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NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers
  14. CDN Services

We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC3652335/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC3652335/. The analysis below is for the second page.

Title[redir]:
Lin28: Primal Regulator of Growth and Metabolism in Stem Cells - PMC
Description:
In recent years, the highly conserved Lin28 RNA-binding proteins have emerged as factors that define stemness in several tissue lineages. Lin28 proteins repress let-7 microRNAs and influence mRNA translation, thereby regulating the self-renewal of ...

Matching Content Categories {📚}

  • Education
  • Science
  • Fitness & Wellness

Content Management System {📝}

What CMS is ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of ncbi.nlm.nih.gov audience?

🚀🌠 Tremendous Traffic: 10M - 20M visitors per month


Based on our best estimate, this website will receive around 10,538,143 visitors per month in the current month.

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How Does Ncbi.nlm.nih.gov Make Money? {💸}

We find it hard to spot revenue streams.

Not all websites are made for profit; some exist to inform or educate users. Or any other reason why people make websites. And this might be the case. Ncbi.nlm.nih.gov has a revenue plan, but it's either invisible or we haven't found it.

Keywords {🔍}

doi, pubmed, google, scholar, lin, cell, pmc, stem, article, free, cells, lina, linab, cancer, selfrenewal, human, linb, microrna, regulate, elegans, expression, mammalian, metabolism, escs, development, nature, reprogramming, mouse, rna, science, tissue, genes, differentiation, dev, biol, growth, translation, zhu, regulates, wang, overexpression, adult, embryonic, directly, pluripotent, studies, targets, factors, micrornas, moss,

Topics {✒️}

inflammation-/stress-responsive nf-κb cre-er-driven mouse models long-term adult hscs single-cell rna-seq analysis retinoic acid-induced differentiation genome-wide rna-immunoprecipitation studies telomeric g-strand tails pmc beta search long-lived mouse strains rna-induced silencing complex nf-κb transactivate lin28b genome-wide approach reveals x-ray crystallography studies reverts myc-induced growth peripheral t-cell lymphoma nuclear protein-protein complex degrade oligo-uridylated pre single-cell expression analyses neural stem-cell commitment primordial germ-cell development insulin-pi3k-mtor pathway lin-4 homolog mir-125a/ genome-wide studies reveal impaired t-cell development genome-wide association study expect oligo-uridylated pre rna-processing mechanisms relate leukaemia-initiating cells post-transcriptional rna processing tumor-initiating cells fetal hematopoietic stem repress lin-28 post-transcriptionally upregulate insulin/pi3k signaling inhibit neuronal differentiation aging telomerase-deficient mice rna-binding cofactor dgcr8 thr-gly-sam pathway rna-binding protein lin28 mll-fusion/myc/lin-28 neural crest progenitors primitive neural tissue nuclear receptor signaling stem cell exhaustion nuclear receptor nr5a2 regulate stem/progenitor cell bhat-nakshatri embryonic stem cells promising universal factor regulatory signals upstream tumorigenic melanoma cells

Questions {❓}

  • And what conditions or pathways regulate the mechanism of Lin28a or Lin28b within the nucleolus, the cytoplasm, and P-bodies?
  • But is Lin28 also required for pluripotency?
  • But what else could Lin28a/b be doing in the nucleus?
  • Could Lin28 show similar potential in reengineering adult human cells (Figure 3)?
  • Do pluripotent stem cells then require Lin28a/b for indefinite self-renewal?
  • Healthy aging: is smaller better?
  • If Lin28a/b can enhance self-renewal, what effects would it exert on adult tissue repair in vivo?
  • The central role of the Lin28/let-7 bistable switch in governing stem cell self-renewal raises the provocative question: what signaling pathways converge upstream to regulate the switch?

External Links {🔗}(320)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • jQuery
  • jQuery module (jquery-3.6.0)
  • Zoom.js

Emails and Hosting {✉️}

Mail Servers:

  • nihcesxway.hub.nih.gov
  • nihcesxway2.hub.nih.gov
  • nihcesxway3.hub.nih.gov
  • nihcesxway4.hub.nih.gov
  • nihcesxway5.hub.nih.gov

Name Servers:

  • dns1-ncbi.ncbi.nlm.nih.gov
  • dns2-ncbi.ncbi.nlm.nih.gov
  • lhcns1.nlm.nih.gov
  • lhcns2.nlm.nih.gov

CDN Services {📦}

  • Ncbi

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