Here's how NCBI.NLM.NIH.GOV makes money* and how much!

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NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers
  14. CDN Services

We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC4414133/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC4414133/. The analysis below is for the second page.

Title[redir]:
Bcl-2 family proteins in breast development and cancer: could Mcl-1 targeting overcome therapeutic resistance? - PMC
Description:
Apoptosis, cell death executed by caspases, is essential to normal breast development and homeostasis. Pro-apoptotic and anti-apoptotic signals are tightly regulated in normal breast epithelial cells. Dysregulation of this balance is required for ...

Matching Content Categories {πŸ“š}

  • Science
  • Education
  • Health & Fitness

Content Management System {πŸ“}

What CMS is ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {πŸ“ˆ}

What is the average monthly size of ncbi.nlm.nih.gov audience?

πŸš€πŸŒ  Tremendous Traffic: 10M - 20M visitors per month


Based on our best estimate, this website will receive around 10,537,842 visitors per month in the current month.

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How Does Ncbi.nlm.nih.gov Make Money? {πŸ’Έ}

We don’t know how the website earns money.

The purpose of some websites isn't monetary gain; they're meant to inform, educate, or foster collaboration. Everyone has unique reasons for building websites. This could be an example. Ncbi.nlm.nih.gov might be earning cash quietly, but we haven't detected the monetization method.

Keywords {πŸ”}

doi, bcl, breast, cell, cancer, google, scholar, mcl, pubmed, family, proteins, antiapoptotic, apoptosis, pmc, cancers, death, article, mammary, free, abt, cells, tumor, resistance, expression, inhibitor, involution, bax, development, bclxl, bim, members, protein, targeting, proapoptotic, pathway, activity, therapeutic, gland, apoptotic, bakbax, bhonly, overexpression, studies, tumors, targeted, signaling, figure, levels, response, transgenic,

Topics {βœ’οΈ}

accelerates c-myc-induced tumorigenesis mmtv-c-myc transgenic mice bcl-xlfl/fl mmtv-cre c-myc dependent expression enhance bak/bax-mediated momp anti-apoptotic family members counteract anti-apoptotic members bcl-xl/bim fragment complex pre-clinical testing remains sv40 large t-antigen sequestering anti-apoptotic proteins triple-negative breast cancers estrogen-responsive elements present inhibit bak/bax-mediated apoptosis form higher-order oligomers include anti-apoptotic proteins pten-deficient tumor cells targeting bcl-2/bcl-xl/bcl sustains base-line activity 17beta-estradiol inhibits apoptosis oncogene-expressing mammary acini leukemia/lymphoma cell lines anti-apoptotic bcl-2 proteins anti-apoptotic proteins bcl-2 pmc beta search genetic bcl-xl ablation long-term estrogen deprivation small-cell lung cancer anthracene-treated transgenic mice undergo hormonally-induced growth pro-apoptotic bcl-2 protein pi3k/akt kinase pathways patient-level meta-analysis chemo-refractory tnbc samples pro-apoptotic protein bax stabilize protein-protein interactions c-myc induced [25] ubiquitin-mediated proteasomal degradation therapy-induced cell death mcl-1-expressing myeloma cells /sv40-tag transgenic mice anti-apoptotic signals cooperate bcl-2-expressing breast tumors 78-kda glucose-regulated protein rapid pregnancy-induced growth targeted high-affinity inhibitor bak-mediated mitochondrial apoptosis pro-apoptotic bcl-2 activators pre-clinical models support anti-her2 antibody

Questions {❓}

  • Bcl-2 family proteins in breast development and cancer: could Mcl-1 targeting overcome therapeutic resistance?

External Links {πŸ”—}(144)

Analytics and Tracking {πŸ“Š}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {πŸ“š}

  • jQuery
  • jQuery module (jquery-3.6.0)

Emails and Hosting {βœ‰οΈ}

Mail Servers:

  • nihcesxway.hub.nih.gov
  • nihcesxway2.hub.nih.gov
  • nihcesxway3.hub.nih.gov
  • nihcesxway4.hub.nih.gov
  • nihcesxway5.hub.nih.gov

Name Servers:

  • dns1-ncbi.ncbi.nlm.nih.gov
  • dns2-ncbi.ncbi.nlm.nih.gov
  • lhcns1.nlm.nih.gov
  • lhcns2.nlm.nih.gov

CDN Services {πŸ“¦}

  • Ncbi

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