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Keywords {🔍}

cells, ilra, doi, otii, wildtype, mice, fig, effector, day, cell, pubmed, google, scholar, differentiation, production, stat, ilr, expression, lymph, data, cns, response, reduced, shown, eae, postimmunization, node, vivo, days, absence, pmc, required, stimulation, results, time, numbers, compared, cytokine, dln, immunol, signaling, frequency, found, bone, marrow, immunization, blood, article, inflammation, presence,

Topics {✒️}

pacific-blue conjugated anti-cd4 il-7r alpha-expressing cells ifn-γ+il-17− wild type apc-conjugated ifn-γ pmc beta search wild-type mog-specific cd4+ pe-conjugated anti-il-2 supernatant ifn-γ concentrations bone marrow chimeras reduced il-23r-deficient mice cd4+ ifn-γ production cd4+ stat3fl/fl cells ifn-γ+cd4+ cells neutralizing anti-il-23r il-23r deficiency restricted wild-type response progressed stat-dependent cytokine signals fewer mog-specific cd4+ ifn-γ+il-17− cells ifn-γ+il-17+ cells reconstituting bone marrow cell-mediated intestinal inflammation autocrine growth factor schering plough biopharma antigen-specific wild-type responding antigen-specific cells wild-type otii cells il-20r2-dependent mechanisms support pstat3-mediating signals wild-type cd4+ cells il-23r deficiency resulted total otii–cd4+ cells il-17+ wild-type cd4+ stat3-dependent signaling pathway supplementary materials included time points post-immunization il-23ra−/− otii cells pathogens mycobacterium tuberculosis list biological laboratories il-23–il-17 immune pathway mog-specific il-17 production il-23p19-deficient mice induces autoimmune inflammation transcription factor stat317 il-23 signaling began license information pmcid ifn-γ production important pro-inflammatory effects human immune disorders upregulate il-7rα expression

Questions {❓}

• The following question then arises: is IL-23 merely driving proliferation of developing TH-17 cells and thus facilitating their differentiation?

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