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NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers
  14. CDN Services

We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC4329227/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC4329227/. The analysis below is for the second page.

Title[redir]:
The pentose phosphate pathway and cancer - PMC
Description:
The pentose phosphate pathway (PPP), which branches from glycolysis at the first committed step of glucose metabolism, is required for the synthesis of ribonucleotides and is a major source of NADPH. NADPH is required for and consumed during fatty ...

Matching Content Categories {📚}

  • Science
  • Health & Fitness
  • Education

Content Management System {📝}

What CMS is ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Ncbi.nlm.nih.gov Make Money? {💸}

We see no obvious way the site makes money.

Many websites are intended to earn money, but some serve to share ideas or build connections. Websites exist for all kinds of purposes. This might be one of them. Ncbi.nlm.nih.gov has a secret sauce for making money, but we can't detect it yet.

Keywords {🔍}

ppp, doi, cancer, cells, google, scholar, pubmed, oxidative, nadph, cell, pmc, gpdh, nonoxidative, activity, glucose, free, article, phosphate, pentose, tumor, expression, dehydrogenase, ros, enzymes, pathway, stress, mechanisms, glycolysis, flux, glucosephosphate, high, synthesis, glycolytic, branch, levels, survival, enzyme, regulation, generate, dna, tigar, elevated, required, phosphogluconate, transketolase, nrf, activation, biol, metabolic, increase,

Topics {✒️}

c-src-mediated tyrosine phosphorylation k-ras-induced mouse models daunorubicin-resistant p388 cells pmc beta search k-ras include lung fail-safe mechanism adopted pro-apoptotic protein noxa p53-related protein p73 c-amp response element pro-oncogenic signaling pathways platelet-derived growth factor k-ras-dependent cancer erythroid cell-derived protein glucose-6-phosphate dehydrogenase deficiency pi3k/akt signaling pathway52 high-energy phosphate bonds antioxidant n-acetyl cysteine bound glucose-6-phosphate dehydrogenase produce free radicals human glucose-6-phosphate dehydrogenase acetaminophen-induced liver failure red blood cells nrf2-keap1 signaling pathway trends biochem sci primary tumor site license information pmcid concluding remarks pro-apoptotic protein tyrosine kinase src display higher levels oncogenic k-ras nih grants r01ag016927 cell cycle arrest rapidly dividing cells 6-phosphofructo-2-kinase/fructose-2 reactive oxygen species [google scholar] 2 low nadp+/nadph ratio protein phosphatase-2a protein-protein interaction cytochrome p450 reductase promotes cell proliferation epidermal growth factor pentose phosphate cycle ribulose-5-phosphate epimerase pro-tumorigenic inflammation glucose-6-phosphate dehydrogenase glucose-6 phosphate dehydrogenase glucose 6-phosphate dehydrogenase metastatic site-dependent

Questions {❓}

  • Is it possible that the inhibition of the oxidative PPP could increase intracellular ROS, which in turn selectively eradicates cancer cells57?
  • Is it possible to inhibit the PPP at the organismal level to selectively inhibit tumorigenesis without significant adverse physiological Manuscript consequences?
  • Is transketolase-like protein, TKTL1, transketolase?
  • Nonoxidative pentose phosphate pathways and their direct role in ribose synthesis in tumors: is cancer a disease of cellular glucose metabolism?
  • What are the exact mechanisms by which oncogenic Ras or other oncogenic signals elevate the nonoxidative PPP to generate ribonucleotides?

Social Networks {👍}(6)

External Links {🔗}(152)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • jQuery
  • jQuery module (jquery-3.6.0)
  • Zoom.js

Emails and Hosting {✉️}

Mail Servers:

  • nihcesxway.hub.nih.gov
  • nihcesxway2.hub.nih.gov
  • nihcesxway3.hub.nih.gov
  • nihcesxway4.hub.nih.gov
  • nihcesxway5.hub.nih.gov

Name Servers:

  • dns1-ncbi.ncbi.nlm.nih.gov
  • dns2-ncbi.ncbi.nlm.nih.gov
  • lhcns1.nlm.nih.gov
  • lhcns2.nlm.nih.gov

CDN Services {📦}

  • Ncbi

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