Here's how LINK.SPRINGER.COM makes money* and how much!

*Please read our disclaimer before using our estimates.
Loading...

LINK . SPRINGER . COM {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Link.springer.com Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Schema
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. CDN Services

We are analyzing https://link.springer.com/chapter/10.1007/978-3-540-72167-3_1.

Title:
Toll-Like Receptors (TLRs) and Their Ligands | SpringerLink
Description:
The innate immune system is an evolutionally conserved host defense mechanism against pathogens. Innate immune responses are initiated by pattern recognition receptors (PRRs), which recognize microbial components that are essential for the survival of the...
Website Age:
28 years and 1 months (reg. 1997-05-29).

Matching Content Categories {πŸ“š}

  • Education
  • Science
  • Mobile Technology & AI

Content Management System {πŸ“}

What CMS is link.springer.com built with?

Custom-built

No common CMS systems were detected on Link.springer.com, and no known web development framework was identified.

Traffic Estimate {πŸ“ˆ}

What is the average monthly size of link.springer.com audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

check SE Ranking
check Ahrefs
check Similarweb
check Ubersuggest
check Semrush

How Does Link.springer.com Make Money? {πŸ’Έ}

We see no obvious way the site makes money.

The purpose of some websites isn't monetary gain; they're meant to inform, educate, or foster collaboration. Everyone has unique reasons for building websites. This could be an example. Link.springer.com could have a money-making trick up its sleeve, but it's undetectable for now.

Keywords {πŸ”}

google, scholar, cas, pubmed, tolllike, receptor, immunol, drosophila, akira, immune, recognition, innate, tlr, immunity, receptors, response, signaling, nature, nat, responses, med, biol, role, malaria, activation, infection, protein, hoffmann, cell, takeuchi, takeda, toll, cells, science, tlrs, exp, infect, host, defense, hemmi, virus, gene, chapter, bacteria, proc, natl, acad, sci, usa, cruzi,

Topics {βœ’οΈ}

herpes simplex virus-2 ubiquitin-proteasome pathway represses pgrp-lc act synergistically ifn-gamma-primed macrophages syk-dependent cytokine induction murine retroviruses activate double-stranded rna reveals cell-mediated immune responses kurt-jones ea innate anti-fungal immunity jnk-mediated immune response receptor signalling path-ways control dorsoventral patterning activates nk cells cytokine production triggered major beta-glucan receptor species-specific recognition respiratory syncytial virus privacy choices/manage cookies innate antiviral responses activates antibacterial defense innate immune responses toll-mediated antifungal defense plasmacytoid dendritic cells double-stranded rna single-stranded rna reactive oxygen production lipopolysaccharide-signaling complex drosophila immune deficiency gram-positive bacteria van der graaf humoral immune responses life sciences biomedical antibacterial immune responses innate immune response interferon-alpha protein relish regulates innate immune defense pattern recognition pathway innate immune recognition sp Γ€tzle de arruda lb trypanosoma cruzi glycoinositolphospholipids mediating immune response bacterial flagellin innate immune system human cerebral malaria mouse cytomegalovirus infection distinct antipathogen responses drosophila immune response

Questions {❓}

  • Netea MG, van Deuren M, Kullberg BJ, Cavaillon JM, Van der Meer JW (2002b) Does the shape of lipid A determine the interaction of LPS with Toll-like receptors?

Schema {πŸ—ΊοΈ}

ScholarlyArticle:
      headline:Toll-Like Receptors (TLRs) and Their Ligands
      pageEnd:20
      pageStart:1
      image:https://media.springernature.com/w153/springer-static/cover/book/978-3-540-72167-3.jpg
      genre:
         Biomedical and Life Sciences
         Biomedical and Life Sciences (R0)
      isPartOf:
         name:Toll-Like Receptors (TLRs) and Innate Immunity
         isbn:
            978-3-540-72167-3
            978-3-540-72166-6
         type:Book
      publisher:
         name:Springer Berlin Heidelberg
         logo:
            url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
            type:ImageObject
         type:Organization
      author:
            name:Satoshi Uematsu
            affiliation:
            type:Person
            name:Shizuo Akira
            affiliation:
                  name:Osaka University
                  address:
                     name:Department of Host Defense, Research Institute for Microbial Diseases, Osaka University, Suita Osaka, Japan
                     type:PostalAddress
                  type:Organization
            type:Person
      keywords:Respiratory Syncytial Virus, West Nile Virus, Cerebral Malaria, Trypanosoma Cruzi, Toll Pathway
      description:The innate immune system is an evolutionally conserved host defense mechanism against pathogens. Innate immune responses are initiated by pattern recognition receptors (PRRs), which recognize microbial components that are essential for the survival of the microorganism. PRRs are germline-encoded, nonclonal, and expressed constitutively in the host. Different PRRs react with specific ligands and lead to distinct antipathogen responses. Among them, Toll-like receptors (TLRs) are capable of sensing organisms ranging from bacteria to fungi, protozoa, and viruses, and they play a major role in innate immunity. Here, we review the mechanism of pathogen recognition by TLRs.
      datePublished:2008
      isAccessibleForFree:
      hasPart:
         isAccessibleForFree:
         cssSelector:.main-content
         type:WebPageElement
      context:https://schema.org
Book:
      name:Toll-Like Receptors (TLRs) and Innate Immunity
      isbn:
         978-3-540-72167-3
         978-3-540-72166-6
Organization:
      name:Springer Berlin Heidelberg
      logo:
         url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
         type:ImageObject
      name:Osaka University
      address:
         name:Department of Host Defense, Research Institute for Microbial Diseases, Osaka University, Suita Osaka, Japan
         type:PostalAddress
ImageObject:
      url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
Person:
      name:Satoshi Uematsu
      affiliation:
      name:Shizuo Akira
      affiliation:
            name:Osaka University
            address:
               name:Department of Host Defense, Research Institute for Microbial Diseases, Osaka University, Suita Osaka, Japan
               type:PostalAddress
            type:Organization
PostalAddress:
      name:Department of Host Defense, Research Institute for Microbial Diseases, Osaka University, Suita Osaka, Japan
WebPageElement:
      isAccessibleForFree:
      cssSelector:.main-content

External Links {πŸ”—}(270)

Analytics and Tracking {πŸ“Š}

  • Google Tag Manager

Libraries {πŸ“š}

  • Clipboard.js

CDN Services {πŸ“¦}

  • Pbgrd

4.56s.