We are analyzing https://link.springer.com/chapter/10.1007/978-3-540-69184-6_3.

Title:
The Death Receptors | SpringerLink
Description:
In recent years apoptosis, also called programmed cell death, has been recognized to be the physiological way for a nucleated animal cell to die. Apoptosis takes care of unwanted, injured or virus-infected cells (Farber 1994; Collins 1995). Autoreactive T and B cells...
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Keywords {🔍}

google, scholar, pubmed, cas, article, cell, apoptosis, death, receptor, necrosis, factor, tumor, biol, fas, protein, human, chem, cells, tnf, immunol, nature, activation, science, krammer, ligand, antigen, gene, virus, family, protease, dixit, proc, acad, sci, usa, natl, med, peter, mice, domain, signaling, exp, expression, inhibition, apoptotic, virol, wang, member, smith, receptors,

Topics {✒️}

inhibits interleukin-lbeta-converting enzyme jan paul medema prevent il-lbeta-induced fever releases tumour-necrosis factor-alpha tumor necrosis factor-beta interleukin-ibeta converting enzyme tumor necrosis factor-alpha nf-kappab/rel induces apoptosis il-1 beta-converting enzyme death protease cpp32/yama/apopain death-inducing signaling complex tnfr2-traf signaling complex uv-responsive protein kinase lamin-cleaving enzyme mch2alpha nf-kappab signaling pathway cell receptor/cd3 complex protects virus-infected cells mort1/fadd-interacting protease degli-esposti ma tnf/ngf receptor family tie anti-fas antibody virus-induced ced-3/ice ice/ced-3 gene family p55-mediated death signaling p38 signaling pathways alternative pre-mrna splicing cells bearing alpha/beta noncytotoxic traf2-dependent pathway tumor necrosis factor monoclonal anti-fas antibody protein kinase rip sertoli cell-enriched fractions 35-kilodalton protein gene inhibits nf-kappab activation tumour necrosis factor activates multiple ced-3/ice attenuating p53-dependent apoptosis c-jun activation domain cell receptor-induced apoptosis lymphoid-specific death domain cancer therapy-induced apoptosis protein kinase stimulated protein kinase raf-1 tradd-fadd interactions define fas/apo-1 protease mch5 apoptotic mammalian ced-3/ice apoptosis-inhibiting baculovirus gene adenovirus protein e1b19kd fas ligand-induced apoptosis human immunodeficiency virus

Questions {❓}

Peter ME, Medema JP, Krammer PH (1997b) Does the Caenorhabditis elegans protein CED-4 contain a region of homology to the mammalian death effector domain?

