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Keywords {🔍}

stat, doi, pubmed, google, scholar, cell, activation, cells, proteins, signaling, growth, stats, pmc, transformation, constitutive, apoptosis, activated, cancer, jak, tumor, kinase, receptor, expression, activity, free, article, factor, tyrosine, gene, protein, cellular, science, regulation, human, nlm, tumorigenesis, tumors, cancers, lines, phosphorylation, oncogene, found, sjonc, sci, ncbi, search, primary, kinases, normal, mice,

Topics {✒️}

defective tnf-alpha-induced apoptosis men2a-ret-induced cellular transformation carcinogen-treated wild-type animals spontaneously dimerizing-mutant form stat6-deficient animals reveal anaplastic lymphoma kinase-positive tumor-derived cell lines ligand tgf-α stimulates tissue-specific gene targeting chemical carcinogen-induced tumorigenesis cancer-derived cell lines pmc beta search shows growth-suppression activity tumor-specific growth inhibition ret requires phosphorylation pmc copyright notice stat5a/5b double knockouts v-abl–induced leukemias bcr/abl-induced transformation chimeric tel-jak proteins stat1-deficient mice develop stat1-null cells due jak-stat signaling pathway jak-stat signaling pathways stat3-mediated myc expression publisher site pdf tel-jak fusion protein antiapoptotic proteins bcl-xl ifn-gamma target genes constitutive kinase activity tel-jak2 fusion protein proteins cyclin d1 growth factor receptor growth factor signaling cdk inhibitor p21 stat1-deficient animals il-10–mediated signaling methylation-induced silencing /null-cell lymphoma license information pmcid tumor surveillance capabilities growth factor receptors inhibit tumor growth receptor tyrosine kinase proangiogenic factor vegf il-4–mediated proliferation cell growth differ lymphoproliferative disease induced block stat3 expression p53 single mutants

Questions {❓}

• What does Stat3 do?

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Name Servers:

• dns1-ncbi.ncbi.nlm.nih.gov
• dns2-ncbi.ncbi.nlm.nih.gov
• lhcns1.nlm.nih.gov
• lhcns2.nlm.nih.gov

CDN Services {📦}

• Ncbi