Here's how NCBI.NLM.NIH.GOV makes money* and how much!

*Please read our disclaimer before using our estimates.
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NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers
  14. CDN Services

We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC4040260/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC4040260/. The analysis below is for the second page.

Title[redir]:
Apoptotic cell clearance: basic biology and therapeutic potential - PMC
Description:
Prompt removal of apoptotic cells by phagocytes is important for maintaining tissue homeostasis. The molecular and cellular events that underpin apoptotic cell recognition and uptake, and the subsequent biological responses are increasingly better ...

Matching Content Categories {📚}

  • Science
  • Health & Fitness
  • Education

Content Management System {📝}

What CMS is ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Ncbi.nlm.nih.gov Make Money? {💸}

The income method remains a mystery to us.

Not all websites are made for profit; some exist to inform or educate users. Or any other reason why people make websites. And this might be the case. Ncbi.nlm.nih.gov might have a hidden revenue stream, but it's not something we can detect.

Keywords {🔍}

doi, apoptotic, cells, cell, pubmed, google, scholar, pmc, free, article, clearance, macrophages, apoptosis, inflammation, phagocytes, engulfment, death, phagocytosis, macrophage, recognition, membrane, ptdser, protein, promote, atherosclerosis, disease, nature, exposure, therapeutic, tumour, inflammatory, immune, immunol, response, neutrophils, neutrophil, dying, signals, mice, biol, molecular, process, receptor, release, signal, human, blood, mechanisms, tissue, receptors,

Topics {✒️}

abca1-mediated immuno-suppressive effects81 phospholipid scramblase/amino-phospholipid translocase tnf-related apoptosis-inducing ligand advanced glycation end-products elmo1-dock180-rac module anti-cd47 antibody-mediated phagocytosis c-type lectin mincle elmo/dock180/rac module post-myocardial infarct repair low-density lipoprotein cholesterol factor-viii-homologous domains transforming growth factor-β pmc beta search bleomycin-induced pulmonary fibrosis96 thiol compound n-acetylcysteine mediates cell-cell adhesion cyclin-dependent kinase inhibitors181 reduced pro-inflammatory cytokines dismantling cell-cell contacts pro-resolving lipid mediators n-acetylcysteine inhibits rhoa t-cell-dependent antigen peroxisome proliferator-activated receptors inhibitors targeting hydroxymethylglutaryl-coenzyme develop apoptotic cell-based apoptotic cell-mediated suppression termed pro-resolving lipids endogenous lipid-derived autacoids apolipoprotein e-knockout mice anti-cd47 antibody synergizes mannose binding lectin apoptotic cell-based therapy brain-specific angiogenesis inhibitor nadph oxidase-dependent manner display anti-inflammatory effects augment anti-tumour immunity apoptotic jurkat cells--existence s100 calcium-binding proteins pro-resolving lipid production potential cell-based therapy tumour-specific monoclonal antibodies regulating antigen cross-presentation low density lipoprotein soluble sirpα variants apoptotic cell-derived microparticles hmg-coa reductase inhibitors glucose homeostasis-independent manner111 apoptotic cell-based therapies graft-versus-host disease growth arrest-specific gene

Questions {❓}

  • Annexin A1 Membrane lysis ?
  • Apoptotic cell-mediated suppression of streptococcal cell wall-induced arthritis is associated with alteration of macrophage function and local regulatory T-cell increase: a potential cell-based therapy?
  • But, why do human macrophages in situ lose the ability to rapidly clear dead cells?
  • CRT Exocytic?
  • EMAP II ?
  • ICAM-3 Microparticles ?
  • Lactoferrin ?
  • Macrophage reverse cholesterol transport: key to the regression of atherosclerosis?
  • Novel lipid mediators and resolution mechanisms in acute inflammation: to resolve or not?
  • PtdSer Phospholipid scramblase/amino-phospholipid translocase?
  • So, how do apoptotic cells convey such a diverse array of immunological information?

Social Networks {👍}(6)

External Links {🔗}(396)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • jQuery
  • jQuery module (jquery-3.6.0)
  • Zoom.js

Emails and Hosting {✉️}

Mail Servers:

  • nihcesxway.hub.nih.gov
  • nihcesxway2.hub.nih.gov
  • nihcesxway3.hub.nih.gov
  • nihcesxway4.hub.nih.gov
  • nihcesxway5.hub.nih.gov

Name Servers:

  • dns1-ncbi.ncbi.nlm.nih.gov
  • dns2-ncbi.ncbi.nlm.nih.gov
  • lhcns1.nlm.nih.gov
  • lhcns2.nlm.nih.gov

CDN Services {📦}

  • Ncbi

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