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NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers
  14. CDN Services

We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC2711514/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC2711514/. The analysis below is for the second page.

Title[redir]:
MITOCHONDRIAL FRAGMENTATION IN NEURODEGENERATION - PMC
Description:
Mitochondria are remarkably dynamic organelles that migrate, divide and fuse. Cycles of mitochondrial fission and fusion ensure metabolite and mitochondrial DNA (mtDNA) mixing and dictate organelle shape, number and bioenergetic functionality. There ...

Matching Content Categories {📚}

  • Science
  • Fitness & Wellness
  • Health & Fitness

Content Management System {📝}

What CMS is ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 6,399,052 visitors per month in the current month.

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How Does Ncbi.nlm.nih.gov Make Money? {💸}

We don't see any clear sign of profit-making.

Some websites aren't about earning revenue; they're built to connect communities or raise awareness. There are numerous motivations behind creating websites. This might be one of them. Ncbi.nlm.nih.gov could be getting rich in stealth mode, or the way it's monetizing isn't detectable.

Keywords {🔍}

mitochondrial, doi, pubmed, google, scholar, fission, fusion, cell, mitochondria, pmc, mutations, free, article, drp, mtdna, mfn, cells, disease, protein, opa, membrane, biol, death, neurons, neurodegenerative, proteins, function, gtpase, role, domain, exercise, neuronal, diseases, apoptosis, dysfunction, oxidative, dynamics, figure, outer, addition, energy, ros, human, mutant, mol, important, model, muscle, fragmentation, stress,

Topics {✒️}

charcot-marie-tooth subtype 2a calpain-regulated p35/cdk5 plays charcot-marie-tooth syndrome charcot-marie-tooth disease prevent bax/bak-dependent apoptosis drp-1-mediated mitochondrial fragmentation camp-dependent protein kinase nuclear-encoded mitochondrial metalloprotease pmc beta search possess wild-type mtdna ngf-deprived sympathetic neurons small ubiquitin-related modifier reactive oxygen species dominant-negative drp1 mutant multiple proteolytically-processed isoforms wild-type satellite cells egl-1-induced cell death mitochondrial fission activated neuron expressing dsred-mito injury-induced cell death promotes mitochondrial biogenesis type iii pcd common post-translational modification intramitochondrial dynamin-related gtpase cyclin-dependent kinases participate cell-type specific pathogenesis anti-apoptotic actions opa1-mediated optic atrophy classical caspase-dependent apoptosis drp1-dependent mitochondrial fission dynamin-related protein drp1 ca2+-mediated neurotransmitter release mfn2-mediated mitochondrial fusion extensive ros-defense system native ros-defense mechanisms free radical generation native ros-defense system dynamin-related protein result apoptotic cell-death pathways cyclin-dependent kinase 5 n-terminal gtpase domain dynamin-related gtpase mgm1 m-aaa protease amyotrophic lateral sclerosis ganglioside-induced differentiation ganglioside-induced differentiation drp1 partially protected wild-type cells dominant-negative mutation neurons selectively degenerate

Questions {❓}

  • 122 Does exercise improve mitochondrial function?
  • ARE CHANGES IN MITOCHONDRIAL DYNAMICS REVERSIBLE?
  • Are mitochondria critical in the pathogenesis of Alzheimer’s disease?
  • But why can some neurons survive while others cannot?
  • Does resistance exercise induce gene shifting and a decrease in mtDNA mutations in healthy older adults?
  • Mitochondrial dysfunction is a characteristic of many neurodegenerative disorders, but is mitochondrial fission activated in- or causally related to sporadic neurodegenerative disease?
  • The dynamins: redundant or distinct functions for an expanding family of related GTPases?
  • What can compensate for too much fission?
  • What could account for this apparent contradiction?
  • Why do we still have a maternally inherited mitochondrial DNA?

Social Networks {👍}(6)

External Links {🔗}(311)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • jQuery
  • jQuery module (jquery-3.6.0)
  • Zoom.js

Emails and Hosting {✉️}

Mail Servers:

  • nihcesxway.hub.nih.gov
  • nihcesxway2.hub.nih.gov
  • nihcesxway3.hub.nih.gov
  • nihcesxway4.hub.nih.gov
  • nihcesxway5.hub.nih.gov

Name Servers:

  • dns1-ncbi.ncbi.nlm.nih.gov
  • dns2-ncbi.ncbi.nlm.nih.gov
  • lhcns1.nlm.nih.gov
  • lhcns2.nlm.nih.gov

CDN Services {📦}

  • Ncbi

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