NCBI . NLM . NIH . GOV {
}
We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC3365221/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC3365221/. The analysis below is for the second page.
Title[redir]:
Critical role of soluble amyloid-β for early hippocampal hyperactivity in a mouse model of Alzheimer’s disease - PMC
Description:
Alzheimer’s disease (AD) is characterized by a progressive dysfunction of central neurons. Recent experimental evidence indicates that in the cortex, in addition to the silencing of a fraction of neurons, other neurons are hyperactive in amyloid-β ...
Title[redir]:
Critical role of soluble amyloid-β for early hippocampal hyperactivity in a mouse model of Alzheimer’s disease - PMC
Description:
Alzheimer’s disease (AD) is characterized by a progressive dysfunction of central neurons. Recent experimental evidence indicates that in the cortex, in addition to the silencing of a fraction of neurons, other neurons are hyperactive in amyloid-β ...
Matching Content Categories {📚}
- Education
- Pets
- Science
Content Management System {📝}
What CMS is ncbi.nlm.nih.gov built with?
Custom-built
No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.
Traffic Estimate {📈}
What is the average monthly size of ncbi.nlm.nih.gov audience?
🌠 Phenomenal Traffic: 5M - 10M visitors per month
Based on our best estimate, this website will receive around 6,399,052 visitors per month in the current month.
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How Does Ncbi.nlm.nih.gov Make Money? {💸}
We can't tell how the site generates income.
Some websites aren't about earning revenue; they're built to connect communities or raise awareness. There are numerous motivations behind creating websites. This might be one of them. Ncbi.nlm.nih.gov could have a money-making trick up its sleeve, but it's undetectable for now.
Keywords {🔍}
mice, doi, neurons, transgenic, neuronal, pubmed, google, scholar, hippocampal, activity, fig, transients, soluble, wildtype, pmc, vivo, hyperactivity, article, free, hyperactive, plaques, spontaneous, mouse, test, cells, disease, hippocampus, found, cortex, imaging, levels, dimers, alzheimers, students, silent, γsecretase, brain, synaptic, compared, significantly, action, neurosci, early, twophoton, functional, protein, amyloidβ, evidence, amyloid, directly,
Topics {✒️}
carl-von-linde senior fellow aqueous-soluble [tris-buffered saline γ-secretase inhibitor n2-[ acute γ-secretase inhibition γ-secretase inhibitor ly-411575 γ-secretase inhibitor treatment amyloid precursor protein long-term outcome targeted patch-clamp recordings age-matched wild-type mice resonant galvo-mirror system ]azepin-7-yl]-l-alaninamide org/lookup/suppl/doi l-2-amino-5-phosphonovaleric acid nmda receptor trafficking pmc beta search soluble amyloid-β amyloid β oligomers somatic cell-attached recordings γ-secretase inhibition β-amyloid peptide 25–35 cell-specific pathology isolates high-level neuronal expression ly-411575–treated wt controls combined cell-attached recordings artificial cerebrospinal fluid custom-written software based video-rate confocal microscopy ca2+ influx induced plaque-bearing ca1 region primary bioactive form soluble aβ rapidly plaque-bearing transgenic mice ly-411575–treated transgenic mice aβ-induced neuronal hyperactivity cellular prion protein full-frame images ly-411575–treated tg mouse synaptic plasticity defects app/ps1 transgenic mice ly-411575–treated tg mice synthetic aβ40s26c dimers ionotropic glutamate receptors amyloid-β supporting information online soluble aβ levels involve elevated levels control wild-type mice synaptic glutamate uptake publisher site pdf
Questions {❓}
- How do soluble oligomers of amyloid β-protein impair hippocampal synaptic plasticity?
Social Networks {👍}(6)
External Links {🔗}(116)
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