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We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC4446723/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC4446723/. The analysis below is for the second page.
Title[redir]:
Variation in cancer risk among tissues can be explained by the number of stem cell divisions - PMC
Description:
Some tissue types give rise to human cancers millions of times more often than other tissue types. Although this has been recognized for more than a century, it has never been explained. Here, we show that the lifetime risk of cancers of many ...
Title[redir]:
Variation in cancer risk among tissues can be explained by the number of stem cell divisions - PMC
Description:
Some tissue types give rise to human cancers millions of times more often than other tissue types. Although this has been recognized for more than a century, it has never been explained. Here, we show that the lifetime risk of cancers of many ...
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🌠 Phenomenal Traffic: 5M - 10M visitors per month
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Keywords {🔍}
doi, cancer, google, scholar, pubmed, risk, pmc, stem, factors, cell, cells, number, article, divisions, free, environmental, supplementary, tissues, lifetime, stochastic, materials, tissue, fig, tumors, ers, types, inherited, nlm, science, cancers, small, hereditary, effects, search, dna, replication, nature, correlation, high, research, prevention, ncbi, total, mutations, differences, intestine, aers, data, health, human,
Topics {✒️}
nih grants p30-ca006973 pmc beta search reducing cancer deaths [google scholar] 3 [google scholar] 12 nonhereditary d-tumors extensive literature search natl cancer inst sci transl med supplementary materials included publisher site pdf basal epidermal cells basal cell carcinomas organ-specific cancer risk final edited form license information pmcid familial adenomatous polyposis insightful ideas relating typically short-lived hierarchical division patterns machine learning methods clusters set equal sol goldman center small intestinal epithelium primary prevention measures endogenous mutation rate human papilloma virus environ health perspect factors versus environmental stem cell division somatic mutation theory genome-wide analyses data availability statements renewing cells maintaining pmc disclaimer stem cell divisions factors underlying neoplasia noncancerous stem cells stochastic processes involved cancer prev res american cancer society random mutations arising small intestine undergoes small intestinal tumors fully differentiated cells highly positive correlation linear correlation equal blood-brain barrier national cancer institute united states government
Questions {❓}
- How successful can such approaches be?
- If hereditary and environmental factors cannot fully explain the differences in organ-specific cancer risk, how else can these differences be explained?
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