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Keywords {🔍}

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Topics {✒️}

age-adjusted partial correlations postmenopausal estrogen/progestin intervention anti-androgen treatment physician-led telephone interviews sex hormone-binding globulin estrogen/progestin replacement study short-term randomised trials post-challenge glucose differences postmenopausal estrogen/progestin interventions previous cross-sectional analyses sex steroid influence cross-sectional observational studies nested case–control study c-reactive protein levels matched case–control pairs risk-set sampling approach short-term trials plasma sex hormones mailed supplementary questionnaires endogenous sex hormones privacy choices/manage cookies oral conjugated estrogen hormone replacement therapy sex hormones biomarkers short-term exposure sex hormone biology transdermal 17 beta-estradiol sex hormone precursor hormonal substitution therapy improved insulin action oral oestrogen therapy upstream precursor hormones long-term cryoconservation promoting glucose intolerance plasma hormone levels postmenopausal hormone therapy endothelial adhesion molecules human plasma proteins c-reactive protein oral oestrogen trials combined supplementary questionnaire 1 log-sd increase van baal wm downstream hormone metabolites clinical data suggests pregnancy lasting ≥6 months increase visceral adiposity risk-set sampling conjugated equine oestrogen improve glucose control

Schema {🗺️}

WebPage:
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         headline:Plasma sex steroid hormones and risk of developing type 2 diabetes in women: a prospective study
         description:Prospective data directly investigating the role of endogenous sex hormones in diabetes risk have been scant, particularly in women. We aimed to examine comprehensively plasma sex hormones in connection with risk of developing type 2 diabetes in postmenopausal women. We conducted a prospective, nested case–control study of plasma oestradiol, testosterone and dehydroepiandrosterone sulfate and risk of type 2 diabetes in a cohort of women health professionals with a mean age of 60.3 and 12.2 years since menopause. Among women not using hormone therapy and free of baseline cardiovascular disease, cancer and diabetes, 359 incident cases of type 2 diabetes were matched with 359 controls during an average follow-up of 10 years. Oestradiol and testosterone were each strongly and positively associated with risk of type 2 diabetes. After adjustment for BMI, family history, lifestyle and reproductive variables, the multivariable relative risks (95% CI) comparing the highest vs lowest quintile were 12.6 (2.83–56.3) for total oestradiol (p = 0.002 for trend), 13.1 (4.18–40.8) for free oestradiol (p < 0.001 for trend), 4.15 (1.21–14.2) for total testosterone (p = 0.019 for trend) and 14.8 (4.44–49.2) for free testosterone (p < 0.001 for trend). These associations remained robust after adjusting and accounting for other metabolic syndrome components and baseline HbA1c levels. In postmenopausal women, higher plasma levels of oestradiol and testosterone were strongly and prospectively related to increased risk of developing type 2 diabetes. These prospective data indicate that endogenous levels of sex hormones may play important roles in the pathogenesis of type 2 diabetes. ClinicalTrials.gov ID no.: NCT00000479.
         datePublished:2007-08-14T00:00:00Z
         dateModified:2007-08-14T00:00:00Z
         pageStart:2076
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            Dehydroepiandrosterone sulphate
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            Sex hormones
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      headline:Plasma sex steroid hormones and risk of developing type 2 diabetes in women: a prospective study
      description:Prospective data directly investigating the role of endogenous sex hormones in diabetes risk have been scant, particularly in women. We aimed to examine comprehensively plasma sex hormones in connection with risk of developing type 2 diabetes in postmenopausal women. We conducted a prospective, nested case–control study of plasma oestradiol, testosterone and dehydroepiandrosterone sulfate and risk of type 2 diabetes in a cohort of women health professionals with a mean age of 60.3 and 12.2 years since menopause. Among women not using hormone therapy and free of baseline cardiovascular disease, cancer and diabetes, 359 incident cases of type 2 diabetes were matched with 359 controls during an average follow-up of 10 years. Oestradiol and testosterone were each strongly and positively associated with risk of type 2 diabetes. After adjustment for BMI, family history, lifestyle and reproductive variables, the multivariable relative risks (95% CI) comparing the highest vs lowest quintile were 12.6 (2.83–56.3) for total oestradiol (p = 0.002 for trend), 13.1 (4.18–40.8) for free oestradiol (p < 0.001 for trend), 4.15 (1.21–14.2) for total testosterone (p = 0.019 for trend) and 14.8 (4.44–49.2) for free testosterone (p < 0.001 for trend). These associations remained robust after adjusting and accounting for other metabolic syndrome components and baseline HbA1c levels. In postmenopausal women, higher plasma levels of oestradiol and testosterone were strongly and prospectively related to increased risk of developing type 2 diabetes. These prospective data indicate that endogenous levels of sex hormones may play important roles in the pathogenesis of type 2 diabetes. ClinicalTrials.gov ID no.: NCT00000479.
      datePublished:2007-08-14T00:00:00Z
      dateModified:2007-08-14T00:00:00Z
      pageStart:2076
      pageEnd:2084
      sameAs:https://doi.org/10.1007/s00125-007-0785-y
      keywords:
         Androgen
         Dehydroepiandrosterone sulphate
         Estradiol
         Estrogen
         Oestradiol
         Oestrogen
         Sex hormones
         Steroid hormones
         Testosterone
         Type 2 diabetes
         Women
         Internal Medicine
         Metabolic Diseases
         Human Physiology
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            name:E. L. Ding
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      name:Division of Preventive Medicine, Department of Medicine, Brigham and Women’s Hospital and Harvard Medical School, Boston, USA
      name:Division of Preventive Medicine, Department of Medicine, Brigham and Women’s Hospital and Harvard Medical School, Boston, USA
      name:Department of Epidemiology, Harvard School of Public Health, Boston, USA
      name:Department of Laboratory Medicine, Children’s Hospital and Harvard Medical School, Boston, USA
      name:Division of Preventive Medicine, Department of Medicine, Brigham and Women’s Hospital and Harvard Medical School, Boston, USA
      name:Department of Epidemiology, Harvard School of Public Health, Boston, USA
      name:Division of Preventive Medicine, Department of Medicine, Brigham and Women’s Hospital and Harvard Medical School, Boston, USA
      name:Department of Epidemiology, Harvard School of Public Health, Boston, USA
      name:School of Public Health, Department of Epidemiology, University of California, Los Angeles, Los Angeles, USA

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