NCBI . NLM . NIH . GOV {
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We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC2772133/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC2772133/. The analysis below is for the second page.
Title[redir]:
Amyloid β-Protein Dimers Isolated Directly from Alzheimer Brains Impair Synaptic Plasticity and Memory - PMC
Description:
Alzheimer’s disease (AD) constitutes a rising threat to public health. Despite extensive research in cellular and animal models, identifying the pathogenic agent present in the human brain and showing that it confers key features of AD have not been ...
Title[redir]:
Amyloid β-Protein Dimers Isolated Directly from Alzheimer Brains Impair Synaptic Plasticity and Memory - PMC
Description:
Alzheimer’s disease (AD) constitutes a rising threat to public health. Despite extensive research in cellular and animal models, identifying the pathogenic agent present in the human brain and showing that it confers key features of AD have not been ...
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Keywords {🔍}
tbs, doi, fig, ltp, soluble, dimers, con, extracts, brain, pubmed, google, scholar, amyloid, fractions, sec, supp, pmc, data, slices, cores, article, human, cortex, synapse, extract, tbssec, free, spine, synaptic, disease, oligomers, effects, species, monomers, loss, insoluble, summary, red, core, plaque, ipwb, treated, kda, protein, subjects, density, antibodies, prepared, control, significantly,
Topics {✒️}
n-methyl-d-aspartate receptor blockade supplementary materials nihms154255-supplement-supplement_1 supplementary material nihms154255-supplement-supplement_1 alters long-term potentiation wild-type synthetic aβ40 rescue long-term potentiation pmc beta search synthetic amyloid beta-peptide homosynaptic long-term depression fepsp2 slope/fepsp1 slope plaque-rich ad brain remaining core-rich pellet aβ-mediated spine loss16 amyloid β-protein high-level neuronal expression long-term potentiation impaired synaptic plasticity plaque-rich ad cortex abeta oligomer-induced aberrations gfp-transfected pyramidal cells sensitive immunoprecipitation/western blotting aβ-immunoreactive species migrating long-term depression epitope-specific aβ antibodies free n-terminus impair synaptic plasticity called tbs-tx extract low-molecular-weight proteins inhibit hippocampal ltp amyloid plaque fibers fa/naoh core extracts strongly dose-dependent ad tbs-sec treatment tbs-insoluble pellet prepared ip/wb analyses revealed influence synaptic plasticity alter synaptic plasticity homogenized tbs-insoluble pellets tris-buffered saline neurotransmitter release probability supplementary materials included tbs-soluble aβ correlated time-dependent increase ad amyloid cores c-terminal antibodies neutralized insoluble amyloid plaques oxidized aβ40-s26c scattered amyloid plaques sec-enriched tbs extract paired-pulse responses
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