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article, google, scholar, cas, ferroptosis, stroke, wang, cell, liu, iron, chen, brain, death, httpsdoiorgs, intracerebral, zhang, hemorrhage, yang, biol, zhao, ischemic, zhou, injury, res, cerebral, med, tang, chem, lipid, cells, sun, stockwell, role, lin, ischemia, sci, gao, glutathione, mol, neurol, kang, lee, peroxidation, nature, neuronal, httpsdoiorg, huang, model, protects, biophys,

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mir-122-5p/tp53/slc7a11 pathway improves long-term outcomes p62-keap1-nrf2 pathway protects month download article/chapter apoptosis-inducing factor-homologous mitochondrion p53-mediated ferroptotic responses ischemia-induced glutamate release targeting pro-oxidant iron p53-inducible apoptogenic gene hotair/upf1/acsl4 axis glutathione-independent ferroptosis suppressor cox-2/pge2 pathway inhibits focal cerebral ischemia-reperfusion ferroptosis-mediated tissue injury pyrrolizidine alkaloids—metabolic degradation family member protein dhodh-mediated ferroptosis defence late-onset iron deposition collagenase-induced intracerebral hemorrhage haemin-induced cell death hypoxic-ischemic brain injury human blood-brain barrier cysteinyl-trna synthetase p53-dependent apoptotic cells cerebral ischemic/traumatic injuries induces caspase-independent apoptosis structure-activity relationship studies american heart association active-controlled clinical trial mir-214-mediated tfr1 ischemia–reperfusion injury reduces infarct volume short-time applying atorvastatin cerebral ischemia reperfusion autophagy-dependent cell death full article pdf intestinal ischemia/reperfusion p53-mediated activity coq10 coenzyme q10 privacy choices/manage cookies acute cerebral ischemia blood-brain barrier fundamental research funds lipid metabolism disorders stimulating iron export traumatic brain injury oxidative damage insults 2-hydroxy arachidonic acid sesamin attenuates neurotoxicity drosophila peroxidasin gene

Questions {❓}

• Feng H, Stockwell BR (2018) Unsolved mysteries: How does lipid peroxidation cause ferroptosis?
• Stoyanovsky DA, Tyurina YY, Shrivastava I, Bahar I, Tyurin VA, Protchenko O, Jadhav S, Bolevich SB, Kozlov AV, Vladimirov YA, Shvedova AA, Philpott CC, Bayir H, Kagan VE (2019) Iron catalysis of lipid peroxidation in ferroptosis: regulated enzymatic or random free radical reaction?

