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nature, cell, death, article, content, prohibitin, research, cookies, privacy, upregulated, data, information, differentiation, apoptosis, welburn, open, advertising, july, rack, trypanosomes, terminally, differentiated, susan, murphy, genes, trypanosoma, programmed, access, prodigiosin, permissions, optional, analysis, media, personal, parties, policy, journals, original, paper, published, homologues, induced, undergo, naturally, occurring, forms, noel, cite, identified, form,

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nature portfolio permissions reprints privacy policy nature tsetse research group advertising programmed cell death tissue research social media proteomics-based prognostic signature cell death pathways cell death differ personal data data protection permissions cona-induced apoptosis putative death domain cell-cycle control gene rack homologues explore content similar content privacy journals search log undergo apoptosis european economic area proto-oncogene originally activated protein kinase active nuclear transport mutations synthetic-lethal accepting optional cookies multicyclic prodigiosin formed content manage preferences https miltefosine-sensitive cell survival thompson cell article welburn prodigiosin cyclization genes article cite knight rights optional cookies article //doi choices essential cookies cookies skip trypanosomes induced kenya susan

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         headline:Prohibitin and RACK homologues are up-regulated in trypanosomes induced to undergo apoptosis and in naturally occurring terminally differentiated forms
         description:Two genes have been identified as up-regulated late during ConA-induced apoptosis in procyclic form Trypanosoma brucei rhodesiense. The first represents a homologue of prohibitin, a proto-oncogene originally described in mammals and subsequently in yeast, which is involved in cell-cycle control and senescence. The Trypanosoma prohibitin homologue appears to contain within it a putative death domain. The second gene, homologous to a family of regulatory proteins which are receptors for activated protein kinase C (RACKs), is also shown to be up-regulated in terminally differentiated bloodstream form trypanosomes. These are the first endogenous genes to be identified as up-regulated in programmed cell death (PCD) in unicellular organisms.
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      headline:Prohibitin and RACK homologues are up-regulated in trypanosomes induced to undergo apoptosis and in naturally occurring terminally differentiated forms
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