NCBI . NLM . NIH . GOV {
}
We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC1606493/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC1606493/. The analysis below is for the second page.
Title[redir]:
Consequences of Copper Accumulation in the Livers of the Atp7b−/− (Wilson Disease Gene) Knockout Mice - PMC
Description:
Wilson disease is a severe genetic disorder associated with intracellular copper overload. The affected gene, ATP7B, has been identified, but the molecular events leading to Wilson disease remain poorly understood. Here, we demonstrate that ...
Title[redir]:
Consequences of Copper Accumulation in the Livers of the Atp7b−/− (Wilson Disease Gene) Knockout Mice - PMC
Description:
Wilson disease is a severe genetic disorder associated with intracellular copper overload. The affected gene, ATP7B, has been identified, but the molecular events leading to Wilson disease remain poorly understood. Here, we demonstrate that ...
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Keywords {🔍}
copper, atpb, doi, mice, liver, google, scholar, pubmed, figure, weeks, animals, disease, serum, livers, nuclear, accumulation, nuclei, control, ceruloplasmin, activity, pathology, concentration, hepatocellular, bile, mouse, fraction, observed, elevated, cell, normal, hepatocytes, ducts, marked, protein, lec, rats, fractions, staining, wilsons, model, patients, inflammation, dna, synthesis, development, hepatic, levels, age, wilson, cholangiocarcinoma,
Topics {✒️}
copper-transporting p-type atpase goat anti-human-ceruloplasmin antibody abc-type transporter mrp2 loose-fitting dounce homogenizer long-evans cinnamon rat benign bile-duct stricture long-term undiagnosed wilson low-molecular weight molecules low-molecular weight compounds pmc beta search long-evans cinnamon rats pmc copyright notice [google scholar] duchrow strong genotype-phenotype correlations human copper-transporting atpase mouse monoclonal anti-na/ wide-spread hepatocellular injury er/golgi-enriched fraction high nuclear-cytoplasmic ratio copper-handling mechanisms independent references pena mm publisher site pdf study copper-induced tumorigenesis ceruloplasmin-mediated oxidase activity numerous wd-causing mutations plasma membrane-enriched fractions low-serum oxidase activity species-specific hepatic response plasma membrane-enriched fraction license information pmcid atp7b gene-coding region mouse apo-ceruloplasmin appears age-matched ko livers copper-transporting atpase rabbit anti-cytochrome similar age-dependent decrease toxic milk mouse total membrane-bound copper anti-mouse antibody dilated bile canaliculus distinct phases depending copper levels decreased increased copper excretion toxic milk mice low oxidase activity severe genetic disorder sam jackson park molecular weight standards intact bile duct bile duct ligation
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