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Title[redir]:
Metabolic connections during apoptotic cell engulfment - PMC
Description:
Billions of cells die via apoptosis every day and are swiftly and efficiently removed. When a phagocyte engulfs an apoptotic cell, it essentially doubles its cellular contents, raising the question of how a phagocyte may manage the excess metabolic ...
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Keywords {🔍}
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Topics {✒️}
ulk1-atg13-fip200 pre-initiation complex pmc beta search thioglycollate-elicited peritoneal macrophages t-cell immunoglobulin domain peroxisome-proliferator activated receptors dampen anti-tumor responses ligand-activated transcriptional activators future directions key rac-regulator required pre-doctoral support publisher site pdf apoptotic cell engagement double-edged sword apoptotic cells activates general nutrient sensing excess material acquired atherosclerotic plaque versus continued corpse uptake existing data suggest anti-inflammatory cytokines altered nutrient availability altered nutrient state phagocytic receptor tim-4 apoptotic cell leads partial hypoxic environment subsequent derivatives feed apoptotic cells uptake cellular growth purposes insulin- growth factor nerve growth factor growth factor progranulin turn potentially contribute united states government final edited form extensively characterized eat liver x-receptor existing evidence suggests play important roles conferring properties ranging redox reactions occurs recent study showed elegans mutants lacking progranulin homologue display positive feedback loop general medical sciences american asthma foundation final citable form menendez-gutierrez mp key energy sensor mitochondrial membrane potential
Questions {❓}
- Hence, an interesting, but largely unanswered question is, how does the phagocyte handle the excess metabolic burden acquired from the ingested corpse?
- How does stimulation of PPARs or LXR regulate engulfment?
- In this scenario, how does the ingested cargo regulate the phagocytic machinery and continued clearance by phagocytes?
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