Here's how NCBI.NLM.NIH.GOV makes money* and how much!

*Please read our disclaimer before using our estimates.
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NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers
  14. CDN Services

We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC3679459/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC3679459/. The analysis below is for the second page.

Title[redir]:
Non-canonical kinase signaling by the death ligand TRAIL in cancer cells: discord in the death receptor family - PMC
Description:
Tumor necrosis factor-related apoptosis-inducing ligand (TRAIL)-based therapy is currently evaluated in clinical studies as a tumor cell selective pro-apoptotic approach. However, besides activating canonical caspase-dependent apoptosis by binding ...

Matching Content Categories {📚}

  • Science
  • Education
  • Health & Fitness

Content Management System {📝}

What CMS is ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of ncbi.nlm.nih.gov audience?

🚀🌠 Tremendous Traffic: 10M - 20M visitors per month


Based on our best estimate, this website will receive around 10,538,143 visitors per month in the current month.

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How Does Ncbi.nlm.nih.gov Make Money? {💸}

We can't tell how the site generates income.

Websites don't always need to be profitable; some serve as platforms for education or personal expression. Websites can serve multiple purposes. And this might be one of them. Ncbi.nlm.nih.gov might be plotting its profit, but the way they're doing it isn't detectable yet.

Keywords {🔍}

doi, trail, cells, pubmed, google, scholar, activation, apoptosis, cell, tumor, signaling, cancer, death, kinase, receptor, receptors, trailinduced, protein, caspase, pmc, inhibition, jnk, kinases, ligand, article, pathway, survival, free, proliferation, expression, erk, trailr, akt, activity, nfκb, resistance, leading, apoptosisinducing, complex, phosphorylation, proapoptotic, src, human, family, rip, found, tak, prosurvival, biol, necrosis,

Topics {✒️}

nuclear factor kappa-light-chain-enhancer urokinase-type plasminogen activator tnf-related apoptosis-inducing ligand chromosome ten c-jun n-terminal kinase c-jun n-terminal kinases reactive oxygen species transforming growth factor-β transforming growth factor-alpha receptor-interacting protein 1 trail receptor-interacting proteins tumor necrosis factor-alpha nuclear factor-kappab-dependent transcription mitogen-activated protein kinase mitogen-activated protein kinases casitas b-lineage lymphoma epidermal growth factor fas ligand/apo1 ligand p60-c-src suppresses apoptosis traf2-mekk1-mkk4-dependent signaling pathway rho kinase nf-κb-dependent pro-survival signals p38 mitogen-activated protein egf/erbb1 signaling pathway nf-kappab/ikappa balpha pathways transcription factor nf-κb growth factor-1 receptor nuclear factor-kappab blockade pmc beta search sk-n-mc neuronal cells inducing caspase-dependent apoptosis nf-κb subunit p65/rela stress-activated members death-inducing signaling complex egfr/her2 pro-survival signaling extracellular regulated kinases x-linked inhibitor inhibitory protein inhibiting smac/diablo release cyclin-dependent kinases p21 serine/threonine kinase mtor cell type-dependent variation mammalian sterile 20 x-linked iap rip1/src/stat3 axis human epidermal receptor actin-severing protein cofilin cell type-dependent manner reference iκb/nf-κb /lim kinase-mediated phosphorylation

Questions {❓}

  • Can TRAIL apoptosis sensitivity in tumor cells be predicted by not yet identified biomarkers, allowing the preselection of patients eligible for TRAIL receptor agonistic therapy?
  • Can possible identified mechanism(s)/proteins that function as an apoptotic switch in the TRAIL pathway be used as a target for developing therapeutic strategies for sensitizing tumor cells?
  • What is the molecular basis of the apparent differences between TRAIL-R1- and TRAIL-R2-dependent signaling?
  • What is/are the molecular mechanism(s) underlying the dichotomy in TRAIL signaling in sensitive versus resistant tumor cells?
  • Which factors produced by the tumor microenvironment determine the outcome of TRAIL signaling and through what mechanism(s)?

Social Networks {👍}(6)

External Links {🔗}(250)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • AOS
  • jQuery
  • jQuery module (jquery-3.6.0)
  • Zoom.js

Emails and Hosting {✉️}

Mail Servers:

  • nihcesxway.hub.nih.gov
  • nihcesxway2.hub.nih.gov
  • nihcesxway3.hub.nih.gov
  • nihcesxway4.hub.nih.gov
  • nihcesxway5.hub.nih.gov

Name Servers:

  • dns1-ncbi.ncbi.nlm.nih.gov
  • dns2-ncbi.ncbi.nlm.nih.gov
  • lhcns1.nlm.nih.gov
  • lhcns2.nlm.nih.gov

CDN Services {📦}

  • Ncbi

4.13s.