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Keywords {🔍}

cancer, hlaa, prostate, sart, patients, peptides, cells, peptide, supertype, pbmcs, article, google, scholar, alleles, itoh, ctls, cra, pubmed, cas, peptidespecific, expression, candidates, cytotoxicity, cell, lncap, cytotoxic, harada, iggs, lymphocytes, lines, lncapa, identified, induction, assay, cancerreactive, based, hiv, molecules, reactive, table, protein, sartderived, potential, induce, vaccine, binding, igg, plasma, japan, peptidestimulated,

Topics {✒️}

fitc-conjugated anti-mouse immunoglobulin inducing hla-a24-restricted cytotoxic ctl precursor-oriented vaccines peptide-based anti-cancer vaccine peptide-pulsed unlabeled c1r-a11 hla-a11+ t-cell blasts hla-a3 supertype alleles parathyroid hormone-related protein hiv peptide-pulsed c1r-a11 epithelial cancer-related antigens sart3 tumor-rejection antigen north american african–americans individual peptide side-chains induce peptide-specific cytotoxic induce cancer-reactive cytotoxic prostate cancer-reactive cytotoxic administered ctl-directed peptides measure anti-peptide antibody generate peptide-specific ctls subjects included hla-a11+ enzyme-linked immunosorbent assay detect peptide-specific cytotoxic induce peptide-specific ctls maximum release—spontaneous release overlapping peptide-binding repertoires induce cancer-reactive ctls generating peptide-specific ctls anti-hla-a33 mab thousand 51cr-labeled cells superscript-pre-amplification system stimulated t-cell blasts peptide-specific cellular responses recurrent hormone-refractory peptide-pulsed c1r-a11 peptide-binding motif analyses 51cr-labeled target cells antibodies—anti-hla-a11 hla-a68 molecules belong sart3 peptide-specific igg sart3-derived peptides listed tumor-rejection antigens 0201-restricted ctl epitope rt-pcr-based method anti-cancer vaccine human prostate cancer prostate cancer patients hla-a24 alleles innovative cancer therapy peptide-specific cytotoxic induce peptide-specific

Schema {🗺️}

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         headline:Identification of SART3-derived peptides having the potential to induce cancer-reactive cytotoxic T lymphocytes from prostate cancer patients with HLA-A3 supertype alleles
         description:SART3-derived peptides applicable to prostate cancer patients with HLA-A3 supertype alleles were identified in order to expand the possibility of an anti-cancer vaccine, because the peptide vaccine candidates receiving the most attention thus far have been the HLA-A2 and HLA-A24 alleles. Twenty-nine SART3-derived peptides that were prepared based on the binding motif to the HLA-A3 supertype alleles (HLA-A11, -A31, and -A33) were first screened for their recognizability by immunoglobulin G (IgG) of prostate cancer patients and subsequently for the potential to induce peptide-specific cytotoxic T lymphocytes (CTLs) from HLA-A3 supertype+ prostate cancer patients. As a result, five SART3 peptides were frequently recognized by IgG, and two of them—SART3 511-19 and SART3 734-42—efficiently induced peptide-specific and cancer-reactive CTLs. Their cytotoxicity toward prostate cancer cells was ascribed to peptide-specific and CD8+ T cells. These results indicate that these two SART3 peptides could be promising candidates for peptide-based immunotherapy for HLA-A3 supertype+ prostate cancer patients.
         datePublished:2006-08-26T00:00:00Z
         dateModified:2006-08-26T00:00:00Z
         pageStart:689
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            Prostate cancer
            SART3
            CTL
            Peptide
            HLA-A3 supertype
            Oncology
            Immunology
            Cancer Research
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      headline:Identification of SART3-derived peptides having the potential to induce cancer-reactive cytotoxic T lymphocytes from prostate cancer patients with HLA-A3 supertype alleles
      description:SART3-derived peptides applicable to prostate cancer patients with HLA-A3 supertype alleles were identified in order to expand the possibility of an anti-cancer vaccine, because the peptide vaccine candidates receiving the most attention thus far have been the HLA-A2 and HLA-A24 alleles. Twenty-nine SART3-derived peptides that were prepared based on the binding motif to the HLA-A3 supertype alleles (HLA-A11, -A31, and -A33) were first screened for their recognizability by immunoglobulin G (IgG) of prostate cancer patients and subsequently for the potential to induce peptide-specific cytotoxic T lymphocytes (CTLs) from HLA-A3 supertype+ prostate cancer patients. As a result, five SART3 peptides were frequently recognized by IgG, and two of them—SART3 511-19 and SART3 734-42—efficiently induced peptide-specific and cancer-reactive CTLs. Their cytotoxicity toward prostate cancer cells was ascribed to peptide-specific and CD8+ T cells. These results indicate that these two SART3 peptides could be promising candidates for peptide-based immunotherapy for HLA-A3 supertype+ prostate cancer patients.
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      dateModified:2006-08-26T00:00:00Z
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         Prostate cancer
         SART3
         CTL
         Peptide
         HLA-A3 supertype
         Oncology
         Immunology
         Cancer Research
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      name:Department of Immunology, Kurume University School of Medicine, Kurume, Japan
      name:Department of Immunology, Kurume University School of Medicine, Kurume, Japan
      name:Department of Urology, Kurume University School of Medicine, Kurume, Japan
      name:Department of Urology, Kinki University School of Medicine, Osaka, Japan
      name:Cancer Vaccine Development Division, Kurume University Research Center for Innovative Cancer Therapy, Kurume University School of Medicine, Kurume, Japan
      name:Department of Immunology, Kurume University School of Medicine, Kurume, Japan
      name:Cancer Vaccine Development Division, Kurume University Research Center for Innovative Cancer Therapy, Kurume University School of Medicine, Kurume, Japan
      name:Department of Immunology, Kurume University School of Medicine, Kurume, Japan
      name:Department of Immunology, Shimane University School of Medicine, Izumo, Japan

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