Here's how NCBI.NLM.NIH.GOV makes money* and how much!

*Please read our disclaimer before using our estimates.
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NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers
  14. CDN Services

We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC3552512/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC3552512/. The analysis below is for the second page.

Title[redir]:
Translational Control in Cancer Etiology - PMC
Description:
The link between perturbations in translational control and cancer etiology is becoming a primary focus in cancer research. It has now been established that genetic alterations in several components of the translational apparatus underlie ...

Matching Content Categories {📚}

  • Education
  • Health & Fitness
  • Science

Content Management System {📝}

What CMS is ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Ncbi.nlm.nih.gov Make Money? {💸}

We're unsure if the website is profiting.

Not all websites focus on profit; some are designed to educate, connect people, or share useful tools. People create websites for numerous reasons. And this could be one such example. Ncbi.nlm.nih.gov might be plotting its profit, but the way they're doing it isn't detectable yet.

Keywords {🔍}

scholar, google, pubmed, doi, cancer, cell, translation, translational, pmc, article, protein, free, control, initiation, eife, cells, tumor, mrnas, specific, ribosome, ribosomal, oncogenic, growth, factor, expression, factors, synthesis, gene, cellular, tumors, biol, human, transformation, development, mrna, mol, increased, increase, activity, role, overexpression, regulation, key, solid, rna, myc, ruggero, rrna, components, important,

Topics {✒️}

bcr-abl-positive leukemias identified hypoxia-inducible factor-1α protein vivo-assembled rna-protein complexes tumor-propagating-cancer stem cells eukaryotic initiation factor-4e pmc beta search clinical atp-site inhibitor deep-sequence ribosome-protected mrnas polypyrimidine tract-binding protein rna-dependent protein kinase atp-active site inhibitors ires trans-acting factors ires-trans-acting factors decreasing ires-dependent translation impairs ires-dependent translation eif-4e bp1 phosphorylation rna-binding protein interactions initiator met-trnai binding dual pi3k/mtor inhibitor pi3k-akt-mtor pathway putative chromatin-remodeling protein ras-map kinase signaling cap-dependent translation initiation perk/eif2α/atf4 module promote cap-independent translation pyrimidine-rich translation element mrna cap-binding protein regulate cap-independent translation rapamycin-insensitive kinase kinases x-linked dyskeratosis congenita hypophosphorylated 4e-bps bind 4e-bps/eif4e axis p53 mrna polyadenylation/translation ires-dependent mrna translation catalog genome-wide variations y-box binding protein y-box binding protein-1 site-specific rrna modifications bone marrow failure carboxy-terminal activation domain cold spring harbor poor-prognosis prostate cancer cis-regulatory elements present overcoming tumor-suppressive barriers fine-tune gene expression l11-mdm2-p53 pathway atp-competitive mammalian target modulating ires-mediated translation allogenic antibody-mediated identification transcript-specific translational control

Questions {❓}

  • But how does the cancer program control specific mRNAs to support the growth of cancer cells?
  • Can such mutant forms of the translational machinery be referred to as “cancer ribosomes”?
  • Do Mutations in the Ribosome Cause Cancer?
  • Do genetic perturbations in mRNA regulatory elements directly lead to cancer formation?
  • Do these findings indicate that cells transformed by mTOR hyperactivation become “addicted” to oncogenic eIF4E-mediated translation control?
  • Does the ribosome translate cancer?
  • How do these signaling pathways alter translational control when mutated in cancer?
  • In particular, what regulates ribosomal RNA (rRNA) modifications?
  • Is it possible that, on its own, increased cell growth represents one of the oncogenic “hits” that leads to cellular transformation?
  • What is the evidence that these changes exert direct and causal effects in cancer?
  • Why Has the Translational Control of Specific mRNAs Been Selected for Cancer Development?
  • Why is translational control of specific mRNAs important for the coordination of cell invasion and metastasis?

Social Networks {👍}(6)

External Links {🔗}(474)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • jQuery
  • jQuery module (jquery-3.6.0)
  • Zoom.js

Emails and Hosting {✉️}

Mail Servers:

  • nihcesxway.hub.nih.gov
  • nihcesxway2.hub.nih.gov
  • nihcesxway3.hub.nih.gov
  • nihcesxway4.hub.nih.gov
  • nihcesxway5.hub.nih.gov

Name Servers:

  • dns1-ncbi.ncbi.nlm.nih.gov
  • dns2-ncbi.ncbi.nlm.nih.gov
  • lhcns1.nlm.nih.gov
  • lhcns2.nlm.nih.gov

CDN Services {📦}

  • Ncbi

4.96s.