NATURE . COM {}

Matching Content Categories {📚}

• Telecommunications
• Health & Fitness
• Science

Content Management System {📝}

What CMS is nature.com built with?

Custom-built

No common CMS systems were detected on Nature.com, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of nature.com audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

check SE Ranking
check Ahrefs
check Similarweb
check Ubersuggest
check Semrush

How Does Nature.com Make Money? {💸}

Display Ads {🎯}

Direct Advertisers (10)

Reseller Advertisers (38)

How Much Does Nature.com Make? {💰}

Display Ads {🎯}

$63,100 per month
Our analysis indicates Nature.com generates between $42,042 and $115,616 monthly online from display ads.

Keywords {🔍}

cancer, cell, res, biol, nature, chem, oncogene, fulda, debatin, nat, med, wang, apoptosis, death, access, mol, article, science, krammer, sci, natl, eldeiry, van, content, rev, exp, kroemer, cookies, clin, srinivasula, ther, proc, acad, usa, differ, friesen, liu, privacy, anticancer, chemotherapy, open, zhivotovsky, sun, int, zhang, yang, data, extrinsic, intrinsic, pathways,

Topics {✒️}

nature portfolio permissions reprints cancer research privacy policy advertising social media recent development author information authors develop molecular-targeted therapies nature 406 nature 407 nature 9 nature 349 nature 435 nature 396 nature 410 nature 408 nature author correspondence personal data data protection permissions springerlink instant access privacy explore content subscription content european economic area physiological growth control suicide gene therapy institutional subscriptions read fernandez de mattos mcl-1 inhibitor synergizes ]pyrimidine derivatives farid accepting optional cookies el-deiry ws neck cancer pathogenesis wilhelm-sander-stiftung kind-phillipp-stiftung programmed cell death journals search log de brabander jk dimanche-boitrel mt fernandez-salas ea manage preferences simoes-wust ap van noesel mm content molecular events molecular docking issue learn

Questions {❓}

• Platinum drugs and taxanes: can we overcome resistance?

Schema {🗺️}

WebPage:
      mainEntity:
         headline:Extrinsic versus intrinsic apoptosis pathways in anticancer chemotherapy
         description:Apoptosis or programmed cell death is a key regulator of physiological growth control and regulation of tissue homeostasis. One of the most important advances in cancer research in recent years is the recognition that cell death mostly by apoptosis is crucially involved in the regulation of tumor formation and also critically determines treatment response. Killing of tumor cells by most anticancer strategies currently used in clinical oncology, for example, chemotherapy, γ-irradiation, suicide gene therapy or immunotherapy, has been linked to activation of apoptosis signal transduction pathways in cancer cells such as the intrinsic and/or extrinsic pathway. Thus, failure to undergo apoptosis may result in treatment resistance. Understanding the molecular events that regulate apoptosis in response to anticancer chemotherapy, and how cancer cells evade apoptotic death, provides novel opportunities for a more rational approach to develop molecular-targeted therapies for combating cancer.
         datePublished:2006-08-07T00:00:00Z
         dateModified:2006-08-07T00:00:00Z
         pageStart:4798
         pageEnd:4811
         sameAs:https://doi.org/10.1038/sj.onc.1209608
         keywords:
            apoptosis
            death receptor
            mitochondria
            cancer
            chemotherapy
            Medicine/Public Health
            general
            Internal Medicine
            Cell Biology
            Human Genetics
            Oncology
            Apoptosis
         image:
            https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fsj.onc.1209608/MediaObjects/41388_2006_Article_BF1209608_Fig1_HTML.jpg
            https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fsj.onc.1209608/MediaObjects/41388_2006_Article_BF1209608_Fig2_HTML.jpg
         isPartOf:
            name:Oncogene
            issn:
               1476-5594
               0950-9232
            volumeNumber:25
            type:
               Periodical
               PublicationVolume
         publisher:
            name:Nature Publishing Group UK
            logo:
               url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
               type:ImageObject
            type:Organization
         author:
               name:S Fulda
               affiliation:
                     name:University Children's Hospital
                     address:
                        name:University Children's Hospital, Ulm, Germany
                        type:PostalAddress
                     type:Organization
               email:[email protected]
               type:Person
               name:K-M Debatin
               affiliation:
                     name:University Children's Hospital
                     address:
                        name:University Children's Hospital, Ulm, Germany
                        type:PostalAddress
                     type:Organization
               type:Person
         isAccessibleForFree:
         hasPart:
            isAccessibleForFree:
            cssSelector:.main-content
            type:WebPageElement
         type:ScholarlyArticle
      context:https://schema.org
ScholarlyArticle:
      headline:Extrinsic versus intrinsic apoptosis pathways in anticancer chemotherapy
      description:Apoptosis or programmed cell death is a key regulator of physiological growth control and regulation of tissue homeostasis. One of the most important advances in cancer research in recent years is the recognition that cell death mostly by apoptosis is crucially involved in the regulation of tumor formation and also critically determines treatment response. Killing of tumor cells by most anticancer strategies currently used in clinical oncology, for example, chemotherapy, γ-irradiation, suicide gene therapy or immunotherapy, has been linked to activation of apoptosis signal transduction pathways in cancer cells such as the intrinsic and/or extrinsic pathway. Thus, failure to undergo apoptosis may result in treatment resistance. Understanding the molecular events that regulate apoptosis in response to anticancer chemotherapy, and how cancer cells evade apoptotic death, provides novel opportunities for a more rational approach to develop molecular-targeted therapies for combating cancer.
      datePublished:2006-08-07T00:00:00Z
      dateModified:2006-08-07T00:00:00Z
      pageStart:4798
      pageEnd:4811
      sameAs:https://doi.org/10.1038/sj.onc.1209608
      keywords:
         apoptosis
         death receptor
         mitochondria
         cancer
         chemotherapy
         Medicine/Public Health
         general
         Internal Medicine
         Cell Biology
         Human Genetics
         Oncology
         Apoptosis
      image:
         https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fsj.onc.1209608/MediaObjects/41388_2006_Article_BF1209608_Fig1_HTML.jpg
         https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fsj.onc.1209608/MediaObjects/41388_2006_Article_BF1209608_Fig2_HTML.jpg
      isPartOf:
         name:Oncogene
         issn:
            1476-5594
            0950-9232
         volumeNumber:25
         type:
            Periodical
            PublicationVolume
      publisher:
         name:Nature Publishing Group UK
         logo:
            url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
            type:ImageObject
         type:Organization
      author:
            name:S Fulda
            affiliation:
                  name:University Children's Hospital
                  address:
                     name:University Children's Hospital, Ulm, Germany
                     type:PostalAddress
                  type:Organization
            email:[email protected]
            type:Person
            name:K-M Debatin
            affiliation:
                  name:University Children's Hospital
                  address:
                     name:University Children's Hospital, Ulm, Germany
                     type:PostalAddress
                  type:Organization
            type:Person
      isAccessibleForFree:
      hasPart:
         isAccessibleForFree:
         cssSelector:.main-content
         type:WebPageElement
["Periodical","PublicationVolume"]:
      name:Oncogene
      issn:
         1476-5594
         0950-9232
      volumeNumber:25
Organization:
      name:Nature Publishing Group UK
      logo:
         url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
         type:ImageObject
      name:University Children's Hospital
      address:
         name:University Children's Hospital, Ulm, Germany
         type:PostalAddress
      name:University Children's Hospital
      address:
         name:University Children's Hospital, Ulm, Germany
         type:PostalAddress
ImageObject:
      url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
Person:
      name:S Fulda
      affiliation:
            name:University Children's Hospital
            address:
               name:University Children's Hospital, Ulm, Germany
               type:PostalAddress
            type:Organization
      email:[email protected]
      name:K-M Debatin
      affiliation:
            name:University Children's Hospital
            address:
               name:University Children's Hospital, Ulm, Germany
               type:PostalAddress
            type:Organization
PostalAddress:
      name:University Children's Hospital, Ulm, Germany
      name:University Children's Hospital, Ulm, Germany
WebPageElement:
      isAccessibleForFree:
      cssSelector:.main-content

