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Keywords {🔍}

ace, pubmed, article, google, scholar, sarscov, cas, central, receptor, infection, covid, expression, cell, human, enzyme, respiratory, entry, role, angiotensin, binding, protein, angiotensinconverting, severe, cells, system, angii, clin, soluble, inflammation, plays, raas, coronavirus, lung, people, therapeutic, spike, httpsdoiorgs, sci, increased, level, shedding, kks, disease, high, sace, epithelial, increase, hypertension, tmprss, ang,

Topics {✒️}

renin-angiotensin-aldosterone-sars-cov axis des-arg9 bradykinin/bkb1r axis severe-acute respiratory syndrome-coronavirus angiotensin-converting enzyme-related carboxypeptidase full-length sars-cov-2 receptor /ace2/alamandine/mas-related gpcr tnf-alpha-converting enzyme cliniques universitaires saint-luc angiotensin ii-dependent superoxide endogenous counter-regulatory pathway /jun n-terminal kinase maria gabriella matera mas-related gpcr member single-chain antibody fragment renin–angiotensin–aldosterone system renin-angiotensin-aldosterone system cell entry receptors intracellular c-terminal domain kinin b1 receptors promoting sars-cov uptake mediating sars-cov-2 entry tumor necrosis factor lucio pastore sars-cov-2 entry receptor lys-[des-arg9]-bk lambert dw gen-covid multicenter study determining sars-cov infectivity sars-cov-2 receptor ace2 c-terminal membrane anchor competitively bind sars-cov-2 inactivate sars-cov-2 present reduce virus/cell binding sars-cov-2 spike protein surface ace2 receptor article scialo angiotensin converting enzyme-2 angiotensin-converting enzyme angiotensin-converting enzyme 2 angiotensin-converting enzyme-2 decrease sars-cov-2 infection bradykinin receptor-b1 sars-cov-2/ace2 binding single amino acid [des-arg 937]-bradykinin privacy choices/manage cookies viral treatment induced pre-existing conditions [8] facilitates viral entry interferon-stimulated gene

Questions {❓}

• Batlle D, Wysocki J, Satchell K (2020) Soluble angiotensin-converting enzyme 2: a potential approach for coronavirus infection therapy?
• Bianco A, Whiteman SC, Sethi SK, Allen JT, Knight RA, Spiteri MA (2000) Expression of intercellular adhesion molecule-1 (ICAM-1) in nasal epithelial cells of atopic subjects: a mechanism for increased rhinovirus infection?
• Gouda AS, Mégarbane B (2020) Snake venom-derived bradykinin-potentiating peptides: a promising therapy for COVID-19?
• Perrotta F, Matera MG, Cazzola M, Bianco A (2020) Severe respiratory SARS-CoV2 infection: does ACE2 receptor matter?
• Saeedi Saravi SS, Beer JH (2020) Apelin-potential therapy for COVID-19?
• Ye M, Wysocki J, Naaz P, Salabat MR, LaPointe MS, Batlle D (2004) Increased ACE 2 and decreased ACE protein in renal tubules from diabetic mice: a renoprotective combination?

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      description:Despite the unprecedented effort of the scientific community, the novel SARS-CoV-2 virus has infected more than 46 million people worldwide, killing over one million two hundred thousand. Understanding the mechanisms by which some individuals are more susceptible to SARS-CoV-2 infection and why a subgroup of them are prone to experience severe pneumonia, and death should lead to a better approach and more effective treatments for COVID-19. Here, we focus our attention on ACE2, a primary receptor of SARS-CoV-2. We will discuss its biology, tissue expression, and post-translational regulation that determine its potential to be employed by SARS-CoV-2 for cell entry. Particular attention will be given to how the ACE2 soluble form can have a great impact on disease progression and thus be used in a potential therapeutic strategy. Furthermore, we will discuss repercussions that SARS-CoV-2/ACE2 binding has on the renin–angiotensin system and beyond. Indeed, although mostly neglected, ACE2 can also act on [des-Arg 937]-bradykinin of the kinin–kallikrein system regulating coagulation and inflammation. Thorough comprehension of the role that ACE2 plays in different pathways will be the key to assess the impact that SARS-CoV-2/ACE2 binding has on organismal physiology and will help us to find better therapies and diagnostic tools.
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