Schema {🗺️}

ScholarlyArticle:
      headline:The Death Receptors
      pageEnd:63
      pageStart:25
      image:https://media.springernature.com/w153/springer-static/cover/book/978-3-540-69184-6.jpg
      genre:
         Biomedical and Life Sciences
         Biomedical and Life Sciences (R0)
      isPartOf:
         name:Apoptosis: Biology and Mechanisms
         isbn:
            978-3-540-69184-6
            978-3-662-21623-1
         type:Book
      publisher:
         name:Springer Berlin Heidelberg
         logo:
            url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
            type:ImageObject
         type:Organization
      author:
            name:Marcus E. Peter
            affiliation:
                  name:German Cancer Research Center
                  address:
                     name:Tumor Immunology Program, German Cancer Research Center, Heidelberg, Germany
                     type:PostalAddress
                  type:Organization
            type:Person
            name:Carsten Scaffidi
            affiliation:
                  name:German Cancer Research Center
                  address:
                     name:Tumor Immunology Program, German Cancer Research Center, Heidelberg, Germany
                     type:PostalAddress
                  type:Organization
            type:Person
            name:Jan Paul Medema
            affiliation:
                  name:University Hospital Leiden
                  address:
                     name:Department of Immunohematology and Bloodbank, University Hospital Leiden, Leiden, The Netherlands
                     type:PostalAddress
                  type:Organization
            type:Person
            name:Frank Kischkel
            affiliation:
                  name:German Cancer Research Center
                  address:
                     name:Tumor Immunology Program, German Cancer Research Center, Heidelberg, Germany
                     type:PostalAddress
                  type:Organization
            type:Person
            name:Peter H. Krammer
            affiliation:
                  name:German Cancer Research Center
                  address:
                     name:Tumor Immunology Program, German Cancer Research Center, Heidelberg, Germany
                     type:PostalAddress
                  type:Organization
            type:Person
      keywords:Death Receptor, Death Domain, African Swine Fever Virus, Death Effector Domain, Acidic SMase
      description:In recent years apoptosis, also called programmed cell death, has been recognized to be the physiological way for a nucleated animal cell to die. Apoptosis takes care of unwanted, injured or virus-infected cells (Farber 1994; Collins 1995). Autoreactive T and B cells that are produced by the immune system by the millions every day are also eliminated by apoptosis. A large number of stimuli can trigger apoptosis. However, only the discovery of the existence of receptors that could trigger apoptosis convinced everyone that a certain substance would not just kill a cell due to its high toxicity but involve special apoptosis-inducing mechanisms. A number of receptors that were first shown to have other functions besides induction of apoptosis could kill cells. These receptors include the T cell receptor/CD3 complex (Smith et al. 1989; Takahashi et al. 1989; Newell et al. 1990), the B cell receptor (Ales-Martinez et al. 1992), CD2 (Merkenschlager and Fisher 1991), CD4 (Wadsworth et al. 1990) and the mouse antigen Thy-1 (Ucker et al. 1989).
      datePublished:1999
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Book:
      name:Apoptosis: Biology and Mechanisms
      isbn:
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         978-3-662-21623-1
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      name:Springer Berlin Heidelberg
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         type:ImageObject
      name:German Cancer Research Center
      address:
         name:Tumor Immunology Program, German Cancer Research Center, Heidelberg, Germany
         type:PostalAddress
      name:German Cancer Research Center
      address:
         name:Tumor Immunology Program, German Cancer Research Center, Heidelberg, Germany
         type:PostalAddress
      name:University Hospital Leiden
      address:
         name:Department of Immunohematology and Bloodbank, University Hospital Leiden, Leiden, The Netherlands
         type:PostalAddress
      name:German Cancer Research Center
      address:
         name:Tumor Immunology Program, German Cancer Research Center, Heidelberg, Germany
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         type:PostalAddress
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      name:Marcus E. Peter
      affiliation:
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            address:
               name:Tumor Immunology Program, German Cancer Research Center, Heidelberg, Germany
               type:PostalAddress
            type:Organization
      name:Carsten Scaffidi
      affiliation:
            name:German Cancer Research Center
            address:
               name:Tumor Immunology Program, German Cancer Research Center, Heidelberg, Germany
               type:PostalAddress
            type:Organization
      name:Jan Paul Medema
      affiliation:
            name:University Hospital Leiden
            address:
               name:Department of Immunohematology and Bloodbank, University Hospital Leiden, Leiden, The Netherlands
               type:PostalAddress
            type:Organization
      name:Frank Kischkel
      affiliation:
            name:German Cancer Research Center
            address:
               name:Tumor Immunology Program, German Cancer Research Center, Heidelberg, Germany
               type:PostalAddress
            type:Organization
      name:Peter H. Krammer
      affiliation:
            name:German Cancer Research Center
            address:
               name:Tumor Immunology Program, German Cancer Research Center, Heidelberg, Germany
               type:PostalAddress
            type:Organization
PostalAddress:
      name:Tumor Immunology Program, German Cancer Research Center, Heidelberg, Germany
      name:Tumor Immunology Program, German Cancer Research Center, Heidelberg, Germany
      name:Department of Immunohematology and Bloodbank, University Hospital Leiden, Leiden, The Netherlands
      name:Tumor Immunology Program, German Cancer Research Center, Heidelberg, Germany
      name:Tumor Immunology Program, German Cancer Research Center, Heidelberg, Germany
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