Social Networks {👍}(1)

• https://twitter.com/oncogenejournal

External Links {🔗}(17)

• Explore the financials of https://link.springer.com/article/10.1038/sj.onc.1209608?utm_source=nature&utm_medium=referral&utm_campaign=buyArticle
• What's the financial gain of https://citation-needed.springer.com/v2/references/10.1038/sj.onc.1209608?format=refman&flavour=references?
• Monthly income for https://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=search&term=S%20Fulda
• How much income does https://scholar.google.co.uk/scholar?as_q=&num=10&btnG=Search+Scholar&as_epq=&as_oq=&as_eq=&as_occt=any&as_sauthors=%22S%20Fulda%22&as_publication=&as_ylo=&as_yhi=&as_allsubj=all&hl=en have?
• How much cash flow does https://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=search&term=K-M%20Debatin have monthly?
• What's the profit of https://scholar.google.co.uk/scholar?as_q=&num=10&btnG=Search+Scholar&as_epq=&as_oq=&as_eq=&as_occt=any&as_sauthors=%22K-M%20Debatin%22&as_publication=&as_ylo=&as_yhi=&as_allsubj=all&hl=en?
• How much cash flow does https://s100.copyright.com/AppDispatchServlet?title=Extrinsic%20versus%20intrinsic%20apoptosis%20pathways%20in%20anticancer%20chemotherapy&author=S%20Fulda%20et%20al&contentID=10.1038%2Fsj.onc.1209608©right=Springer%20Nature%20Limited&publication=0950-9232&publicationDate=2006-08-07&publisherName=SpringerNature&orderBeanReset=true have monthly?
• How much revenue does https://citation-needed.springer.com/v2/references/10.1038/sj.onc.1209608?format=refman&flavour=citation bring in?
• Learn about the earnings of https://doi.org/10.1007/s13273-022-00327-w
• How much profit does https://doi.org/10.1007/s10895-024-03587-y make?
• How much does https://doi.org/10.1186/s12859-023-05206-2 bring in each month?
• Get to know what's the income of https://doi.org/10.1186/s10020-022-00565-7
• https://doi.org/10.1186/s13065-023-01014-0's total income per month
• How much does https://www.protocols.io/ make?
• How much profit does https://www.natureindex.com/ make?
• Find out how much http://www.naturechina.com earns monthly
• What's the income generated by https://www.natureasia.com/ja-jp each month?

Analytics and Tracking {📊}

• Google Tag Manager

Libraries {📚}

• Prism.js
• Zoom.js

Emails and Hosting {✉️}

Mail Servers:

• mxa-002c5801.gslb.pphosted.com
• mxb-002c5801.gslb.pphosted.com

Name Servers:

• pdns1.ultradns.net
• pdns2.ultradns.net
• pdns3.ultradns.org
• pdns4.ultradns.org
• pdns5.ultradns.info
• pdns6.ultradns.co.uk

CDN Services {📦}

• Crossref