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lines, cell, doi, number, ubc, copy, cancer, tumors, bladder, table, google, scholar, gene, expression, pubmed, tumor, data, umuc, additional, upd, file, figure, mut, genes, genomic, analysis, chromosome, fgfr, mutations, genome, ucc, loh, pmc, alterations, article, mutation, high, regions, altered, loss, information, identified, lost, chr, free, status, frequency, line, provided, probes,

Topics {✒️}

enrique carrillo-de-santa-pau 1 enrique carrillo-de-santa-pau carrillo-de santa pau carrillo-de-santa-pau fgfr3-driven pathway showed genetic/epigenetic alterations inactivate tumor suppressor-driven pathways fgfr3-driven tumor pathway pmc beta search 4/amp4/fgfr3-tacc3 fusion 13 anticancer drug sensitivity muscle invasive muscle-invasive genomically unstable-high group oncogene/tumor suppressor status minimal common regions copy number calls urothelial bladder cancer papillary/luminal tumor phenotypes high-grade lines tended mycoplasma-free cultures specific tumor subtypes copy number neutral 0 = copy number neutral urothelial bladder carcinoma chromosome/chromosome arm gain/loss copy number alterations um-uc-7 demonstrated amplification uniparental disomy high-resolution analysis intrinsic molecular subtypes publisher site pdf gene copy number gene-level expression values human cancer pathogenesis urinary bladder cancer urothelial neoplastic progression complete copy number copy number data copy call results copy number analysis genome instability variable cel files web-based search [google scholar] 42 affymetrix u133a array van der kwast comprehensive genomic characterization urothelial bladder tumors urothelial cell carcinoma

Questions {❓}

• P53: it has it all, but will it make it to the clinic as a marker in bladder cancer?

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