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         headline:Role of Ferroptosis in Stroke
         description:Stroke is a common and serious nervous system disease caused by the rupture or blockage of the cardiovascular system. It causes millions of deaths and disabilities every year, which is a huge burden on humanity. It may be induced by thrombosis, hypertension, hyperlipidemia, hyperglycemia, smoking, advanced age and so on. According to different causes, stroke can be generally divided into hemorrhagic stroke and ischemic stroke, whose pathogenesis and treatment are quite different. Ferroptosis is a new type of cell death first defined in 2012, which is characterized by non-apoptotic, iron-dependent, and over-accumulated lipid peroxides. Excess lipid reactive oxygen species produced during ferroptosis eventually leads to oxidative cell death. Ferroptosis has been shown to occur and play an important role in tumors, neurological diseases, kidney injury, and ischemia–reperfusion injury. Ferroptosis is also closely related to the pathogenesis of stroke. Moreover, scientists have successfully intervened in the process of stroke in animal models by regulating ferroptosis, indicating that ferroptosis is a new potential target for the treatment of stroke. This paper systematically summarizes the involvement and role of ferroptosis in the pathogenesis of stroke and predicts the potential of ferroptosis in the treatment of stroke. Ferroptosis in stroke. Stroke induces iron overload and lipid metabolism disorders. Elevated iron catalyzes lipid peroxidation and eventually triggers ferroptosis. Conversely, the GSH/GPX4 pathway, as well as CoQ10, Fer-1, and Lip-1, inhibits lipid peroxidation and, thus, alleviates ferroptosis. GSH glutathione; GPX4 glutathione peroxidase 4; CoQ10 coenzyme Q10; Lip-1 liproxstatin-1; Fer-1 ferostatin-1.
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            name:Central South University
            address:
               name:Department of Neurosurgery, Xiangya Hospital, Central South University, Changsha, China
               type:PostalAddress
            type:Organization
            name:Central South University
            address:
               name:Department of Pathophysiology, Xiangya School of Medicine, Central South University, Changsha, China
               type:PostalAddress
            type:Organization
            name:Sepsis Translational Medicine Key Lab of Hunan Province
            address:
               name:Sepsis Translational Medicine Key Lab of Hunan Province, Changsha, China
               type:PostalAddress
            type:Organization
            name:Central South University
            address:
               name:China-Africa Research Center of Infectious Diseases, Central South University, Changsha, China
               type:PostalAddress
            type:Organization
      name:Lin Zhou
      url:http://orcid.org/0000-0002-1823-6228
      affiliation:
            name:Central South University
            address:
               name:Department of Neurosurgery, Xiangya Hospital, Central South University, Changsha, China
               type:PostalAddress
            type:Organization
            name:Central South University
            address:
               name:Department of Pathophysiology, Xiangya School of Medicine, Central South University, Changsha, China
               type:PostalAddress
            type:Organization
            name:Sepsis Translational Medicine Key Lab of Hunan Province
            address:
               name:Sepsis Translational Medicine Key Lab of Hunan Province, Changsha, China
               type:PostalAddress
            type:Organization
            name:Central South University
            address:
               name:China-Africa Research Center of Infectious Diseases, Central South University, Changsha, China
               type:PostalAddress
            type:Organization
      name:Ying Liu
      url:http://orcid.org/0000-0003-3392-5977
      affiliation:
            name:Central South University
            address:
               name:Department of Neurosurgery, Xiangya Hospital, Central South University, Changsha, China
               type:PostalAddress
            type:Organization
            name:Central South University
            address:
               name:Department of Pathophysiology, Xiangya School of Medicine, Central South University, Changsha, China
               type:PostalAddress
            type:Organization
            name:Sepsis Translational Medicine Key Lab of Hunan Province
            address:
               name:Sepsis Translational Medicine Key Lab of Hunan Province, Changsha, China
               type:PostalAddress
            type:Organization
            name:Central South University
            address:
               name:China-Africa Research Center of Infectious Diseases, Central South University, Changsha, China
               type:PostalAddress
            type:Organization
      email:[email protected]
      name:Jie Zhao
      url:http://orcid.org/0000-0003-3760-4038
      affiliation:
            name:Central South University
            address:
               name:Department of Neurosurgery, Xiangya Hospital, Central South University, Changsha, China
               type:PostalAddress
            type:Organization
      email:[email protected]
PostalAddress:
      name:Department of Neurosurgery, Xiangya Hospital, Central South University, Changsha, China
      name:Department of Pathophysiology, Xiangya School of Medicine, Central South University, Changsha, China
      name:Sepsis Translational Medicine Key Lab of Hunan Province, Changsha, China
      name:China-Africa Research Center of Infectious Diseases, Central South University, Changsha, China
      name:Department of Neurosurgery, Xiangya Hospital, Central South University, Changsha, China
      name:Department of Pathophysiology, Xiangya School of Medicine, Central South University, Changsha, China
      name:Sepsis Translational Medicine Key Lab of Hunan Province, Changsha, China
      name:China-Africa Research Center of Infectious Diseases, Central South University, Changsha, China
      name:Department of Neurosurgery, Xiangya Hospital, Central South University, Changsha, China
      name:Department of Pathophysiology, Xiangya School of Medicine, Central South University, Changsha, China
      name:Sepsis Translational Medicine Key Lab of Hunan Province, Changsha, China
      name:China-Africa Research Center of Infectious Diseases, Central South University, Changsha, China
      name:Department of Neurosurgery, Xiangya Hospital, Central South University, Changsha, China
      name:Department of Pathophysiology, Xiangya School of Medicine, Central South University, Changsha, China
      name:Sepsis Translational Medicine Key Lab of Hunan Province, Changsha, China
      name:China-Africa Research Center of Infectious Diseases, Central South University, Changsha, China
      name:Department of Neurosurgery, Xiangya Hospital, Central South University, Changsha, China
      name:Department of Pathophysiology, Xiangya School of Medicine, Central South University, Changsha, China
      name:Sepsis Translational Medicine Key Lab of Hunan Province, Changsha, China
      name:China-Africa Research Center of Infectious Diseases, Central South University, Changsha, China
      name:Department of Neurosurgery, Xiangya Hospital, Central South University, Changsha, China
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• What's the financial gain of https://support.springernature.com/en/support/home?
• https://support.springernature.com/en/support/solutions/articles/6000255911-subscription-cancellations's total income per month
• How much money does https://www.springernature.com/ generate?

Analytics and Tracking {📊}

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