NCBI . NLM . NIH . GOV {
}
We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC3582387/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC3582387/. The analysis below is for the second page.
Title[redir]:
A Competitive Stapled Peptide Screen Identifies a Selective Small Molecule that Overcomes MCL-1-dependent Leukemia Cell Survival - PMC
Description:
Cancer cells hijack BCL-2 family survival proteins to suppress the death effectors and thereby enforce an immortal state. This is accomplished biochemically by an anti-apoptotic surface groove that neutralizes the pro-apoptotic BH3 α-helix of death ...
Title[redir]:
A Competitive Stapled Peptide Screen Identifies a Selective Small Molecule that Overcomes MCL-1-dependent Leukemia Cell Survival - PMC
Description:
Cancer cells hijack BCL-2 family survival proteins to suppress the death effectors and thereby enforce an immortal state. This is accomplished biochemically by an anti-apoptotic surface groove that neutralizes the pro-apoptotic BH3 α-helix of death ...
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Keywords {🔍}
doi, mcl, mim, google, scholar, pubmed, mclδnδc, binding, abt, bcl, figure, cells, pmc, antiapoptotic, small, cell, article, bax, cancer, proteins, free, selective, molecule, protein, molecules, inhibitor, competitive, interaction, activity, bclxlδc, sahbd, activation, family, residues, peptide, bclxl, chem, stapled, bhbinding, apoptosis, sahba, assay, fitcmcl, ball, screen, death, chemical, assays, leukemia, analysis,
Topics {✒️}
bcl-xl-reconstituted p185+arf−/−/mcl-1-deleted b bcl-xl-rescued p185+arf−/−mcl-1-deleted b bcl-xl-rescued p185+arf−/−/mcl-1-deleted b mcl-1-reconstituted p185+arf−/−/mcl-1-deleted b mcl-1-rescued p185+arf−/−mcl-1-deleted b transformed p185+mcl-1f/farf−/− b p185+arf−/−/mcl-1-deleted b p185+arf−/−mcl-1-deleted b p-xylene-bis-pyridinium bromide diverse alpha/beta-backbone patterns comparative fitc-bid bh3/mcl-1δnδc bcl-2/bcl-xl-mediated apoptotic blockade fitc-mcl-1 sahba/mcl-1δnδc complex fitc-mcl-1 sahba/mcl-1δnδc interaction fitc-bid bh3/mcl-1δnδc interaction negated bcl-xlδc-mediated suppression pcr-based site-directed mutagenesis bcl-2/bcl-xl-selective antagonist abt-737 mcl-1δnδc cross-peak assignments mcl-1δnδc-mediated bax suppression isogenic p185+arf−/− b pro-apoptotic bh3 α-helix selective bcl-2/bcl-xl inhibitor parental p185+arf−/− b mcl-1-dependent anti-leukemia activity bcl-xl-bak peptide complex fitc-mcl-1 sahba/mcl-1δnδc 15n-mcl-1δnδc chemical shift selective bcl-2/bcl-xl inhibition bcl-2/bcl-xl-selective inhibitors blocking mcl-1-mediated suppression [google scholar] doi mcl-1 sahbd/mcl-1δnδc complex wild-type mcl-1δnδc binding fitc-bad bh3/bcl-xlδc pmc beta search overcomes mcl-1-mediated resistance targeted high-affinity inhibitor bcl-2/bcl-xl subclass mcl-1δnδc disrupts interaction hydrocarbon-stapled bh3 helices somatic copy-number alteration marine-derived ascomycete related strictly mcl-1-dependent leukemia peptidic stabilized alpha-helix incubating fitc-mcl-1 sahba stapled peptide/protein complex p185+arf−/− b selective anti-apoptotic inhibitor anti-apoptotic proteins represents
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- Income figures for https://scholar.google.com/scholar_lookup?journal=J%20Biol%20Chem&title=Identification%20of%20a%20novel%20Mcl-1%20protein%20binding%20motif&author=WJ%20Placzek&author=M%20Sturlese&author=B%20Wu&author=JF%20Cellitti&author=J%20Wei&volume=286&publication_year=2011&pages=39829-39835&pmid=21953453&doi=10.1074/jbc.M111.305326&
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