NCBI . NLM . NIH . GOV {
}
We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC3343184/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC3343184/. The analysis below is for the second page.
Title[redir]:
Acquired cancer stem cell phenotypes through Oct4-mediated dedifferentiation - PMC
Description:
There is enormous interest to target cancer stem cells (CSCs) for clinical treatment because these cells are highly tumorigenic and resistant to chemotherapy. Oct4 is expressed by CSC-like cells in different types of cancer. However, function of ...
Title[redir]:
Acquired cancer stem cell phenotypes through Oct4-mediated dedifferentiation - PMC
Description:
There is enormous interest to target cancer stem cells (CSCs) for clinical treatment because these cells are highly tumorigenic and resistant to chemotherapy. Oct4 is expressed by CSC-like cells in different types of cancer. However, function of ...
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- Science
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No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.
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What is the average monthly size of ncbi.nlm.nih.gov audience?
π Phenomenal Traffic: 5M - 10M visitors per month
Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
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While many websites aim to make money, others are created to share knowledge or showcase creativity. People build websites for various reasons. This could be one of them. Ncbi.nlm.nih.gov could be getting rich in stealth mode, or the way it's monetizing isn't detectable.
Keywords {π}
cells, oct, doi, expression, melanoma, cell, pubmed, google, scholar, tumor, dedifferentiation, wmoct, stem, showed, protein, data, cancer, control, dedifferentiated, pmc, significantly, hypoxia, human, performed, mice, article, free, gene, increased, induced, supplementary, fig, experiments, shown, reprogramming, differentiation, infected, csc, similar, levels, replicate, analysis, xenografts, phenotypes, expressed, study, capacity, factors, examined, information,
Topics {βοΈ}
naΓ―ve nod/scid/il-Ξ³ mice nod/scid/il-Ξ³ mice total dna-free rna prostate carcinoma-initiating stem reference list ben-porath quantitative rt-pcr showed nod/scid il2rgβ/β mice pmc beta search involve epithelial-mesenchymal transition oct4 promoter-luc construct high nucleus/cytoplasm ratios hif-2alpha regulates oct-4 high-efficiency rna interference psin-ef2-oct4-pur epithelial progenitor/stem cells slow-cycling melanoma cells oct4-sphere-forming cells infectious diseases/national institutes limiting dilution assays quality-checked crna samples colony-forming cells carried advanced human malignancies nerve tissue formation quantitative rt-pcr quantitative rt pcr epithelial-mesenchymal transition oct4-mediated dedifferentiation suresh nod/scid il2rΞ³ oct4-protein-dedifferentiated cells Ξ²-actin gene expression hypoxia inducible factor human embryonic stem methods cell culture oct4-infected 1205lu cells induced pluripotent stem melanoma cell lines adult human fibroblasts neural stem cells melanoma initiating cells publisher site pdf significantly larger compared sphere forming cells sphere-forming cells sphere forming capacity oct4 significantly increases human malignant melanoma tumor-initiating cells western blot analysis human gastric cancer global gene expression
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