We are analyzing https://link.springer.com/chapter/10.1007/978-1-4939-0897-4_1.

Title:
Microbial Endocrinology and the Microbiota-Gut-Brain Axis | SpringerLink
Description:
Microbial endocrinology is defined as the study of the ability of microorganisms to both produce and recognize neurochemicals that originate either within the microorganisms themselves or within the host they inhabit. As such, microbial endocrinology represents the...
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Keywords {🔍}

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Topics {✒️}

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Questions {❓}

Iyer LM, Aravind L, Coon SL, Klein DC, Koonin EV (2004) Evolution of cell–cell signaling in animals: did late horizontal gene transfer from bacteria have a role?

Schema {🗺️}

ScholarlyArticle:
      headline:Microbial Endocrinology and the Microbiota-Gut-Brain Axis
      pageEnd:24
      pageStart:3
      image:https://media.springernature.com/w153/springer-static/cover/book/978-1-4939-0897-4.jpg
      genre:
         Biomedical and Life Sciences
         Biomedical and Life Sciences (R0)
      isPartOf:
         name:Microbial Endocrinology: The Microbiota-Gut-Brain Axis in Health and Disease
         isbn:
            978-1-4939-0897-4
            978-1-4939-0896-7
         type:Book
      publisher:
         name:Springer New York
         logo:
            url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
            type:ImageObject
         type:Organization
      author:
            name:Mark Lyte
            affiliation:
                  name:Texas Tech University Health Sciences Center
                  address:
                     name:Department of Immunotherapeutics and Biotechnology, Texas Tech University Health Sciences Center, Abilene, USA
                     type:PostalAddress
                  type:Organization
            email:[email protected]
            type:Person
      keywords:Enteric Nervous System, Host Behavior, Neuroendocrine Hormone, Neuroactive Compound, Specific Bacterial Species
      description:Microbial endocrinology is defined as the study of the ability of microorganisms to both produce and recognize neurochemicals that originate either within the microorganisms themselves or within the host they inhabit. As such, microbial endocrinology represents the intersection of the fields of microbiology and neurobiology. The acquisition of neurochemical-based cell-to-cell signaling mechanisms in eukaryotic organisms is believed to have been acquired due to late horizontal gene transfer from prokaryotic microorganisms. When considered in the context of the microbiota’s ability to influence host behavior, microbial endocrinology with its theoretical basis rooted in shared neuroendocrine signaling mechanisms provides for testable experiments with which to understand the role of the microbiota in host behavior and as importantly the ability of the host to influence the microbiota through neuroendocrine-based mechanisms.
      datePublished:2014
      isAccessibleForFree:
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Book:
      name:Microbial Endocrinology: The Microbiota-Gut-Brain Axis in Health and Disease
      isbn:
         978-1-4939-0897-4
         978-1-4939-0896-7
Organization:
      name:Springer New York
      logo:
         url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
         type:ImageObject
      name:Texas Tech University Health Sciences Center
      address:
         name:Department of Immunotherapeutics and Biotechnology, Texas Tech University Health Sciences Center, Abilene, USA
         type:PostalAddress
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      url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
Person:
      name:Mark Lyte
      affiliation:
            name:Texas Tech University Health Sciences Center
            address:
               name:Department of Immunotherapeutics and Biotechnology, Texas Tech University Health Sciences Center, Abilene, USA
               type:PostalAddress
            type:Organization
      email:[email protected]
PostalAddress:
      name:Department of Immunotherapeutics and Biotechnology, Texas Tech University Health Sciences Center, Abilene, USA
WebPageElement:
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