or promote social causes. People have different reasons for creating websites. This might be one such reason. Ncbi.nlm.nih.gov might be making money, but it's not detectable how they're doing it.
Keywords {π}
comet, alterations, genes, doi, cancer, mutations, sets, exclusivity, exclusive, samples, google, scholar, alteration, gene, number, additional, algorithm, pubmed, dataset, set, datasets, mutual, results, file, mutually, tcga, mutex, data, table, pmc, modules, pathways, article, signaling, test, subtype, mutation, gbm, free, collections, methods, multidendrix, tables, score, probability, subtypes, module, identify, contingency, multiple,
Topics {βοΈ}
comprehensive molecular characterization regulating microrna-29b expression pmc beta search cancer genome atlas simultaneously analyze type-specific e-cadherin-based adhesions protein-protein interaction network high-scoring gene triples cancer-type-specific alterations dna/histone methylation cancer subtype-specific mutations acute myeloid leukemia myh11-cbf Ξ² rtk/ras signaling pathway cancer de novo license information pmcid [google scholar] 28 [google scholar] 56 [google scholar] 61 [google scholar] 65 scavenger receptor genes somatic genomic landscape scavenger receptor srcrb4d acute promyelocytic leukemia acute megakaryoblastic leukemia human breast tumours subtype-specific altered genes tcga research network integer linear program comet outperformed multi-dendrix downloaded subtype information male-specific lethal de novo methods de gonzΓ‘lez ab cancer genome studies cancer genome projects de novo algorithms sloan research fellowship myeloid transcription factors important confounding factor remove low-weight edges pan-cancer analysis false positives resulting multiple sets simultaneously protein tyrosine phosphatase single nucleotide variations genome-scale data receptor tyrosine kinases epithelial-mesenchymal transition publisher site pdf
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