PMC . NCBI . NLM . NIH . GOV {
}
We are analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC2586288/.
Title:
Huntingtin Modulates Transcription, Occupies Gene Promoters In Vivo, and Binds Directly to DNA in a Polyglutamine-Dependent Manner - PMC
Description:
Transcriptional dysregulation is a central pathogenic mechanism in Huntington
Website Age:
27 years and 8 months (reg. 1997-10-02).
Title:
Huntingtin Modulates Transcription, Occupies Gene Promoters In Vivo, and Binds Directly to DNA in a Polyglutamine-Dependent Manner - PMC
Description:
Transcriptional dysregulation is a central pathogenic mechanism in Huntington
Website Age:
27 years and 8 months (reg. 1997-10-02).
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🌠 Phenomenal Traffic: 5M - 10M visitors per month
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Keywords {🔍}
htt, binding, dna, doi, transcription, mutant, gene, sthdh, expression, cell, wildtype, factor, genes, google, scholar, lines, pubmed, factors, protein, transcriptional, fig, probes, present, chip, disease, data, genomic, proteins, huntingtons, exon, huntingtin, mrna, open, promoters, receptor, shown, activity, regions, conformation, brain, mouse, bound, pmc, nuclear, chiponchip, presence, increased, directly, polyq, altered,
Topics {✒️}
[google scholar] luthi-carter low-molecular-weight dna species mutant-htt-mediated transcriptional dysregulation anti-acetylated histone h3 high-molecular-weight fragments low-molecular-weight fragments order “gene-function word-gene interferon-γ activation factor high-molecular-weight state high-molecular-weight fragment steady-state florescent measurements peroxisome proliferator-activated receptor ccaat/enhancer binding protein low-molecular-weight species pmc beta search high-molecular-weight dna c-terminal binding protein interferon-stimulated response element double-stranded dna fragments binding-site estimation suite activity-dependent transcriptional activation ataxin-7 carboxy-terminal region ligation-mediated pcr amplification post-transcriptional mrna stability sequence-specific preference existed comparing htt-promoter binding pmc copyright notice mutant/wild-type htt binding poly-l-lysine molecules leaving protein–dna complexes blue/white colony selection gene–transcription factor cocitations full-length cell models double-stranded plasmid dna [google scholar] che identifying htt-enriched regions nonspecific dna-binding domain aberrant htt–dna binding nrse-controlled neuronal genes polyglutamine-dependent manner caroline individual protein–protein interaction htt-induced transcriptional events gene-promoter-specific primers polyglutamine-expanded htt exhibits gst-hd53q/gst-hd20q htt–dna binding events conformation-dependent fluorescent quenching endogenous wild-type protein interrogating chip-immunoenriched dna htt-chip product analyses
Social Networks {👍}(6)
External Links {🔗}(213)
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